1,25-Dihydroxyvitamin D as Monotherapy for XLH: Back to the Future?

scientific article published on 19 April 2016

1,25-Dihydroxyvitamin D as Monotherapy for XLH: Back to the Future? is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1002/JBMR.2858
P698PubMed publication ID27093323

P50authorDiana Ovejero CrespoQ86894757
P2093author name stringMichael T Collins
Rachel I Gafni
P2860cites workAnti-FGF-23 neutralizing antibodies ameliorate muscle weakness and decreased spontaneous movement of Hyp miceQ46295463
Fibroblast growth factor-23 is regulated by 1alpha,25-dihydroxyvitamin D.Q46761922
Therapeutic effects of anti-FGF23 antibodies in hypophosphatemic rickets/osteomalaciaQ47871507
X-linked hypophosphatemia: effect of calcitriol on renal handling of phosphate, serum phosphate, and bone mineralization.Q51655584
Healing of bone lesions with 1,25-dihydroxyvitamin D3 in the young X-linked hypophosphatemic male mouse.Q54512903
Bone response to phosphate and vitamin D metabolites in the hypophosphatemic male mouseQ54520940
Bone Response to Phosphate Salts, Ergocalciferol, and Calcitriol in Hypophosphatemic Vitamin D-Resistant RicketsQ54546729
X-linked hypophosphatemia: skeletal mass in adults assessed by histomorphometry, computed tomography, and absorptiometryQ58086003
Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23Q24290481
FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasisQ24319751
Treatment of X-linked hypophosphatemia with calcitriol and phosphate increases circulating fibroblast growth factor 23 concentrationsQ24595718
FGF23, PHEX, and MEPE regulation of phosphate homeostasis and skeletal mineralizationQ28177435
Klotho converts canonical FGF receptor into a specific receptor for FGF23Q28272505
Long-term follow-up of patients with hypoparathyroidismQ28276548
Effects of therapy in X-linked hypophosphatemic ricketsQ28321935
X linked hypophosphataemia: treatment, height gain, and nephrocalcinosisQ30495036
FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wastingQ34227442
Hypophosphatemia: mouse model for human familial hypophosphatemic (vitamin D-resistant) ricketsQ35044977
A clinician's guide to X-linked hypophosphatemiaQ35167526
Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism.Q35728669
A Phex mutation in a murine model of X-linked hypophosphatemia alters phosphate responsiveness of bone cellsQ35783146
Prolonged Correction of Serum Phosphorus in Adults With X-Linked Hypophosphatemia Using Monthly Doses of KRN23.Q35829300
Assessment of 24,25(OH)2D levels does not support FGF23-mediated catabolism of vitamin D metabolitesQ36288312
CYP24 inhibition as a therapeutic target in FGF23-mediated renal phosphate wasting disordersQ36515176
Regulation of phosphate homeostasis by PTH, vitamin D, and FGF23.Q36649088
Calcimimetics as an adjuvant treatment for familial hypophosphatemic ricketsQ36662050
1,25-Dihydroxyvitamin D Alone Improves Skeletal Growth, Microarchitecture, and Strength in a Murine Model of XLH, Despite Enhanced FGF23 Expression.Q36889321
Evaluation of a Role for 1,25-Dihydroxyvitamin D3 in the Pathogenesis and Treatment of X-linked Hypophosphatemic Rickets and OsteomalaciaQ37024792
Role of 1,25-dihydroxyvitamin D3 on intestinal phosphate absorption in rats with a normal vitamin D supplyQ37046700
Multilineage somatic activating mutations in HRAS and NRAS cause mosaic cutaneous and skeletal lesions, elevated FGF23 and hypophosphatemiaQ37406418
Tumor localization and biochemical response to cure in tumor-induced osteomalacia.Q37509096
The PTH-Vitamin D-FGF23 axisQ38571195
An FGF23 missense mutation causes familial tumoral calcinosis with hyperphosphatemia.Q40482177
Risk factors affecting the development of nephrocalcinosis, the most common complication of hypophosphatemic ricketsQ41555789
Cinacalcet in the management of tumor-induced osteomalaciaQ42163969
Fibroblast growth factor 23 is a counter-regulatory phosphaturic hormone for vitamin D.Q42493065
X-linked hypophosphatemia: normal renal function despite medullary nephrocalcinosis 25 years after transient vitamin D2-induced renal azotemiaQ42545654
Fibroblast growth factor 23 in oncogenic osteomalacia and X-linked hypophosphatemiaQ44414604
Pharmacological inhibition of fibroblast growth factor (FGF) receptor signaling ameliorates FGF23-mediated hypophosphatemic rickets.Q45020373
FGF-23 is elevated by chronic hyperphosphatemiaQ45050609
P433issue5
P304page(s)925-928
P577publication date2016-05-01
P1433published inJournal of Bone and Mineral ResearchQ15750941
P1476title1,25-Dihydroxyvitamin D as Monotherapy for XLH: Back to the Future?
P478volume31

Reverse relations

cites work (P2860)
Q90624325FGF23 and Associated Disorders of Phosphate Wasting
Q89810553Slc20a1/Pit1 and Slc20a2/Pit2 are essential for normal skeletal myofiber function and survival

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