scholarly article | Q13442814 |
P50 | author | Christiane Richter-Landsberg | Q21256969 |
P2093 | author name string | Anwar Norazit | |
Adrian C B Meedeniya | |||
Dean L Pountney | |||
Wei Ping Gai | |||
Jacob Goodwin | |||
Mathew B Wong | |||
P2860 | cites work | Nix is a selective autophagy receptor for mitochondrial clearance | Q22001532 |
Association of metallothionein-III with oligodendroglial cytoplasmic inclusions in multiple system atrophy | Q24293470 | ||
The deacetylase HDAC6 regulates aggresome formation and cell viability in response to misfolded protein stress | Q24301895 | ||
Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice | Q24303752 | ||
p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy | Q24312147 | ||
Histone deacetylase 6 interacts with the microtubule-associated protein tau | Q24315060 | ||
A role for NBR1 in autophagosomal degradation of ubiquitinated substrates | Q24316135 | ||
BAG5 inhibits parkin and enhances dopaminergic neuron degeneration | Q24322908 | ||
LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing | Q24597817 | ||
The molecular chaperone Hsp90 plays a role in the assembly and maintenance of the 26S proteasome | Q24681597 | ||
Protein Aggregates Are Recruited to Aggresome by Histone Deacetylase 6 via Unanchored Ubiquitin C Termini | Q27675460 | ||
The SUMO pathway: emerging mechanisms that shape specificity, conjugation and recognition | Q27865256 | ||
Progressive supranuclear palsy: clinicopathological concepts and diagnostic challenges | Q28235853 | ||
Review: autophagy in neurodegeneration: firefighter and/or incendiarist? | Q28249911 | ||
Aggresome formation and neurodegenerative diseases: therapeutic implications | Q28265926 | ||
Mutual interactions between the SUMO and ubiquitin systems: a plea of no contest | Q28269305 | ||
Parkin-mediated K63-linked polyubiquitination targets misfolded DJ-1 to aggresomes via binding to HDAC6 | Q28397050 | ||
Microautophagy of cytosolic proteins by late endosomes | Q28509224 | ||
p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death | Q29547297 | ||
Protein modification by SUMO | Q29547919 | ||
PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy | Q29616005 | ||
HDAC6 controls autophagosome maturation essential for ubiquitin-selective quality-control autophagy | Q30014829 | ||
Selective molecular alterations in the autophagy pathway in patients with Lewy body disease and in models of alpha-synucleinopathy | Q33670282 | ||
Brain-permeable small-molecule inhibitors of Hsp90 prevent alpha-synuclein oligomer formation and rescue alpha-synuclein-induced toxicity | Q33714875 | ||
Tau-positive glial inclusions in progressive supranuclear palsy, corticobasal degeneration and Pick's disease | Q33750054 | ||
Parkin-mediated ubiquitin signalling in aggresome formation and autophagy | Q33757729 | ||
Elevated global SUMOylation in Ubc9 transgenic mice protects their brains against focal cerebral ischemic damage | Q34053347 | ||
Chaperone-mediated autophagy at a glance | Q34161740 | ||
HDAC6 at the intersection of autophagy, the ubiquitin-proteasome system and neurodegeneration | Q34697419 | ||
Proteasome inhibition induces α-synuclein SUMOylation and aggregate formation | Q35085788 | ||
Convergence of heat shock protein 90 with ubiquitin in filamentous alpha-synuclein inclusions of alpha-synucleinopathies | Q35088093 | ||
Sumoylation inhibits alpha-synuclein aggregation and toxicity. | Q35102441 | ||
CHIP: a link between the chaperone and proteasome systems | Q35759085 | ||
Sumoylation in axons triggers retrograde transport of the RNA-binding protein La. | Q35921738 | ||
SUMO on the road to neurodegeneration | Q36809376 | ||
Tau protein in the glial cytoplasmic inclusions of multiple system atrophy can be distinguished from abnormal tau in Alzheimer's disease. | Q36876676 | ||
Emerging extranuclear roles of protein SUMOylation in neuronal function and dysfunction | Q36995104 | ||
Multiple system atrophy: a primary oligodendrogliopathy. | Q37282526 | ||
Recent developments in multiple system atrophy. | Q37496792 | ||
Protein SUMOylation in neuropathological conditions. | Q37553976 | ||
Identification of a SUMO-binding motif that recognizes SUMO-modified proteins | Q37569061 | ||
Chaperone-assisted degradation: multiple paths to destruction | Q37714916 | ||
Targets and consequences of protein SUMOylation in neurons. | Q37728628 | ||
Membrane lipid modification by docosahexaenoic acid (DHA) promotes the formation of α-synuclein inclusion bodies immunopositive for SUMO-1 in oligodendroglial cells after oxidative stress | Q39665306 | ||
