PI3K/Akt signaling is involved in the disruption of gap junctional communication caused by v-Src and TNF-α.

scientific article published on 19 August 2010

PI3K/Akt signaling is involved in the disruption of gap junctional communication caused by v-Src and TNF-α. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/J.BBRC.2010.08.045
P698PubMed publication ID20727856

P2093author name stringHong Yuan
Hitoki Hasegawa
Michinari Hamaguchi
Satoko Ito
Takeshi Senga
Toshinori Hyodo
P433issue2
P407language of work or nameEnglishQ1860
P921main subjectTNFQ18032037
P304page(s)230-235
P577publication date2010-08-19
P1433published inBiochemical and Biophysical Research CommunicationsQ864228
P1476titlePI3K/Akt signaling is involved in the disruption of gap junctional communication caused by v-Src and TNF-α.
P478volume400

Reverse relations

cites work (P2860)
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Q38851729Differential effects of polyoma virus middle tumor antigen mutants upon gap junctional, intercellular communication
Q47220797Gap junctional intercellular communication dysfunction mediates the cognitive impairment induced by cerebral ischemia-reperfusion injury: PI3K/Akt pathway involved
Q37246555Managing the complexity of communication: regulation of gap junctions by post-translational modification
Q35832975Oncogenic extracellular HSP70 disrupts the gap-junctional coupling between capillary cells
Q64244360PI3k and Stat3: Oncogenes that are Required for Gap Junctional, Intercellular Communication
Q34766134Polycyclic aromatic hydrocarbon-induced signaling events relevant to inflammation and tumorigenesis in lung cells are dependent on molecular structure
Q51541259Prostaglandin E₂ maintains mouse ESC undifferentiated state through regulation of connexin31, connexin43 and connexin45 expression: involvement of glycogen synthase kinase 3β/β-catenin.
Q56330679Signalling of DNA damage and cytokines across cell barriers exposed to nanoparticles depends on barrier thickness

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