Pentraxin-3 Attenuates Renal Damage in Diabetic Nephropathy by Promoting M2 Macrophage Differentiation

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Pentraxin-3 Attenuates Renal Damage in Diabetic Nephropathy by Promoting M2 Macrophage Differentiation is …
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scholarly articleQ13442814

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P6179Dimensions Publication ID1011353757
P356DOI10.1007/S10753-015-0151-Z
P698PubMed publication ID25761429

P2093author name stringJun Tian
Xiangdong Yang
Tingting Xie
Huaibin Sun
Wanhua Xian
P2860cites workDetermination of physiological plasma pentraxin 3 (PTX3) levels in healthy populationsQ46071688
Pentraxin 3 protects from MCMV infection and reactivation through TLR sensing pathways leading to IRF3 activationQ56942879
PPARγ Activation Primes Human Monocytes into Alternative M2 Macrophages with Anti-inflammatory PropertiesQ57244208
Ex vivo programmed macrophages ameliorate experimental chronic inflammatory renal diseaseQ59391845
The relationship between albuminuria, MCP-1/CCL2, and interstitial macrophages in chronic kidney diseaseQ61863531
Preventive effect of sulphated colominic acid on P-selectin-dependent infiltration of macrophages in experimentally induced crescentic glomerulonephritisQ74417583
CSF-1 signaling mediates recovery from acute kidney injuryQ28279068
SIRPα interacts with nephrin at the podocyte slit diaphragmQ28565762
Alternative activation of macrophages: an immunologic functional perspectiveQ29614353
MicroRNA-29a promotion of nephrin acetylation ameliorates hyperglycemia-induced podocyte dysfunctionQ33974555
Associations of pentraxin-3 with cardiovascular events, incident heart failure, and mortality among persons with coronary heart disease: data from the Heart and Soul StudyQ34150807
Pentraxins at the crossroads between innate immunity, inflammation, matrix deposition, and female fertilityQ34403569
Distinct macrophage phenotypes contribute to kidney injury and repairQ34534902
IL-25 induces M2 macrophages and reduces renal injury in proteinuric kidney diseaseQ35108863
Inverse relationship between the inflammatory marker pentraxin-3, fat body mass, and abdominal obesity in end-stage renal diseaseQ35664554
Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular diseaseQ35698969
Macrophages and the kidneyQ35741778
Fibrotic disease and the T(H)1/T(H)2 paradigmQ35852270
Macrophage skewing by Phd2 haplodeficiency prevents ischaemia by inducing arteriogenesisQ36319395
eNOS deficiency predisposes podocytes to injury in diabetes.Q36352132
Novel links between the long pentraxin 3, endothelial dysfunction, and albuminuria in early and advanced chronic kidney diseaseQ36736700
Macrophage diversity in renal injury and repair.Q36955562
Reversibility of structural and functional damage in a model of advanced diabetic nephropathyQ36977327
The pathogenesis of diabetic nephropathyQ37209550
Evaluation of the association of plasma pentraxin 3 levels with type 2 diabetes and diabetic nephropathy in a Malay populationQ37370584
Recent insights in inflammation-associated wasting in patients with chronic kidney diseaseQ37882287
Long pentraxin 3 (PTX3) in the light of its structure, mechanism of action and clinical implicationsQ37944361
The role of non-coding RNAs in diabetic nephropathy: potential applications as biomarkers for disease development and progressionQ38055945
Recent advances in understanding the biochemical and molecular mechanism of diabetic nephropathyQ38094415
Plasma PTX3 protein levels inversely correlate with insulin secretion and obesity, whereas visceral adipose tissue PTX3 gene expression is increased in obesityQ39477901
Protective effect of the long pentraxin PTX3 against histone-mediated endothelial cell cytotoxicity in sepsisQ41737016
mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in miceQ41912848
Plasma pentraxin 3 in patients with chronic kidney disease: associations with renal function, protein-energy wasting, cardiovascular disease, and mortalityQ44571381
P433issue5
P921main subjectmacrophageQ184204
diabetic nephropathyQ1129105
P304page(s)1739-1747
P577publication date2015-03-13
P1433published inInflammationQ2560341
P1476titlePentraxin-3 Attenuates Renal Damage in Diabetic Nephropathy by Promoting M2 Macrophage Differentiation
P478volume38

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cites work (P2860)
Q88634530Aerobic fitness alters the capacity of mononuclear cells to produce pentraxin 3 following maximal exercise
Q50962850Aqueous humor pentraxin-3 levels in patients with diabetes mellitus.
Q47148726Attenuation of partial unilateral ureteral obstruction - induced renal damage with hyperbaric oxygen therapy in a rat model
Q47216784Biochemical and Clinical Impact of Organic Uremic Retention Solutes: A Comprehensive Update
Q38380295Effects of 6 months of resveratrol versus placebo on pentraxin 3 in patients with type 2 diabetes mellitus: a double-blind randomized controlled trial
Q48036999Macrophage Cyclooxygenase-2 Protects Against Development of Diabetic Nephropathy
Q92208806Overexpression of Sirt6 promotes M2 macrophage transformation, alleviating renal injury in diabetic nephropathy
Q99556551Pentraxin 3 promotes airway inflammation in experimental asthma
Q52761490Relationship between plasma pentraxin 3 level and risk of chronic kidney disease in the Korean elderly: the Dong-gu study.
Q88035415Role of the Immune System in Diabetic Kidney Disease
Q90059408Roles of pattern recognition receptors in diabetic nephropathy
Q55447799Study on Association of Pentraxin 3 and Diabetic Nephropathy in a Rat Model.
Q37733630The Potential Role of Aerobic Exercise-Induced Pentraxin 3 on Obesity-Related Inflammation and Metabolic Dysregulation

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