| scholarly article | Q13442814 |
| P50 | author | Zhiren Zhang | Q56027888 |
| P2093 | author name string | Jia Li | |
| Yu Liu | |||
| Jinsong Wang | |||
| Kai Xu | |||
| Zigang Shen | |||
| Zhi-Yuan Zhang | |||
| Man Jiang | |||
| Zongwei Liu | |||
| Bangwei Luo | |||
| Fuyu Han | |||
| P2860 | cites work | Guillain-Barré syndrome | Q24276200 |
| Anti-inflammatory therapy in chronic disease: challenges and opportunities | Q26860162 | ||
| Novel docosatrienes and 17S-resolvins generated from docosahexaenoic acid in murine brain, human blood, and glial cells. Autacoids in anti-inflammation | Q28210028 | ||
| The inhibition of cutaneous T cell apoptosis may prevent resolution of inflammation in atopic eczema | Q57181368 | ||
| Resolvin D1 and Its Precursor Docosahexaenoic Acid Promote Resolution of Adipose Tissue Inflammation by Eliciting Macrophage Polarization toward an M2-Like Phenotype | Q61575249 | ||
| Apoptotic cell death of T-lymphocytes in experimental autoimmune neuritis of the Lewis rat | Q72830219 | ||
| A study of associated cell-mediated immune mechanisms in experimental autoimmune neuritis rats | Q79904541 | ||
| Anti-cytokine autoantibodies in experimental autoimmune neuritis in Lewis rats | Q80973484 | ||
| Crucial role of apoptosis in the resolution of experimental autoimmune anterior uveitis | Q81503644 | ||
| Regulatory T cells in rheumatoid arthritis | Q82076756 | ||
| TGF-β regulates hepatocellular carcinoma progression by inducing Treg cell polarization | Q87021946 | ||
| Resolvin D1 and its aspirin-triggered 17R epimer. Stereochemical assignments, anti-inflammatory properties, and enzymatic inactivation | Q28218126 | ||
| Resolvin D1 binds human phagocytes with evidence for proresolving receptors | Q28270638 | ||
| Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation | Q28344659 | ||
| Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF | Q29615685 | ||
| Anti-inflammatory T-cell shift in neuropathic pain | Q30420222 | ||
| Erythropoietin ameliorates rat experimental autoimmune neuritis by inducing transforming growth factor-β in macrophages | Q34063671 | ||
| Attenuated EAN in TNF-α deficient mice is associated with an altered balance of M1/M2 macrophages | Q34292861 | ||
| From Monocytes to M1/M2 Macrophages: Phenotypical vs. Functional Differentiation | Q34359869 | ||
| Effect of transgenic overexpression of FLIP on lymphocytes on development and resolution of experimental autoimmune thyroiditis | Q35168021 | ||
| FoxP3+ regulatory T cells determine disease severity in rodent models of inflammatory neuropathies | Q35305030 | ||
| Apoptotic neutrophils and T cells sequester chemokines during immune response resolution through modulation of CCR5 expression | Q35633812 | ||
| TGF-beta: the perpetrator of immune suppression by regulatory T cells and suicidal T cells | Q35668744 | ||
| Myelin-specific regulatory T cells accumulate in the CNS but fail to control autoimmune inflammation. | Q36189566 | ||
| Regulation of CD8+ regulatory T cells: Interruption of the NKG2A-Qa-1 interaction allows robust suppressive activity and resolution of autoimmune disease | Q37039579 | ||
| Th17 Cells and autoimmune encephalomyelitis (EAE/MS). | Q37143735 | ||
| The phenotype of human Th17 cells and their precursors, the cytokines that mediate their differentiation and the role of Th17 cells in inflammation. | Q37280352 | ||
| Resolvin D1 controls inflammation initiated by glutathione-lipid conjugates formed during oxidative stress | Q37447489 | ||
| Phagocytic clearance of apoptotic cells: role in lung disease | Q37640810 | ||
| Resolvins and protectins: natural pharmacophores for resolution biology | Q37708842 | ||
| Autoimmune neuropathies: insights from animal models | Q38006760 | ||
| Resolvin D1 and its GPCRs in resolution circuits of inflammation | Q38090253 | ||
| Current and prospective pharmacotherapy for autoimmune hepatitis | Q38222882 | ||
