review article | Q7318358 |
scholarly article | Q13442814 |
P356 | DOI | 10.1007/S00101-017-0329-X |
P698 | PubMed publication ID | 28677016 |
P2093 | author name string | L Martin | |
C Thiemermann | |||
T Schürholz | |||
M Derwall | |||
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Matrix metalloproteinase inhibitors attenuate endotoxemia induced cardiac dysfunction: a potential role for MMP-9 | Q59200919 | ||
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Right ventricular dysfunction and dilatation, similar to left ventricular changes, characterize the cardiac depression of septic shock in humans | Q68692688 | ||
The coronary circulation in human septic shock | Q70014120 | ||
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[Is there any indication for perioperative use of the pulmonary artery catheter?] | Q83610683 | ||
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Effects and mechanism analysis of combined infusion by levosimendan and vasopressin on acute lung injury in rats septic shock | Q87363331 | ||
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Sepsis and burn complicated by sepsis alter cardiac transporter expression. | Q53585501 | ||
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Vasoactive agents for the treatment of sepsis | Q28068818 | ||
The Endothelial Glycocalyx: New Diagnostic and Therapeutic Approaches in Sepsis | Q28072373 | ||
Bacterial flagellin triggers cardiac innate immune responses and acute contractile dysfunction | Q28573976 | ||
The immunopathogenesis of sepsis | Q29622909 | ||
Severity of cardiac impairment in the early stage of community-acquired sepsis determines worse prognosis | Q30317801 | ||
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Myocardial dysfunction in sepsis: a large, unsolved puzzle | Q33353302 | ||
New noninvasive method for assessment of left ventricular rotation: speckle tracking echocardiography | Q34467386 | ||
Role of endothelial heparanase in delayed-type hypersensitivity | Q34557376 | ||
A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance | Q34560402 | ||
Dysregulation of intracellular calcium transporters in animal models of sepsis-induced cardiomyopathy | Q34725636 | ||
Vasopressin versus norepinephrine infusion in patients with septic shock. | Q34756330 | ||
Levosimendan attenuates multiple organ injury and improves survival in peritonitis-induced septic shock: studies in a rat model | Q34761833 | ||
Myocardial dysfunction in the patient with sepsis | Q34925777 | ||
Role of extracellular histones in the cardiomyopathy of sepsis. | Q35555107 | ||
Silencing of uncoupling protein 2 by small interfering RNA aggravates mitochondrial dysfunction in cardiomyocytes under septic conditions | Q35609951 | ||
Peptide 19-2.5 inhibits heparan sulfate-triggered inflammation in murine cardiomyocytes stimulated with human sepsis serum | Q35646306 | ||
To beta block or not to beta block; that is the question | Q36088227 | ||
An essential role for complement C5a in the pathogenesis of septic cardiac dysfunction | Q36227873 | ||
Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock | Q36356245 | ||
Toll-like receptor 3 plays a central role in cardiac dysfunction during polymicrobial sepsis | Q36825545 | ||
Toll-like receptor 9 promotes cardiac inflammation and heart failure during polymicrobial sepsis | Q37019795 | ||
The pulmonary endothelial glycocalyx regulates neutrophil adhesion and lung injury during experimental sepsis. | Q37045970 | ||
Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia. | Q37120814 | ||
Continuous terlipressin versus vasopressin infusion in septic shock (TERLIVAP): a randomized, controlled pilot study | Q37360357 | ||
Bench-to-bedside review: Beta-adrenergic modulation in sepsis. | Q37623067 | ||
Complications associated with pulmonary artery catheters: a comprehensive clinical review | Q37706486 | ||
Proteoglycans in health and disease: the multiple roles of syndecan shedding | Q37788814 | ||
Toll-like receptor 4 stimulation initiates an inflammatory response that decreases cardiomyocyte contractility | Q37815210 | ||
Lipopolysaccharide and sepsis-associated myocardial dysfunction | Q37850260 | ||
Role of toll-like receptors in cardiovascular diseases | Q37854080 | ||
Management of myocardial dysfunction in severe sepsis. | Q37867058 | ||
Characterization of cardiac dysfunction in sepsis: an ongoing challenge | Q38172334 | ||
Myocardial depression in sepsis: from pathogenesis to clinical manifestations and treatment | Q38209176 | ||
Effect of eritoran, an antagonist of MD2-TLR4, on mortality in patients with severe sepsis: the ACCESS randomized trial | Q38316900 | ||
Effect of heart rate control with esmolol on hemodynamic and clinical outcomes in patients with septic shock: a randomized clinical trial | Q38443276 | ||
