scholarly article | Q13442814 |
P50 | author | Gabriel Rabinovich | Q17987510 |
Mercedes Fuertes | Q37841931 | ||
Norberto Zwirner | Q42880727 | ||
Raúl German Spallanzani | Q49191851 | ||
Carolina I Domaica | Q56166792 | ||
Diego O Croci | Q130239211 | ||
P2093 | author name string | Lucas E Rossi | |
Andrea Ziblat | |||
Lara Lapyckyj | |||
Damián E Avila | |||
P2860 | cites work | Histone acetyltransferases | Q29547823 |
Functions of natural killer cells | Q29614300 | ||
NK cell recognition | Q29615107 | ||
Histone deacetylase inhibitors and the promise of epigenetic (and more) treatments for cancer | Q29615649 | ||
Anticancer activities of histone deacetylase inhibitors | Q29616624 | ||
Up on the tightrope: natural killer cell activation and inhibition | Q29619175 | ||
Role of thioredoxin in the response of normal and transformed cells to histone deacetylase inhibitors | Q33697130 | ||
Natural cytotoxic activity of peripheral-blood lymphocytes and cancer incidence: an 11-year follow-up study of a general population | Q33928216 | ||
Polycystic ovarian syndrome in women with epilepsy: a review | Q34345051 | ||
Histone deacetylase inhibitors in cancer therapy | Q34659283 | ||
Reduced natural killer (NK) function associated with high-risk myelodysplastic syndrome (MDS) and reduced expression of activating NK receptors | Q35828771 | ||
NKG2D function protects the host from tumor initiation | Q36402828 | ||
NKG2D-deficient mice are defective in tumor surveillance in models of spontaneous malignancy | Q36484325 | ||
Surface NK receptors and their ligands on tumor cells | Q36490899 | ||
Antiepileptic drugs in non-epilepsy disorders: relations between mechanisms of action and clinical efficacy | Q37029357 | ||
Histone deacetylase inhibitor suberoylanilide hydroxamic acid reduces acute graft-versus-host disease and preserves graft-versus-leukemia effect | Q37095317 | ||
Drug Insight: histone deacetylase inhibitor-based therapies for cutaneous T-cell lymphomas | Q37288403 | ||
Histone deacetylase inhibitors: a new perspective for the treatment of leukemia | Q37599067 | ||
Distinct roles of IL-12 and IL-15 in human natural killer cell activation by dendritic cells from secondary lymphoid organs | Q37692292 | ||
Cytokine regulation of natural killer cell effector functions | Q37771915 | ||
Tumor immunoediting by NKp46. | Q39713057 | ||
Histone deacetylase inhibitors prevent activation of tumour-reactive NK cells and T cells but do not interfere with their cytolytic effector functions | Q39722163 | ||
Histone deacetylase inhibitors sensitize tumour cells for cytotoxic effects of natural killer cells | Q39948537 | ||
Intracellular retention of the NKG2D ligand MHC class I chain-related gene A in human melanomas confers immune privilege and prevents NK cell-mediated cytotoxicity. | Q40000380 | ||
NKG2D ligand expression in AML increases in response to HDAC inhibitor valproic acid and contributes to allorecognition by NK-cell lines with single KIR-HLA class I specificities | Q40053992 | ||
Engagement of TLR3, TLR7, and NKG2D regulate IFN-gamma secretion but not NKG2D-mediated cytotoxicity by human NK cells stimulated with suboptimal doses of IL-12. | Q40084538 | ||
Regulation of the expression of MHC class I-related chain A, B (MICA, MICB) via chromatin remodeling and its impact on the susceptibility of leukemic cells to the cytotoxicity of NKG2D-expressing cells | Q40108262 | ||
Histone deacetylase inhibitors suppress natural killer cell cytolytic activity | Q40161252 | ||
Histone deacetylase inhibitors suppress the inducibility of nuclear factor-kappaB by tumor necrosis factor-alpha receptor-1 down-regulation | Q40278633 | ||
Cancer cells become susceptible to natural killer cell killing after exposure to histone deacetylase inhibitors due to glycogen synthase kinase-3-dependent expression of MHC class I-related chain A and B. | Q40344948 | ||
Butyrate suppression of colonocyte NF-kappa B activation and cellular proteasome activity | Q40778493 | ||
Differential effects of selective HDAC inhibitors on macrophage inflammatory responses to the Toll-like receptor 4 agonist LPS. | Q43143715 | ||
Induction of HDAC2 expression upon loss of APC in colorectal tumorigenesis | Q44897188 | ||
Translocation-positive acute myeloid leukemia associated with valproic acid therapy | Q44926636 | ||
HDAC6 expression is correlated with better survival in breast cancer. | Q45122014 | ||
Cancer risk in long-term users of valproate: a population-based case-control study. | Q45977880 | ||
HDAC3 represses the expression of NKG2D ligands ULBPs in epithelial tumour cells: potential implications for the immunosurveillance of cancer. | Q46017171 | ||
Effective in vivo induction of NKG2D ligands in acute myeloid leukaemias by all-trans-retinoic acid or sodium valproate | Q46158181 | ||
Acute leukemia associated with valproic acid treatment: a novel mechanism for leukemogenesis? | Q46408704 | ||
HDAC inhibition prevents NF-kappa B activation by suppressing proteasome activity: down-regulation of proteasome subunit expression stabilizes I kappa B alpha | Q46541460 | ||
Natural killer cell-mediated lysis of hepatoma cells via specific induction of NKG2D ligands by the histone deacetylase inhibitor sodium valproate | Q46604673 | ||
Deacetylase inhibition promotes the generation and function of regulatory T cells. | Q46961808 | ||
Enhanced in vivo growth of lymphoma tumors in the absence of the NK-activating receptor NKp46/NCR1. | Q51833288 | ||
Histone deacetylase activities are required for innate immune cell control of Th1 but not Th2 effector cell function. | Q52005889 | ||
Autocrine role of endogenous interleukin-18 on inflammatory cytokine generation by human neutrophils. | Q53455289 | ||
P433 | issue | 2 | |
P304 | page(s) | 321-331 | |
P577 | publication date | 2011-11-28 | |
P1433 | published in | Journal of Leukocyte Biology | Q1524048 |
P1476 | title | Histone deacetylase inhibitors impair NK cell viability and effector functions through inhibition of activation and receptor expression | |
P478 | volume | 91 |
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