scholarly article | Q13442814 |
P356 | DOI | 10.1002/AJH.24451 |
P698 | PubMed publication ID | 27312795 |
P50 | author | Gilles Salles | Q17511518 |
Diane Damotte | Q37279300 | ||
Corinne Haioun | Q59200159 | ||
Christiane Copie Bergman | Q59621239 | ||
Philippe Gaulard | Q59676235 | ||
Anaïs Pujals | Q60473627 | ||
Francois Lemonnier | Q61054426 | ||
Elodie Bohers | Q90699425 | ||
Richard Delarue | Q117264788 | ||
Sydney Dubois | Q125187577 | ||
Karen Leroy | Q40687224 | ||
Alexandra Traverse-Glehen | Q41605418 | ||
Brigitte Sola | Q42638665 | ||
Sylvain Mareschal | Q43485518 | ||
Martin Dyer | Q53138021 | ||
Noel Jean Milpied | Q55743774 | ||
Thierry J Molina | Q56850574 | ||
Pierre-Julien Viailly | Q56939796 | ||
Thierry Fest | Q56997299 | ||
Martin Figeac | Q58914971 | ||
P2093 | author name string | Philippe Bertrand | |
Peter Moeller | |||
Fabrice Jardin | |||
Hervé Tilly | |||
Yosef Landesman | |||
Vincent Camus | |||
Laura Pelletier | |||
Christian Bastard | |||
Aspasia Stamatoullas | |||
Jean-Philippe Jais | |||
Catherine Maingonnat | |||
Marie Cornic | |||
Jean Michel Picquenot | |||
Christer Sundstrom | |||
Christian Argueta | |||
Emilie Lemasle | |||
Marlène Ochmann | |||
P2860 | cites work | Structural basis for leucine-rich nuclear export signal recognition by CRM1 | Q24604395 |
Molecular subtypes of diffuse large B-cell lymphoma arise by distinct genetic pathways | Q24652565 | ||
Molecular diagnosis of primary mediastinal B cell lymphoma identifies a clinically favorable subgroup of diffuse large B cell lymphoma related to Hodgkin lymphoma | Q24672476 | ||
Selective inhibitors of nuclear export show that CRM1/XPO1 is a target in chronic lymphocytic leukemia | Q27674083 | ||
The GTP-binding domain of class II transactivator regulates its nuclear export | Q28202212 | ||
MHC class II transactivator CIITA is a recurrent gene fusion partner in lymphoid cancers | Q28306431 | ||
Genomic and molecular characterization of esophageal squamous cell carcinoma | Q28657686 | ||
A general framework for estimating the relative pathogenicity of human genetic variants | Q29615730 | ||
limma powers differential expression analyses for RNA-sequencing and microarray studies | Q29617988 | ||
B-aggressive lymphoma family proteins have unique domains that modulate transcription and exhibit poly(ADP-ribose) polymerase activity | Q29977777 | ||
Mutations of the tumor suppressor gene SOCS-1 in classical Hodgkin lymphoma are frequent and associated with nuclear phospho-STAT5 accumulation | Q33236181 | ||
Recurrent mutations of the STAT6 DNA binding domain in primary mediastinal B-cell lymphoma. | Q33669371 | ||
Report of an international workshop to standardize response criteria for non-Hodgkin's lymphomas. NCI Sponsored International Working Group | Q33773306 | ||
Induction of p53-mediated transcription and apoptosis by exportin-1 (XPO1) inhibition in mantle cell lymphoma | Q33934010 | ||
Jetset: selecting the optimal microarray probe set to represent a gene | Q34102011 | ||
FOXO1 downregulation contributes to the oncogenic program of primary mediastinal B-cell lymphoma. | Q34221760 | ||
Identification of nucleocytoplasmic cycling as a remote sensor in cellular signaling by databased modeling | Q34328572 | ||
Nucleo-cytoplasmic transport as a therapeutic target of cancer | Q34452108 | ||
Primary mediastinal B-cell lymphoma and mediastinal gray zone lymphoma: do they require a unique therapeutic approach? | Q34802443 | ||
Recurrent somatic mutations of PTPN1 in primary mediastinal B cell lymphoma and Hodgkin lymphoma | Q35095028 | ||
Survival impact of rituximab combined with ACVBP and upfront consolidation autotransplantation in high-risk diffuse large B-cell lymphoma for GELA. | Q35143471 | ||
Nucleocytoplasmic shuttling by nucleoporins Nup153 and Nup214 and CRM1-dependent nuclear export control the subcellular distribution of latent Stat1. | Q36322486 | ||
CRM1 inhibition induces tumor cell cytotoxicity and impairs osteoclastogenesis in multiple myeloma: molecular mechanisms and therapeutic implications. | Q37445828 | ||
KPT-330 inhibitor of CRM1 (XPO1)-mediated nuclear export has selective anti-leukaemic activity in preclinical models of T-cell acute lymphoblastic leukaemia and acute myeloid leukaemia. | Q37693609 | ||
The molecular pathogenesis of primary mediastinal large B-cell lymphoma | Q37893841 | ||
Gray zone lymphoma: better treated like hodgkin lymphoma or mediastinal large B-cell lymphoma? | Q38029564 | ||
Targeting the nuclear transport machinery by rational drug design | Q38053475 | ||
Understanding XPO1 target networks using systems biology and mathematical modeling | Q38093183 | ||
Flow sorting and exome sequencing reveal the oncogenome of primary Hodgkin and Reed-Sternberg cells. | Q38930717 | ||
U-2940, a human B-cell line derived from a diffuse large cell lymphoma sequential to Hodgkin lymphoma | Q40384149 | ||
NFkappaB activity, function, and target-gene signatures in primary mediastinal large B-cell lymphoma and diffuse large B-cell lymphoma subtypes | Q40427138 | ||
Biallelic mutation of SOCS-1 impairs JAK2 degradation and sustains phospho-JAK2 action in the MedB-1 mediastinal lymphoma line | Q40486172 | ||