| Docosahexaenoic acid induces M2 macrophage polarization through peroxisome proliferator-activated receptor γ activation | Q38935967 | ||
| Mitigated Tregs and augmented Th17 cells and cytokines are associated with severity of experimental autoimmune neuritis | Q39178621 | ||
| Therapeutic potential of atorvastatin-modified dendritic cells in experimental autoimmune neuritis by decreased Th1/Th17 cytokines and up-regulated T regulatory cells and NKR-P1(+) cells | Q39245587 | ||
| Atorvastatin ameliorates experimental autoimmune neuritis by decreased Th1/Th17 cytokines and up-regulated T regulatory cells | Q39711062 | ||
| Compound A, a plant origin ligand of glucocorticoid receptors, increases regulatory T cells and M2 macrophages to attenuate experimental autoimmune neuritis with reduced side effects | Q39813329 | ||
| Adoptive transfer-experimental allergic neuritis in newborn Lewis rats results in inflammatory infiltrates, mast cell activation, and increased Ia expression with only minor nerve fiber degeneration | Q40693125 | ||
| Statin attenuates experimental anti-glomerular basement membrane glomerulonephritis together with the augmentation of alternatively activated macrophages | Q42045387 | ||
| Disclosure of the Culprits: Macrophages-Versatile Regulators of Wound Healing | Q42862281 | ||
| MS-275, an histone deacetylase inhibitor, reduces the inflammatory reaction in rat experimental autoimmune neuritis | Q43069570 | ||
| Distribution of Foxp3(+) T-regulatory cells in experimental autoimmune neuritis rats | Q43663214 | ||
| Glucocorticosteroids modulate antigen-induced T cell apoptosis in experimental autoimmune neuritis and cause T cell proliferation in situ | Q43731162 | ||
| Astrocytic Fas ligand expression is required to induce T-cell apoptosis and recovery from experimental autoimmune encephalomyelitis | Q44758785 | ||
| Distinct expression of Tim-3 during different stages of rat experimental autoimmune neuritis | Q45819359 | ||
| IL-10, IL-4, and STAT6 promote an M2 milieu required for termination of P0(106-125)-induced murine experimental autoimmune neuritis. | Q50847670 | ||
| Synovial lymphoid neogenetic factors in Behçet's synovitis: do they play a role in self-limiting and subacute course of arthritis? | Q53517057 | ||
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 37 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | inflammation | Q101991 |
| resolvin D1 | Q60998703 | ||
| P304 | page(s) | 9590-9603 | |
| P577 | publication date | 2016-09-01 | |
| P1433 | published in | Journal of Neuroscience | Q1709864 |
| P1476 | title | Resolvin D1 Programs Inflammation Resolution by Increasing TGF-β Expression Induced by Dying Cell Clearance in Experimental Autoimmune Neuritis | |
| P478 | volume | 36 |
| Q93378528 | Eicosanoid and Specialized Proresolving Mediator Regulation of Lymphoid Cells |
| Q38718096 | Fundamentals of and Critical Issues in Lipid Autacoid Medicine: A Review |
| Q64100827 | In vitro and in vivo assessment of the proresolutive and antiresorptive actions of resolvin D1: relevance to arthritis |
| Q98465727 | M1 Macrophage Derived Exosomes Aggravate Experimental Autoimmune Neuritis via Modulating Th1 Response |
| Q61445165 | Pro-resolving lipid mediator ameliorates obesity induced osteoarthritis by regulating synovial macrophage polarisation |
| Q41233563 | Resolvin D1 Increases Mucin Secretion in Cultured Rat Conjunctival Goblet Cells via Multiple Signaling Pathways |
| Q92132148 | Resolvin D1 Protects Against Ischemia/Reperfusion-Induced Acute Kidney Injury by Increasing Treg Percentages via the ALX/FPR2 Pathway |
| Q37743273 | Resolvin D1 drives establishment of Leishmania amazonensis infection |
| Q54974225 | Resolvin D1 promotes corneal epithelial wound healing and restoration of mechanical sensation in diabetic mice. |
| Q95840704 | Role of Resolvins in the Inflammatory Resolution of Neurological Diseases |
| Q64923754 | The Resolution of Intestinal Inflammation: The Peace-Keeper's Perspective. |
Search more.