scholarly article | Q13442814 |
P50 | author | Benjamin Cravatt | Q4888433 |
P2093 | author name string | Loren H Parsons | |
Fernando Rodriguez de Fonseca | |||
Antonia Serrano | |||
Nimish Sidhpura | |||
Rémi Martin-Fardon | |||
Ilham Polis | |||
David Stouffer | |||
Francisco J Pavon | |||
P2860 | cites work | Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase | Q24555807 |
Braking dopamine systems: a new GABA master structure for mesolimbic and nigrostriatal functions | Q24603596 | ||
Discovery and Characterization of a Highly Selective FAAH Inhibitor that Reduces Inflammatory Pain | Q27655121 | ||
Inhibition of FAAH and activation of PPAR: new approaches to the treatment of cognitive dysfunction and drug addiction | Q28283718 | ||
Enzymatic pathways that regulate endocannabinoid signaling in the nervous system | Q28390932 | ||
Blockade of nicotine reward and reinstatement by activation of alpha-type peroxisome proliferator-activated receptors | Q28582567 | ||
Effects of rimonabant on the reinstatement of nicotine-conditioned place preference by drug priming in rats | Q28584275 | ||
Endocannabinoid signalling in reward and addiction. | Q30373359 | ||
Properties of rat liver N-acylethanolamine amidohydrolase | Q34200418 | ||
The role of fatty acid amide hydrolase inhibition in nicotine reward and dependence | Q34282073 | ||
Effects of fatty acid amide hydrolase inhibition on neuronal responses to nicotine, cocaine and morphine in the nucleus accumbens shell and ventral tegmental area: involvement of PPAR-alpha nuclear receptors. | Q35196626 | ||
Endogenous fatty acid ethanolamides suppress nicotine-induced activation of mesolimbic dopamine neurons through nuclear receptors | Q35202878 | ||
Endocannabinoid influence in drug reinforcement, dependence and addiction-related behaviors | Q35532072 | ||
Nicotine: from molecular mechanisms to behaviour | Q36015617 | ||
Neuronal nicotinic acetylcholine receptors: neuroplastic changes underlying alcohol and nicotine addictions | Q36137598 | ||
The role of the cannabinoid system in nicotine addiction. | Q36144660 | ||
Effects of Fatty Acid Amide Hydrolase (FAAH) Inhibitors in Non-Human Primate Models of Nicotine Reward and Relapse | Q36184904 | ||
Nicotine and cannabinoids: parallels, contrasts and interactions | Q36627713 | ||
Inhibition of anandamide hydrolysis by cyclohexyl carbamic acid 3'-carbamoyl-3-yl ester (URB597) reverses abuse-related behavioral and neurochemical effects of nicotine in rats | Q37146387 | ||
The endogenous cannabinoid system modulates nicotine reward and dependence | Q37353755 | ||
The genetic basis of the endocannabinoid system and drug addiction in humans. | Q37936694 | ||
Endocannabinoids, related compounds and their metabolic routes. | Q38262964 | ||
Endocannabinoids and Their Pharmacological Actions | Q38593112 | ||
5-HT(6) receptor antagonism potentiates the behavioral and neurochemical effects of amphetamine but not cocaine | Q43862483 | ||
Lack of CB1 cannabinoid receptors modifies nicotine behavioural responses, but not nicotine abstinence. | Q44182704 | ||
SR141716, a central cannabinoid (CB(1)) receptor antagonist, blocks the motivational and dopamine-releasing effects of nicotine in rats | Q44190296 | ||
Rimonabant, a CB1 antagonist, blocks nicotine-conditioned place preferences. | Q45108517 | ||
Inhibition of fatty acid amide hydrolase reduces reinstatement of nicotine seeking but not break point for nicotine self-administration--comparison with CB(1) receptor blockade. | Q45988694 | ||
Effects of the cannabinoid CB1 receptor antagonist AM 251 on the reinstatement of nicotine-conditioned place preference by drug priming in rats. | Q46011230 | ||
Cannabinoid receptor stimulation increases motivation for nicotine and nicotine seeking | Q48139928 | ||
P921 | main subject | fatty acid | Q61476 |
dopamine | Q170304 | ||
nucleus accumbens | Q1476178 | ||
niacinamide | Q192423 | ||
P577 | publication date | 2017-06-29 | |
P1433 | published in | Addiction Biology | Q4681109 |
P1476 | title | Fatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens |
Q89635971 | Corticotropin-Releasing Factor Receptor-1 Neurons in the Lateral Amygdala Display Selective Sensitivity to Acute and Chronic Ethanol Exposure |
Q91490079 | D3 dopamine receptors and a missense mutation of fatty acid amide hydrolase linked in mouse and men: implication for addiction |
Q57175144 | Decreased Cannabinoid CB Receptors in Male Tobacco Smokers Examined With Positron Emission Tomography |