scholarly article | Q13442814 |
P356 | DOI | 10.1152/AJPRENAL.00426.2015 |
P8608 | Fatcat ID | release_c36p2nzhxvahvmbuddtz5srxri |
P698 | PubMed publication ID | 27076647 |
P2093 | author name string | Hervé Husson | |
Oxana Ibraghimov-Beskrovnaya | |||
Philippe Beauverger | |||
Philip Janiak | |||
Olivier Duclos | |||
Ryan J Russo | |||
Steven R Ledbetter | |||
Nikolay O Bukanov | |||
Thomas A Natoli | |||
Kelly A Rogers | |||
Christina Bracken | |||
P2860 | cites work | Exome capture reveals ZNF423 and CEP164 mutations, linking renal ciliopathies to DNA damage response signaling | Q24294774 |
NEK8 mutations affect ciliary and centrosomal localization and may cause nephronophthisis | Q24306642 | ||
Polycystin-2 down-regulates cell proliferation via promoting PERK-dependent phosphorylation of eIF2alpha | Q24318555 | ||
Autosomal dominant polycystic kidney disease: the last 3 years | Q24656249 | ||
Mechanisms of CaMKII action in long-term potentiation | Q26998387 | ||
The role of the cilium in normal and abnormal cell cycles: emphasis on renal cystic pathologies | Q27026481 | ||
THM1 negatively modulates mouse sonic hedgehog signal transduction and affects retrograde intraflagellar transport in cilia | Q28505803 | ||
Impaired Wnt-beta-catenin signaling disrupts adult renal homeostasis and leads to cystic kidney ciliopathy | Q28506100 | ||
Increased hedgehog signaling in postnatal kidney results in aberrant activation of nephron developmental programs | Q28588215 | ||
Aberrant Wnt signalling and cellular over-proliferation in a novel mouse model of Meckel-Gruber syndrome | Q28591420 | ||
CaMKII determines mitochondrial stress responses in heart | Q28591986 | ||
Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress | Q29615495 | ||
Effects of lovastatin treatment on the metabolic distributions in the Han:SPRD rat model of polycystic kidney disease | Q30658399 | ||
Nephronophthisis: disease mechanisms of a ciliopathy | Q33594278 | ||
Sirtuin 1 inhibition delays cyst formation in autosomal-dominant polycystic kidney disease | Q33914623 | ||
Inflammation, Oxidative Stress, and Insulin Resistance in Polycystic Kidney Disease | Q34497281 | ||
Cyclic AMP-mediated cyst expansion | Q34850793 | ||
Roles of oxidative stress and antioxidant therapy in chronic kidney disease and hypertension | Q35748416 | ||
Kidney injury accelerates cystogenesis via pathways modulated by heme oxygenase and complement | Q36050899 | ||
Apoptosis and loss of renal tissue in polycystic kidney diseases | Q36707140 | ||
Polycystic kidney disease: genes, proteins, animal models, disease mechanisms and therapeutic opportunities | Q36707529 | ||
Defective glucose metabolism in polycystic kidney disease identifies a new therapeutic strategy. | Q37092766 | ||
Endoplasmic reticulum stress in the kidney | Q37184493 | ||
Structural studies on the regulation of Ca2+/calmodulin dependent protein kinase II | Q37210184 | ||
Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways. | Q37363053 | ||
8-hydroxy-2' -deoxyguanosine (8-OHdG): A critical biomarker of oxidative stress and carcinogenesis | Q37468730 | ||
The role of CaMKII regulation of phospholamban activity in heart disease | Q37555074 | ||
Endoplasmic reticulum stress as a progression factor for kidney injury | Q37666123 | ||
Whole-exome resequencing distinguishes cystic kidney diseases from phenocopies in renal ciliopathies | Q37678462 | ||
Impact of Mammalian Target of Rapamycin Inhibition on Autosomal-Dominant Polycystic Kidney Disease | Q37811516 | ||
Polycystic kidney disease: pathogenesis and potential therapies | Q37819164 | ||
CaMKII in myocardial hypertrophy and heart failure | Q37833230 | ||
Controlling the cell cycle: the role of calcium/calmodulin-stimulated protein kinases I and II. | Q37837372 | ||
Analysis of CaM-kinase signaling in cells | Q37870268 | ||
Autosomal dominant polycystic kidney disease: recent advances in pathogenesis and potential therapies | Q38063460 | ||
CaMKII inhibitors: from research tools to therapeutic agents | Q38193798 | ||
Mitochondrial analysis in living cells: the use of rhodamine 123 and flow cytometry | Q39479961 | ||
The γ-secretase cleavage product of polycystin-1 regulates TCF and CHOP-mediated transcriptional activation through a p300-dependent mechanism | Q40619026 | ||
Inhibition of glucosylceramide accumulation results in effective blockade of polycystic kidney disease in mouse models | Q40899081 | ||
CDK inhibitors R-roscovitine and S-CR8 effectively block renal and hepatic cystogenesis in an orthologous model of ADPKD. | Q41787735 | ||
Everolimus in patients with autosomal dominant polycystic kidney disease. | Q43004249 | ||
Sirolimus and kidney growth in autosomal dominant polycystic kidney disease | Q43004251 | ||
CaMK-II is a PKD2 target that promotes pronephric kidney development and stabilizes cilia | Q43986243 | ||
Characterization of diabetic nephropathy in CaM kinase IIalpha (Thr286Asp) transgenic mice. | Q46205784 | ||
A novel model of insulin-dependent diabetes with renal and retinal lesions by transgenic expression of CaMKIIalpha (Thr286Asp) in pancreatic beta-cells | Q46541900 | ||
Long-lasting arrest of murine polycystic kidney disease with CDK inhibitor roscovitine. | Q52575181 | ||
P433 | issue | 11 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | murine model | Q122890741 |
P304 | page(s) | F1414-22 | |
P577 | publication date | 2016-04-13 | |
P1433 | published in | American Journal of Physiology - Renal Physiology | Q2610177 |
P1476 | title | CaMKII as a pathological mediator of ER stress, oxidative stress, and mitochondrial dysfunction in a murine model of nephronophthisis | |
P478 | volume | 310 |
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