Fatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens

scientific article

Fatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1111/ADB.12531
P932PMC publication ID5747548
P698PubMed publication ID28660730

P50authorBenjamin CravattQ4888433
P2093author name stringLoren H Parsons
Fernando Rodriguez de Fonseca
Antonia Serrano
Nimish Sidhpura
Rémi Martin-Fardon
Ilham Polis
David Stouffer
Francisco J Pavon
P2860cites workSupersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolaseQ24555807
Braking dopamine systems: a new GABA master structure for mesolimbic and nigrostriatal functionsQ24603596
Discovery and Characterization of a Highly Selective FAAH Inhibitor that Reduces Inflammatory PainQ27655121
Inhibition of FAAH and activation of PPAR: new approaches to the treatment of cognitive dysfunction and drug addictionQ28283718
Enzymatic pathways that regulate endocannabinoid signaling in the nervous systemQ28390932
Blockade of nicotine reward and reinstatement by activation of alpha-type peroxisome proliferator-activated receptorsQ28582567
Effects of rimonabant on the reinstatement of nicotine-conditioned place preference by drug priming in ratsQ28584275
Endocannabinoid signalling in reward and addiction.Q30373359
Properties of rat liver N-acylethanolamine amidohydrolaseQ34200418
The role of fatty acid amide hydrolase inhibition in nicotine reward and dependenceQ34282073
Effects of fatty acid amide hydrolase inhibition on neuronal responses to nicotine, cocaine and morphine in the nucleus accumbens shell and ventral tegmental area: involvement of PPAR-alpha nuclear receptors.Q35196626
Endogenous fatty acid ethanolamides suppress nicotine-induced activation of mesolimbic dopamine neurons through nuclear receptorsQ35202878
Endocannabinoid influence in drug reinforcement, dependence and addiction-related behaviorsQ35532072
Nicotine: from molecular mechanisms to behaviourQ36015617
Neuronal nicotinic acetylcholine receptors: neuroplastic changes underlying alcohol and nicotine addictionsQ36137598
The role of the cannabinoid system in nicotine addiction.Q36144660
Effects of Fatty Acid Amide Hydrolase (FAAH) Inhibitors in Non-Human Primate Models of Nicotine Reward and RelapseQ36184904
Nicotine and cannabinoids: parallels, contrasts and interactionsQ36627713
Inhibition of anandamide hydrolysis by cyclohexyl carbamic acid 3'-carbamoyl-3-yl ester (URB597) reverses abuse-related behavioral and neurochemical effects of nicotine in ratsQ37146387
The endogenous cannabinoid system modulates nicotine reward and dependenceQ37353755
The genetic basis of the endocannabinoid system and drug addiction in humans.Q37936694
Endocannabinoids, related compounds and their metabolic routes.Q38262964
Endocannabinoids and Their Pharmacological ActionsQ38593112
5-HT(6) receptor antagonism potentiates the behavioral and neurochemical effects of amphetamine but not cocaineQ43862483
Lack of CB1 cannabinoid receptors modifies nicotine behavioural responses, but not nicotine abstinence.Q44182704
SR141716, a central cannabinoid (CB(1)) receptor antagonist, blocks the motivational and dopamine-releasing effects of nicotine in ratsQ44190296
Rimonabant, a CB1 antagonist, blocks nicotine-conditioned place preferences.Q45108517
Inhibition of fatty acid amide hydrolase reduces reinstatement of nicotine seeking but not break point for nicotine self-administration--comparison with CB(1) receptor blockade.Q45988694
Effects of the cannabinoid CB1 receptor antagonist AM 251 on the reinstatement of nicotine-conditioned place preference by drug priming in rats.Q46011230
Cannabinoid receptor stimulation increases motivation for nicotine and nicotine seekingQ48139928
P921main subjectfatty acidQ61476
dopamineQ170304
nucleus accumbensQ1476178
niacinamideQ192423
P577publication date2017-06-29
P1433published inAddiction BiologyQ4681109
P1476titleFatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens

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cites work (P2860)
Q89635971Corticotropin-Releasing Factor Receptor-1 Neurons in the Lateral Amygdala Display Selective Sensitivity to Acute and Chronic Ethanol Exposure
Q91490079D3 dopamine receptors and a missense mutation of fatty acid amide hydrolase linked in mouse and men: implication for addiction
Q57175144Decreased Cannabinoid CB Receptors in Male Tobacco Smokers Examined With Positron Emission Tomography