Schedule-dependent apoptosis in K-ras mutant non-small-cell lung cancer cell lines treated with docetaxel and erlotinib: rationale for pharmacodynamic separation

scientific article published on November 2007

Schedule-dependent apoptosis in K-ras mutant non-small-cell lung cancer cell lines treated with docetaxel and erlotinib: rationale for pharmacodynamic separation is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.3816/CLC.2007.N.041
P698PubMed publication ID18186959

P50authorDavid R GandaraQ73567194
P2093author name stringPaul H Gumerlock
Philip C Mack
Primo N Lara
Angela M Davies
Brandi Pryde
Christopher M Mahaffey
William Holland
P2860cites workErlotinib, an effective epidermal growth factor receptor tyrosine kinase inhibitor, induces p27KIP1 up-regulation and nuclear translocation in association with cell growth inhibition and G1/S phase arrest in human non-small-cell lung cancer cell linQ40141617
P433issue9
P407language of work or nameEnglishQ1860
P921main subjectlung cancerQ47912
erlotinibQ418369
docetaxelQ420436
pharmacodynamicsQ725307
P304page(s)548-553
P577publication date2007-11-01
P1433published inClinical Lung CancerQ332299
P1476titleSchedule-dependent apoptosis in K-ras mutant non-small-cell lung cancer cell lines treated with docetaxel and erlotinib: rationale for pharmacodynamic separation
P478volume8

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cites work (P2860)
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