In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron.

scientific article

In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1161/HYPERTENSIONAHA.115.07061
P698PubMed publication ID27170740

P50authorRalf PalmisanoQ50027588
P2093author name stringChristoph Korbmacher
Marko Bertog
Viatcheslav Nesterov
Bettina Krueger
Anke Dahlmann
P433issue6
P407language of work or nameEnglishQ1860
P921main subjectLiddle syndromeQ1823656
P304page(s)1256-1262
P577publication date2016-06-01
P1433published inHypertensionQ5958695
P1476titleIn Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron
P478volume67

Reverse relations

cites work (P2860)
Q90715776Effects of syntaxins 2, 3, and 4 on rat and human epithelial sodium channel (ENaC) in Xenopus laevis oocytes
Q38598428Inhibitors of the proteasome stimulate the epithelial sodium channel (ENaC) through SGK1 and mimic the effect of aldosterone
Q52657742Liddle Syndrome: Review of the Literature and Description of a New Case.
Q50033979The Low-Renin Hypertension Phenotype: Genetics and the Role of the Mineralocorticoid Receptor.
Q90341118Ubiquitination of renal ENaC subunits in vivo

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