scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1005670226 |
P356 | DOI | 10.1038/SREP18462 |
P932 | PMC publication ID | 4683529 |
P698 | PubMed publication ID | 26680512 |
P5875 | ResearchGate publication ID | 291354518 |
P50 | author | Tushar Kanti Maiti | Q88055369 |
P2093 | author name string | Pranita Hanpude | |
Sushmita Bhattacharya | |||
P2860 | cites work | Gain of function of mutant p53 by coaggregation with multiple tumor suppressors | Q57670523 |
Emerging Anatomy of the BAP1 Tumor Suppressor System | Q58236865 | ||
Protein stability versus function: effects of destabilizing missense mutations onBRCA1DNA repair activity | Q64461858 | ||
Autodeubiquitination protects the tumor suppressor BAP1 from cytoplasmic sequestration mediated by the atypical ubiquitin ligase UBE2O | Q24293775 | ||
Histone H2A deubiquitinase activity of the Polycomb repressive complex PR-DUB | Q24309403 | ||
The deubiquitinating enzyme BAP1 regulates cell growth via interaction with HCF-1. | Q24323060 | ||
Association of C-terminal ubiquitin hydrolase BRCA1-associated protein 1 with cell cycle regulator host cell factor 1 | Q24329078 | ||
BRCA1-associated protein-1 is a tumor suppressor that requires deubiquitinating activity and nuclear localization | Q24337105 | ||
BAP1 is phosphorylated at serine 592 in S-phase following DNA damage | Q24339530 | ||
Frequent mutation of BAP1 in metastasizing uveal melanomas | Q24601069 | ||
The ubiquitin carboxyl hydrolase BAP1 forms a ternary complex with YY1 and HCF-1 and is a critical regulator of gene expression | Q24633127 | ||
Germline BAP1 mutations predispose to malignant mesothelioma | Q24635326 | ||
Thermodynamic stability of wild-type and mutant p53 core domain | Q24648879 | ||
Prediction of sequence-dependent and mutational effects on the aggregation of peptides and proteins | Q28281645 | ||
BAP1 protein loss by immunohistochemistry: a potentially useful tool for prognostic prediction in patients with uveal melanoma | Q30457154 | ||
Conditional expression of Parkinson's disease-related R1441C LRRK2 in midbrain dopaminergic neurons of mice causes nuclear abnormalities without neurodegeneration | Q30653185 | ||
Further evidence for germline BAP1 mutations predisposing to melanoma and malignant mesothelioma | Q30654878 | ||
Exome sequencing identifies frequent inactivating mutations in BAP1, ARID1A and PBRM1 in intrahepatic cholangiocarcinomas | Q33581027 | ||
The forkhead transcription factor FOXK2 acts as a chromatin targeting factor for the BAP1-containing histone deubiquitinase complex | Q33698585 | ||
A molecular revolution in uveal melanoma: implications for patient care and targeted therapy | Q33717375 | ||
Loss of BAP1 protein expression is an independent marker of poor prognosis in patients with low-risk clear cell renal cell carcinoma | Q33821055 | ||
Disease-causing missense mutations in actin binding domain 1 of dystrophin induce thermodynamic instability and protein aggregation | Q34006640 | ||
Missense mutations in dystrophin that trigger muscular dystrophy decrease protein stability and lead to cross-beta aggregates | Q34093884 | ||
Protein folding in the cytoplasm and the heat shock response | Q34152827 | ||
The nuclear deubiquitinase BAP1 is commonly inactivated by somatic mutations and 3p21.1 losses in malignant pleural mesothelioma | Q34190027 | ||
BAP1 loss defines a new class of renal cell carcinoma | Q34280223 | ||
A novel germline mutation in BAP1 predisposes to familial clear-cell renal cell carcinoma. | Q34413144 | ||
Alzheimer presenilin-1 mutations dramatically reduce trimming of long amyloid β-peptides (Aβ) by γ-secretase to increase 42-to-40-residue Aβ. | Q34467478 | ||