Alpha-synuclein aggregation and Ser-129 phosphorylation-dependent cell death in oligodendroglial cells. | Q39885727 | ||
Functional modulation of parkin through physical interaction with SUMO-1. | Q40235074 | ||
Dual role of sumoylation in the nuclear localization and transcriptional activation of NFAT1. | Q40561279 | ||
Pathological inclusion bodies in tauopathies contain distinct complements of tau with three or four microtubule-binding repeat domains as demonstrated by new specific monoclonal antibodies | Q40642506 | ||
CHIP is a chaperone-dependent E3 ligase that ubiquitylates unfolded protein | Q41776375 | ||
Proteomic analysis of enriched lysosomes at early phase of camptothecin-induced apoptosis in human U-937 cells | Q41868050 | ||
Defining the SUMO-modified proteome by multiple approaches in Saccharomyces cerevisiae | Q42642323 | ||
Progressive loss of dopaminergic neurons induced by unilateral rotenone infusion into the medial forebrain bundle | Q42921156 | ||
Alpha-synuclein-enhanced green fluorescent protein fusion proteins form proteasome sensitive inclusions in primary neurons | Q43664472 | ||
Modulation of polyglutamine-induced cell death by genes identified by expression profiling | Q44129405 | ||
Involvement of macroautophagy in multiple system atrophy and protein aggregate formation in oligodendrocytes | Q44539585 | ||
SUMO-1 marks the nuclear inclusions in familial neuronal intranuclear inclusion disease | Q44668534 | ||
The KDEL receptor induces autophagy to promote the clearance of neurodegenerative disease-related proteins | Q45183772 | ||
Multiple-system atrophy: a new alpha-synuclein disease? | Q48406192 | ||
Alterations in lysosomal and proteasomal markers in Parkinson's disease: relationship to alpha-synuclein inclusions | Q48581414 | ||
NSF, Unc-18-1, dynamin-1 and HSP90 are inclusion body components in neuronal intranuclear inclusion disease identified by anti-SUMO-1-immunocapture | Q48892535 | ||
SUMO-1 marks subdomains within glial cytoplasmic inclusions of multiple system atrophy | Q48894652 | ||
NBR1 and p62 as cargo receptors for selective autophagy of ubiquitinated targets | Q57269938 | ||
A Proteomic Strategy for Gaining Insights into Protein Sumoylation in Yeast | Q57942267 | ||
A Simple Procedure for the Isolation of Highly Purified Lysosomes from Normal Rat Liver | Q72385985 | ||
P433 | issue | 1 | |
P304 | page(s) | 1-21 | |
P577 | publication date | 2012-11-15 | |
P1433 | published in | Neurotoxicity Research | Q15764278 |
P1476 | title | SUMO-1 is associated with a subset of lysosomes in glial protein aggregate diseases | |
P478 | volume | 23 |
Q48314813 | Altered lysosome distribution is an early neuropathological event in neurological forms of Gaucher disease |
Q36639242 | Autophagic and lysosomal defects in human tauopathies: analysis of post-mortem brain from patients with familial Alzheimer disease, corticobasal degeneration and progressive supranuclear palsy |
Q26799306 | Direct and/or Indirect Roles for SUMO in Modulating Alpha-Synuclein Toxicity |
Q41370372 | Lysine-Directed Post-translational Modifications of Tau Protein in Alzheimer's Disease and Related Tauopathies |
Q37566673 | Multiple system atrophy: the application of genetics in understanding etiology |
Q26771536 | Neuroinflammation in Multiple System Atrophy: Response to and Cause of α-Synuclein Aggregation |
Q43679571 | Neurotoxin mechanisms and processes relevant to Parkinson's disease: an update |
Q38851366 | Post-translational protein arginylation in the normal nervous system and in neurodegeneration |
Q38131751 | SUMO and Parkinson's disease |
Q38757045 | SUMO-regulated mitochondrial function in Parkinson's disease |
Q97884803 | SUMOylation and calcium signalling: potential roles in the brain and beyond |
Q61795969 | SUMOylation by SUMO2 is implicated in the degradation of misfolded ataxin-7 via RNF4 in SCA7 models |
Q97516927 | SUMOylation in α-Synuclein Homeostasis and Pathology |
Q58705758 | SUMOylation of Alpha-Synuclein Influences on Alpha-Synuclein Aggregation Induced by Methamphetamine |
Q26783370 | SUMOylation: Novel Neuroprotective Approach for Alzheimer's Disease? |
Q50676884 | Spatiotemporal distribution of SUMOylation components during mouse brain development. |
Q42066873 | Sumo E2 enzyme UBC9 is required for efficient protein quality control in cardiomyocytes |
Q28081548 | Synthetic Proteins and Peptides for the Direct Interrogation of α-Synuclein Posttranslational Modifications |
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