scholarly article | Q13442814 |
P2093 | author name string | P Darrell Neufer | |
Terence E Ryan | |||
Maria J Torres | |||
Kelsey H Fisher-Wellman | |||
Laura A A Gilliam | |||
Chien-Te Lin | |||
Cody D Smith | |||
Lauren R Reese | |||
P2860 | cites work | Mispairing C57BL/6 substrains of genetically engineered mice and wild-type controls can lead to confounding results as it did in studies of JNK2 in acetaminophen and concanavalin A liver injury | Q24615237 |
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Attention to Background Strain Is Essential for Metabolic Research: C57BL/6 and the International Knockout Mouse Consortium | Q26773196 | ||
Central nervous system control of food intake and body weight | Q29619020 | ||
Reactive oxygen species have a causal role in multiple forms of insulin resistance | Q29619831 | ||
Protein Cysteines Map to Functional Networks According to Steady-state Level of Oxidation | Q30416903 | ||
A comparative phenotypic and genomic analysis of C57BL/6J and C57BL/6N mouse strains | Q30436818 | ||
Diet-induced obesity in two C57BL/6 substrains with intact or mutant nicotinamide nucleotide transhydrogenase (Nnt) gene | Q33925525 | ||
Analysis of GSH and GSSG after derivatization with N-ethylmaleimide | Q34362782 | ||
Defective insulin secretory response to intravenous glucose in C57Bl/6J compared to C57Bl/6N mice | Q34687550 | ||
Selective insulin resistance in adipocytes | Q35560991 | ||
Defects in mitochondrial efficiency and H2O2 emissions in obese women are restored to a lean phenotype with aerobic exercise training | Q35630850 | ||
A Comparative Study of the Metabolic and Skeletal Response of C57BL/6J and C57BL/6N Mice in a Diet-Induced Model of Type 2 Diabetes | Q35749390 | ||
Increased reactive oxygen species production and lower abundance of complex I subunits and carnitine palmitoyltransferase 1B protein despite normal mitochondrial respiration in insulin-resistant human skeletal muscle | Q35757202 | ||
Linking mitochondrial bioenergetics to insulin resistance via redox biology | Q35855378 | ||
Isocitrate-to-SENP1 signaling amplifies insulin secretion and rescues dysfunctional β cells | Q36165625 | ||
Behavioral differences among C57BL/6 substrains: implications for transgenic and knockout studies. | Q36971705 | ||
Selective targeting of the cysteine proteome by thioredoxin and glutathione redox systems. | Q37289421 | ||
Inhibiting myosin-ATPase reveals a dynamic range of mitochondrial respiratory control in skeletal muscle. | Q37393404 | ||
Mitochondrial respiratory capacity and content are normal in young insulin-resistant obese humans | Q37403358 | ||
Mitochondrial glutathione depletion reveals a novel role for the pyruvate dehydrogenase complex as a key H2O2-emitting source under conditions of nutrient overload. | Q37662139 | ||
Nicotinamide nucleotide transhydrogenase: a key role in insulin secretion | Q40332664 | ||
Pyruvate dehydrogenase complex and nicotinamide nucleotide transhydrogenase constitute an energy-consuming redox circuit | Q41026815 | ||
Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans | Q42247172 | ||
Control of expression of insulin resistance and hyperglycemia by different genetic factors in diabetic C57BL/6J mice | Q44016561 | ||
A genetic and physiological study of impaired glucose homeostasis control in C57BL/6J mice | Q45281560 | ||
Diet-induced type II diabetes in C57BL/6J mice | Q45730735 | ||
Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. | Q46588693 | ||
A spontaneous mutation in the nicotinamide nucleotide transhydrogenase gene of C57BL/6J mice results in mitochondrial redox abnormalities. | Q46719392 | ||
Genetic modifiers of the phenotype of mice deficient in mitochondrial superoxide dismutase | Q46960424 | ||
Pathogenesis of pre-diabetes: mechanisms of fasting and postprandial hyperglycemia in people with impaired fasting glucose and/or impaired glucose tolerance. | Q51484657 | ||
Genetic analysis of glucose tolerance in inbred mouse strains. Evidence for polygenic control. | Q51625385 | ||
