| scholarly article | Q13442814 |
| P50 | author | Tito Calì | Q47503101 |
| Alessandro Negro | Q88717507 | ||
| Denis Ottolini | Q114399013 | ||
| Marisa Brini | Q39956417 | ||
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| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 22 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | mitochondrion | Q39572 |
| endoplasmic reticulum | Q79927 | ||
| Synuclein | Q24767155 | ||
| P304 | page(s) | 17914-17929 | |
| P577 | publication date | 2012-03-27 | |
| P1433 | published in | Journal of Biological Chemistry | Q867727 |
| P1476 | title | α-Synuclein controls mitochondrial calcium homeostasis by enhancing endoplasmic reticulum-mitochondria interactions | |
| P478 | volume | 287 |
| Q51770889 | A Futile Battle? Protein Quality Control and the Stress of Aging. |
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| Q27021628 | Ageing and Parkinson's disease: why is advancing age the biggest risk factor? |
| Q92220037 | Alpha-Synuclein Preserves Mitochondrial Fusion and Function in Neuronal Cells |
| Q51732455 | Alpha-synuclein aggregates activate calcium pump SERCA leading to calcium dysregulation. |
| Q41962173 | Autophagy, a novel pathway to regulate calcium mobilization in T lymphocytes |
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| Q39075636 | Calcium: Alpha-Synuclein Interactions in Alpha-Synucleinopathies |
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| Q92570368 | Causative Links between Protein Aggregation and Oxidative Stress: A Review |
| Q38915163 | Coherent and Contradictory Facts, Feats and Fictions Associated with Metal Accumulation in Parkinson's Disease: Epicenter or Outcome, Yet a Demigod Question |
| Q42255446 | Connecting Ca2+ and lysosomes to Parkinson disease |
| Q28074143 | Converging roles of ion channels, calcium, metabolic stress, and activity pattern of Substantia nigra dopaminergic neurons in health and Parkinson's disease |
| Q58994518 | Crosstalk between Endoplasmic Reticulum Stress and Protein Misfolding in Neurodegenerative Diseases |
| Q43100153 | DISC1-dependent Regulation of Mitochondrial Dynamics Controls the Morphogenesis of Complex Neuronal Dendrites |
| Q92811394 | Dysregulated Interorganellar Crosstalk of Mitochondria in the Pathogenesis of Parkinson's Disease |
| Q28071890 | Dysregulation of autophagy and mitochondrial function in Parkinson's disease |
| Q38988714 | ER-mitochondria associations are regulated by the VAPB-PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43. |
| Q54987116 | ER-mitochondria signaling in Parkinson's disease. |
| Q90139291 | Endoplasmic Reticulum-Mitochondrial Cross-Talk in Neurodegenerative and Eye Diseases |
| Q41494591 | Endoplasmic reticulum-mitochondria tethering in neurodegenerative diseases |
| Q35073958 | Environmental neurotoxic challenge of conditional alpha-synuclein transgenic mice predicts a dopaminergic olfactory-striatal interplay in early PD. |
| Q40707464 | Hereditary spastic paraplegia-linked REEP1 modulates endoplasmic reticulum/mitochondria contacts |
| Q34497765 | Higher vulnerability and stress sensitivity of neuronal precursor cells carrying an alpha-synuclein gene triplication |
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| Q54224883 | Impact of membrane curvature on amyloid aggregation. |
| Q47552497 | Induction of alpha-synuclein pathology in the enteric nervous system of the rat and non-human primate results in gastrointestinal dysmotility and transient CNS pathology |
| Q90072341 | Inter-Organelle Membrane Contact Sites and Mitochondrial Quality Control during Aging: A Geroscience View |
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| Q39287167 | Linking mitochondrial dysfunction to neurodegeneration in lysosomal storage diseases. |
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| Q38974698 | Mitochondria-Associated Membranes (MAMs): Overview and Its Role in Parkinson's Disease |
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| Q36095330 | Mitochondrial Ca(2+) and neurodegeneration |
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| Q99635257 | Neuroprotective Properties of Green Tea (Camellia sinensis) in Parkinson's Disease: A Review |
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| Q90161791 | PINK1/Parkin Mediated Mitophagy, Ca2+ Signalling, and ER-Mitochondria Contacts in Parkinson's Disease |
| Q38152234 | Perturbed mitochondrial Ca2+ signals as causes or consequences of mitophagy induction |
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| Q91637190 | Prion-Like Mechanisms in Parkinson's Disease |
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| Q59128094 | Reduced Cytosolic Calcium as an Early Decisive Cellular State in Parkinson's Disease and Synucleinopathies |
| Q90238537 | Restriction of mitochondrial calcium overload by mcu inactivation renders a neuroprotective effect in zebrafish models of Parkinson's disease |
| Q41714466 | Roles of sigma-1 receptors on mitochondrial functions relevant to neurodegenerative diseases |
| Q38757045 | SUMO-regulated mitochondrial function in Parkinson's disease |
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| Q33985326 | Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunction. |
| Q55261587 | The Close Encounter Between Alpha-Synuclein and Mitochondria. |
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| Q38374332 | The Parkinson Disease Mitochondrial Hypothesis: Where Are We at? |
| Q48158987 | The Parkinson disease-related protein DJ-1 counteracts mitochondrial impairment induced by the tumour suppressor protein p53 by enhancing endoplasmic reticulum-mitochondria tethering |
| Q28068464 | The contribution of alpha synuclein to neuronal survival and function - Implications for Parkinson's disease |
| Q39748670 | The endoplasmic reticulum-mitochondria interface is perturbed in PARK2 knockout mice and patients with PARK2 mutations |
| Q91303015 | The functional universe of membrane contact sites |
| Q47646796 | The genetic architecture of mitochondrial dysfunction in Parkinson's disease |
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| Q64859135 | The unlikely partnership between LRRK2 and α-synuclein in Parkinson's disease |
| Q26768635 | There's Something Wrong with my MAM; the ER-Mitochondria Axis and Neurodegenerative Diseases |
| Q36372732 | Understanding the susceptibility of dopamine neurons to mitochondrial stressors in Parkinson's disease |
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| Q38077612 | What's to like about the prion-like hypothesis for the spreading of aggregated α-synuclein in Parkinson disease? |
| Q52671146 | Wolfram syndrome: MAMs' connection? |
| Q90230159 | splitGFP Technology Reveals Dose-Dependent ER-Mitochondria Interface Modulation by α-Synuclein A53T and A30P Mutants |
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| Q40986110 | αSynuclein control of mitochondrial homeostasis in human-derived neurons is disrupted by mutations associated with Parkinson's disease. |
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