MedB-1, a human tumor cell line derived from a primary mediastinal large B-cell lymphoma | Q40814255 | ||
Identification of Primary Mediastinal Large B-cell Lymphoma at Nonmediastinal Sites by Gene Expression Profiling | Q41620612 | ||
Identifying drug-target selectivity of small-molecule CRM1/XPO1 inhibitors by CRISPR/Cas9 genome editing | Q42175237 | ||
Cryptic XPO1-MLLT10 translocation is associated with HOXA locus deregulation in T-ALL. | Q42986380 | ||
Rgb: a scriptable genome browser for R. | Q44093114 | ||
A chemical genetic screen identifies inhibitors of regulated nuclear export of a Forkhead transcription factor in PTEN-deficient tumor cells | Q44714283 | ||
Dose-dense rituximab-CHOP compared with standard rituximab-CHOP in elderly patients with diffuse large B-cell lymphoma (the LNH03-6B study): a randomised phase 3 trial. | Q50492479 | ||
Evaluation of structural and evolutionary contributions to deleterious mutation prediction. | Q52032041 | ||
SF3B1 mutations correlated to cytogenetics and mutations in NOTCH1, FBXW7, MYD88, XPO1 and TP53 in 1160 untreated CLL patients. | Q52644149 | ||
Next-Generation Sequencing in Diffuse Large B-Cell Lymphoma Highlights Molecular Divergence and Therapeutic Opportunities: a LYSA Study. | Q52887085 | ||
CRM1 as a new therapeutic target for non-Hodgkin lymphoma. | Q52997079 | ||
The molecular signature of mediastinal large B-cell lymphoma differs from that of other diffuse large B-cell lymphomas and shares features with classical Hodgkin lymphoma. | Q53931762 | ||
Intensified chemotherapy with ACVBP plus rituximab versus standard CHOP plus rituximab for the treatment of diffuse large B-cell lymphoma (LNH03-2B): an open-label randomised phase 3 trial. | Q55055249 | ||
Phase III study of ACVBP versus ACVBP plus rituximab for patients with localized low-risk diffuse large B-cell lymphoma (LNH03-1B) | Q56978661 | ||
Attenuated immunochemotherapy regimen (R-miniCHOP) in elderly patients older than 80 years with diffuse large B-cell lymphoma: a multicentre, single-arm, phase 2 trial | Q56985985 | ||
P433 | issue | 9 | |
P304 | page(s) | 923-930 | |
P577 | publication date | 2016-06-16 | |
P1433 | published in | American Journal of Hematology | Q4744246 |
P1476 | title | Recurrent mutations of the exportin 1 gene (XPO1) and their impact on selective inhibitor of nuclear export compounds sensitivity in primary mediastinal B-cell lymphoma | |
P478 | volume | 91 |
Q64056346 | CRISPR-Cas9 Screening of Kaposi's Sarcoma-Associated Herpesvirus-Transformed Cells Identifies XPO1 as a Vulnerable Target of Cancer Cells |
Q59126311 | Cytoplasmic location of NR4A1 in aggressive lymphomas is associated with a favourable cancer specific survival |
Q38754419 | Detection and prognostic value of recurrent exportin 1 mutations in tumor and cell-free circulating DNA of patients with classical Hodgkin lymphoma. |
Q39363409 | Diagnosis and classification of hematologic malignancies on the basis of genetics. |
Q90482276 | Exportin-1 E571K mutation is a common finding in patients with classical Hodgkin lymphoma |
Q91801414 | Genomic analyses of flow-sorted Hodgkin Reed-Sternberg cells reveal complementary mechanisms of immune evasion |
Q40578878 | Heterozygous mutation of cysteine528 in XPO1 is sufficient for resistance to selective inhibitors of nuclear export |
Q52715766 | KPT-330 inhibition of chromosome region maintenance 1 is cytotoxic and sensitizes chronic myeloid leukemia to Imatinib. |
Q50134836 | Karyopherins in cancer |
Q33729711 | Molecular Testing of Lymphoproliferative Disorders: Current Status and Perspectives |
Q28072797 | New developments in the pathology of malignant lymphoma: a review of the literature published from June-August 2016 |
Q55508866 | New generation sequencing of targeted genes in the classical and the variant form of hairy cell leukemia highlights mutations in epigenetic regulation genes. |
Q37712229 | Nuclear export receptor CRM1 recognizes diverse conformations in nuclear export signals. |
Q102060102 | Once-per-week selinexor, bortezomib, and dexamethasone versus twice-per-week bortezomib and dexamethasone in patients with multiple myeloma (BOSTON): a randomised, open-label, phase 3 trial |
Q92352514 | Prognostic roles of the transcriptional expression of exportins in hepatocellular carcinoma |
Q49579940 | Selective Inhibition of Nuclear Export With Oral Selinexor for Treatment of Relapsed or Refractory Multiple Myeloma. |
Q96647251 | Small Molecule Inhibitors of CRM1 |
Q97093743 | Targeting nuclear import and export in hematological malignancies |
Q101564368 | The nuclear export protein XPO1 - from biology to targeted therapy |
Q64107227 | The past, present, and future of CRM1/XPO1 inhibitors |
Q38729502 | The value of liquid biopsy in diagnosis and monitoring of diffuse large b-cell lymphoma: recent developments and future potential |
Q64944710 | XPO1 Expression Is a Poor-Prognosis Marker in Pancreatic Adenocarcinoma. |
Q39135290 | XPO1 in B cell hematological malignancies: from recurrent somatic mutations to targeted therapy. |