Impaired Akt phosphorylation in insulin-resistant human muscle is accompanied by selective and heterogeneous downstream defects. | Q54454650 | ||
Determination of reduced and oxidized glutathione in biological samples using liquid chromatography with fluorimetric detection | Q57216716 | ||
Mouse strain-dependent variation in obesity and glucose homeostasis in response to high-fat feeding | Q62888408 | ||
Distinct patterns of tissue-specific lipid accumulation during the induction of insulin resistance in mice by high-fat feeding | Q63372079 | ||
Deletion of Nicotinamide Nucleotide Transhydrogenase: A New Quantitive Trait Locus Accounting for Glucose Intolerance in C57BL/6J Mice | Q64355613 | ||
Mitochondrial transhydrogenase--a key enzyme in insulin secretion and, potentially, diabetes | Q83965392 | ||
P433 | issue | 11 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 3249-3261 | |
P577 | publication date | 2016-08-05 | |
P1433 | published in | Diabetes | Q895262 |
P1476 | title | A Direct Comparison of Metabolic Responses to High-Fat Diet in C57BL/6J and C57BL/6NJ Mice | |
P478 | volume | 65 |
Q47722987 | 17β-Estradiol Directly Lowers Mitochondrial Membrane Microviscosity and Improves Bioenergetic Function in Skeletal Muscle |
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Q61446622 | A comparative analysis of hepatic pathological phenotypes in C57BL/6J and C57BL/6N mouse strains in non-alcoholic steatohepatitis models |
Q49893263 | Absence of Nicotinamide Nucleotide Transhydrogenase in C57BL/6J Mice Exacerbates Experimental Atherosclerosis. |
Q94453359 | Analysis of piRNA expression spectra in a non-alcoholic fatty liver disease mouse model induced by a methionine- and choline-deficient diet |
Q91768020 | Association between high-fat diet feeding and male fertility in high reproductive performance mice |
Q47106495 | Brain insulin resistance impairs hippocampal synaptic plasticity and memory by increasing GluA1 palmitoylation through FoxO3a. |
Q98611581 | C57BL/6J substrain differences in response to high-fat diet intervention |
Q52582444 | Characterization and utilization of the flexor digitorum brevis for assessing skeletal muscle function. |
Q99572053 | DR3 stimulation of adipose resident ILC2s ameliorates type 2 diabetes mellitus |
Q92537632 | Decreases in Circulating Concentrations of Long-Chain Acylcarnitines and Free Fatty Acids During the Glucose Tolerance Test Represent Tissue-Specific Insulin Sensitivity |
Q89993262 | Effect of Tff3 Deficiency and ER Stress in the Liver |
Q48323438 | Genetics of metabolic syndrome: potential clues from wild-derived inbred mouse strains |
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Q38991110 | High fat feeding unmasks variable insulin responses in male C57BL/6 mouse substrains |
Q90400070 | Influence of Social Isolation During Prolonged Simulated Weightlessness by Hindlimb Unloading |
Q38670980 | Loss of sirtuin 4 leads to elevated glucose- and leucine-stimulated insulin levels and accelerated age-induced insulin resistance in multiple murine genetic backgrounds. |
Q48249334 | Manifestations and mechanisms of myocardial lipotoxicity in obesity. |
Q58735419 | Mitochondrial - nuclear genetic interaction modulates whole body metabolism, adiposity and gene expression in vivo |
Q90660624 | Mylk3 null C57BL/6N mice develop cardiomyopathy, whereas Nnt null C57BL/6J mice do not |
Q33726651 | NNT reverse mode of operation mediates glucose control of mitochondrial NADPH and glutathione redox state in mouse pancreatic β-cells |
Q92000262 | Neuronal growth regulator 1-deficient mice show increased adiposity and decreased muscle mass |
Q90075126 | Obesity, Neuroinflammation, and Reproductive Function |
Q88891926 | Remodeling of skeletal muscle mitochondrial proteome with high-fat diet involves greater changes to β-oxidation than electron transfer proteins in mice |
Q92473034 | Response of Liver Metabolic Pathways to Ketogenic Diet and Exercise Are Not Additive |
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