| scholarly article | Q13442814 |
| P2093 | author name string | Yang Liu | |
| Michael S Wolfe | |||
| Lilia Rodriguez | |||
| Eleanor Peacey | |||
| P2860 | cites work | 5' splice site mutations in tau associated with the inherited dementia FTDP-17 affect a stem-loop structure that regulates alternative splicing of exon 10. | Q52535062 |
| A phosphorothioate oligonucleotide blocks reverse transcription via an antisense mechanism | Q71614263 | ||
| Role of RNA structure in regulating pre-mRNA splicing | Q24631714 | ||
| Enhanced RNA cleavage within bulge-loops by an artificial ribonuclease | Q24793857 | ||
| Variation in alternative splicing across human tissues | Q24809417 | ||
| Progress toward therapy with antisense-mediated splicing modulation | Q27489807 | ||
| Structure of tau exon 10 splicing regulatory element RNA and destabilization by mutations of frontotemporal dementia and parkinsonism linked to chromosome 17 | Q27618950 | ||
| Structural Basis for Stabilization of the Tau Pre-mRNA Splicing Regulatory Element by Novantrone (Mitoxantrone) | Q27655658 | ||
| Alternative isoform regulation in human tissue transcriptomes | Q27861118 | ||
| Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17 | Q28274687 | ||
| Tau-mediated neurodegeneration in Alzheimer's disease and related disorders | Q29618148 | ||
| The mutational spectrum of single base-pair substitutions in mRNA splice junctions of human genes: causes and consequences | Q29619441 | ||
| Eukaryotic gene transcription with purified components | Q29620213 | ||
| Aberrant splicing of tau pre-mRNA caused by intronic mutations associated with the inherited dementia frontotemporal dementia with parkinsonism linked to chromosome 17. | Q33963856 | ||
| RNA targeting therapeutics: molecular mechanisms of antisense oligonucleotides as a therapeutic platform | Q34020786 | ||
| Splicing of a critical exon of human Survival Motor Neuron is regulated by a unique silencer element located in the last intron | Q34353625 | ||
| RNA structure and the mechanisms of alternative splicing. | Q35146258 | ||
| A G-rich element forms a G-quadruplex and regulates BACE1 mRNA alternative splicing | Q35928555 | ||
| Mutation in the tau gene in familial multiple system tauopathy with presenile dementia | Q36507684 | ||
| Mechanisms of the inhibition of reverse transcription by antisense oligonucleotides | Q36798960 | ||
| Delivery of bifunctional RNAs that target an intronic repressor and increase SMN levels in an animal model of spinal muscular atrophy | Q37154271 | ||
| Tau mutations in neurodegenerative diseases | Q37307240 | ||
| Correction of alternative splicing of tau in frontotemporal dementia and parkinsonism linked to chromosome 17. | Q38296901 | ||
| Effects of RNA secondary structure on cellular antisense activity | Q38315200 | ||
| Mechanisms of the inhibition of reverse transcription by unmodified and modified antisense oligonucleotides | Q38316118 | ||
| Selective inhibition of cell-free translation by oligonucleotides targeted to a mRNA hairpin structure. | Q38336876 | ||
| Stabilization of the tau exon 10 stem loop alters pre-mRNA splicing | Q39958205 | ||
| Antisense 2'-O-alkyl oligoribonucleotides are efficient inhibitors of reverse transcription | Q40392535 | ||
| Mitoxantrone analogues as ligands for a stem-loop structure of tau pre-mRNA. | Q41808723 | ||
| P275 | copyright license | Creative Commons Attribution-NonCommercial 3.0 Unported | Q18810331 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 19 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | alternative mRNA splicing, via spliceosome | Q21114084 |
| P304 | page(s) | 9836-9849 | |
| P577 | publication date | 2012-07-25 | |
| P1433 | published in | Nucleic Acids Research | Q135122 |
| P1476 | title | Targeting a pre-mRNA structure with bipartite antisense molecules modulates tau alternative splicing | |
| P478 | volume | 40 |
| Q30867681 | Ametantrone-based compounds as potential regulators of Tau pre-mRNA alternative splicing |
| Q27007056 | Antisense oligonucleotides: treating neurodegeneration at the level of RNA |
| Q38184276 | Antisense-mediated exon skipping: taking advantage of a trick from Mother Nature to treat rare genetic diseases |
| Q38211673 | Development of therapeutic splice-switching oligonucleotides. |
| Q45870549 | Emerging Diagnostic and Therapeutic Strategies for Tauopathies |
| Q38186137 | Exon-skipping antisense oligonucleotides to correct missplicing in neurogenetic diseases |
| Q41705374 | HIV-1 splicing at the major splice donor site is restricted by RNA structure |
| Q33786251 | Identification of a Peptide for Systemic Brain Delivery of a Morpholino Oligonucleotide in Mouse Models of Spinal Muscular Atrophy |
| Q38263698 | Invited review: Drug development for tauopathies |
| Q90743959 | Nuclear and Cytoplasmatic Quantification of Unconjugated, Label-Free Locked Nucleic Acid Oligonucleotides |
| Q50130748 | Nucleic Acid-Based Theranostics for Tackling Alzheimer's Disease |
| Q92753261 | RNA Secondary Structure-Based Design of Antisense Peptide Nucleic Acids for Modulating Disease-Associated Aberrant Tau Pre-mRNA Alternative Splicing |
| Q93355206 | RNA Splicing and Disease: Animal Models to Therapies |
| Q38242065 | RNA triplexes: from structural principles to biological and biotech applications |
| Q47655684 | Sequence-specific and Selective Recognition of Double-stranded RNAs over Single-stranded RNAs by Chemically Modified Peptide Nucleic Acids |
| Q37211004 | Splice-switching antisense oligonucleotides as therapeutic drugs |
| Q36780738 | Targeting RNA splicing for disease therapy |
| Q27687153 | Targeting mRNA for Alzheimer's and related dementias. |
| Q33835923 | Template-directed synthesis of a small molecule-antisense conjugate targeting an mRNA structure |
| Q28119005 | The Role of MAPT in Neurodegenerative Diseases: Genetics, Mechanisms and Therapy |
| Q27005032 | The role of tau in neurodegenerative diseases and its potential as a therapeutic target |
| Q39185494 | Trans-splicing correction of tau isoform imbalance in a mouse model of tau mis-splicing |
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