| scholarly article | Q13442814 |
| P50 | author | James Paton | Q67088717 |
| Adrienne W Paton | Q86908184 | ||
| P2093 | author name string | Masanori Kitamura | |
| Tao Huang | |||
| Jian Yao | |||
| Masayuki Takeda | |||
| Kunihiro Hayakawa | |||
| Nobuhiko Hiramatsu | |||
| Isao Araki | |||
| Norifumi Sawada | |||
| Shuqi Du | |||
| Ayumi Kasai | |||
| Maro Okamura | |||
| P2860 | cites work | Regulated translation initiation controls stress-induced gene expression in mammalian cells | Q28506388 |
| P433 | issue | 11 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | cyclosporine | Q367700 |
| NF-κB | Q411114 | ||
| P304 | page(s) | 7201-7211 | |
| P577 | publication date | 2009-06-01 | |
| P1433 | published in | Journal of Immunology | Q3521441 |
| P1476 | title | Suppression of NF-kappaB by cyclosporin a and tacrolimus (FK506) via induction of the C/EBP family: implication for unfolded protein response | |
| P478 | volume | 182 |
| Q89939043 | A Review of Topical Cyclosporine A Formulations-A Disease-Modifying Agent for Keratoconjunctivitis Sicca |
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| Q37225434 | C/EBP homologous protein (CHOP) deficiency ameliorates renal fibrosis in unilateral ureteral obstructive kidney disease |
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| Q59330226 | Cyclophilin A as a target in the treatment of cytomegalovirus infections |
| Q38956307 | Detection of the mechanism of immunotoxicity of cyclosporine A in murine in vitro and in vivo models. |
| Q33562248 | ER stress depresses NF-kappaB activation in mesangial cells through preferential induction of C/EBP beta |
| Q38974029 | Effect of immunosuppressants tacrolimus and mycophenolate mofetil on the keratinocyte UVB response. |
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| Q91560156 | Observation of topical tacrolimus on high-risk penetrating keratoplasty patients: a randomized clinical trial study |
| Q101409988 | Oroxylin A alleviates immunoparalysis of CLP mice by degrading CHOP through interacting with FBXO15 |
| Q41583320 | Physiological/pathological ramifications of transcription factors in the unfolded protein response |
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| Q51538354 | TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity. |
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