A recurrent mutation in PARK2 is associated with familial lung cancer | Q35059365 | ||
Effects of heat shock, heat shock protein 40 (HDJ-2), and proteasome inhibition on protein aggregation in cellular models of Huntington's disease | Q35084195 | ||
Propagation of aggregated p53: Cross-reaction and coaggregation vs. seeding | Q35134517 | ||
Mechanism of initiation of aggregation of p53 revealed by Φ-value analysis | Q35134797 | ||
Regulators of homologous recombination repair as novel targets for cancer treatment. | Q35200273 | ||
Development of cancer-initiating cells and immortalized cells with genomic instability | Q35205699 | ||
α-Synuclein-mediated inhibition of ATF6 processing into COPII vesicles disrupts UPR signaling in Parkinson's disease | Q35569310 | ||
MEK guards proteome stability and inhibits tumor-suppressive amyloidogenesis via HSF1. | Q36041862 | ||
Mutant p53 aggregates into prion-like amyloid oligomers and fibrils: implications for cancer. | Q36201740 | ||
Evaluating Drosophila p53 as a model system for studying cancer mutations | Q36492803 | ||
Germline BAP1 mutations predispose to renal cell carcinomas | Q36909238 | ||
BAP1 and cancer | Q37221089 | ||
Tumor suppressor and deubiquitinase BAP1 promotes DNA double-strand break repair | Q37474898 | ||
Loss of the tumor suppressor BAP1 causes myeloid transformation | Q37545704 | ||
Tumours associated with BAP1 mutations. | Q38070634 | ||
The complex function of hsp70 in metastatic cancer. | Q38173257 | ||
Prion-like aggregation of mutant p53 in cancer | Q38207799 | ||
Targeting heat shock proteins in metastatic castration-resistant prostate cancer | Q38289945 | ||
Amyloidogenicity of p53: a hidden link between protein misfolding and cancer | Q38360401 | ||
Structure based aggregation studies reveal the presence of helix-rich intermediate during α-Synuclein aggregation | Q38898283 | ||
Mutant p53 reprograms TNF signaling in cancer cells through interaction with the tumor suppressor DAB2IP. | Q38934179 | ||
Germline mutations in BAP1 impair its function in DNA double-strand break repair | Q38988548 | ||
Amyloid-like aggregates sequester numerous metastable proteins with essential cellular functions | Q39610788 | ||
Implication of necrosis-linked p53 aggregation in acquired apoptotic resistance to 5-FU in MCF-7 multicellular tumour spheroids | Q39698063 | ||
Induction of Hsp90 protein expression in malignant melanomas and melanoma metastases | Q40580807 | ||
Tumorigenic mutations in VHL disrupt folding in vivo by interfering with chaperonin binding | Q40613613 | ||
Breast cancer risk and possible mechanisms of radiation-induced genomic instability in the Swedish hemangioma cohort after reanalyzed dosimetry | Q41118614 | ||
BRCA1-associated protein 1 (BAP1) deubiquitinase antagonizes the ubiquitin-mediated activation of FoxK2 target genes | Q41752870 | ||
Mutation frequencies of GNAQ, GNA11, BAP1, SF3B1, EIF1AX and TERT in uveal melanoma: detection of an activating mutation in the TERT gene promoter in a single case of uveal melanoma | Q41881966 | ||
Germline BAP1 mutations predispose also to multiple basal cell carcinomas. | Q42463261 | ||
Clinical and pathological impact of VHL, PBRM1, BAP1, SETD2, KDM6A, and JARID1c in clear cell renal cell carcinoma. | Q43806776 | ||
Fibrillar aggregates of the tumor suppressor p53 core domain | Q44528743 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 18462 | |
P577 | publication date | 2015-12-18 | |
P1433 | published in | Scientific Reports | Q2261792 |
P1476 | title | Cancer associated missense mutations in BAP1 catalytic domain induce amyloidogenic aggregation: A new insight in enzymatic inactivation | |
P478 | volume | 5 |