Levosimendan reduces mortality in patients with severe sepsis and septic shock: A meta-analysis of randomized trials | Q38533057 | ||
Levosimendan for the Prevention of Acute Organ Dysfunction in Sepsis. | Q39029084 | ||
Cardiac fibroblasts: contributory role in septic cardiac dysfunction | Q39200106 | ||
Surviving Sepsis Campaign: International Guidelines for Management of Sepsis and Septic Shock: 2016. | Q40367577 | ||
Detection of Myocardial Dysfunction in Septic Shock: A Speckle-Tracking Echocardiography Study. | Q40511368 | ||
Is early ventricular dysfunction or dilatation associated with lower mortality rate in adult severe sepsis and septic shock? A meta-analysis | Q40657906 | ||
Receptor-mediated monitoring of tissue well-being via detection of soluble heparan sulfate by Toll-like receptor 4. | Q40733888 | ||
Impact of Dobutamine in Patients With Septic Shock: A Meta-Regression Analysis | Q40748583 | ||
Sequential N-Terminal Pro-B-Type Natriuretic Peptide and High-Sensitivity Cardiac Troponin Measurements During Albumin Replacement in Patients With Severe Sepsis or Septic Shock | Q40908825 | ||
Soluble Heparan Sulfate in Serum of Septic Shock Patients Induces Mitochondrial Dysfunction in Murine Cardiomyocytes | Q40925027 | ||
Circulating Histones Are Major Mediators of Cardiac Injury in Patients With Sepsis. | Q41134828 | ||
Continuous cardiac output measurement by un-calibrated pulse wave analysis and pulmonary artery catheter in patients with septic shock. | Q41343987 | ||
Myocardial Dysfunction in Severe Sepsis and Septic Shock: No Correlation With Inflammatory Cytokines in Real-life Clinical Setting | Q41543244 | ||
The Role of Uncoupling Protein 2 During Myocardial Dysfunction in a Canine Model of Endotoxin Shock | Q41590175 | ||
Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum | Q41903981 | ||
Evaluation of left ventricular systolic function revisited in septic shock. | Q42176944 | ||
G protein and adenylate cyclase complex-mediated signal transduction in the rat heart during sepsis | Q42442342 | ||
Intraaortic balloon support for myocardial infarction with cardiogenic shock | Q42664790 | ||
Ivabradine: a preliminary observation for a new terapeutic role in patients with multiple organ dysfunction syndrome | Q42871311 | ||
Efficacy and safety of dopamine versus norepinephrine in the management of septic shock | Q43256319 | ||
Vasopressin or norepinephrine in early hyperdynamic septic shock: a randomized clinical trial | Q43412778 | ||
Brain natriuretic peptide: A marker of myocardial dysfunction and prognosis during severe sepsis | Q43707142 | ||
Infusion of methylene blue in human septic shock: a pilot, randomized, controlled study | Q43757721 | ||
Infusion of the beta-adrenergic blocker esmolol attenuates myocardial dysfunction in septic rats | Q43995613 | ||
Association between mitochondrial dysfunction and severity and outcome of septic shock | Q44074137 | ||
IL-1beta and IL-6 act synergistically with TNF-alpha to alter cardiac contractile function after burn trauma | Q44188428 | ||
Postlipopolysaccharide oxidative damage of mitochondrial DNA. | Q44248025 | ||
Profound but reversible myocardial depression in patients with septic shock | Q44250178 | ||
Specific inhibition of mitochondrial oxidative stress suppresses inflammation and improves cardiac function in a rat pneumonia-related sepsis model | Q44290150 | ||
Protective role of PARK2/Parkin in sepsis-induced cardiac contractile and mitochondrial dysfunction | Q45793639 | ||
The Ca2+-sensitizer levosimendan improves oxidative damage, BNP and pro-inflammatory cytokine levels in patients with advanced decompensated heart failure in comparison to dobutamine | Q46515116 | ||
Mechanisms of cardiac depression caused by lipoteichoic acids from Staphylococcus aureus in isolated rat hearts | Q46618537 | ||
Toll-like receptor 4, nitric oxide, and myocardial depression in endotoxemia | Q46862129 | ||
High-mobility group box 1 (HMGB1) impaired cardiac excitation-contraction coupling by enhancing the sarcoplasmic reticulum (SR) Ca(2+) leak through TLR4-ROS signaling in cardiomyocytes | Q46878398 | ||
The effects of dobutamine on microcirculatory alterations in patients with septic shock are independent of its systemic effects | Q46902596 | ||
Influence of levosimendan on organ dysfunction in patients with severely reduced left ventricular function undergoing cardiac surgery | Q47877301 | ||
Use of a Short-Acting β1 Blocker During Endotoxemia May Reduce Cerebral Tissue Oxygenation if Hemodynamics are Depressed by a Decrease in Heart Rate | Q47885980 | ||
Nitric oxide regulation of myocardial contractility and calcium cycling: independent impact of neuronal and endothelial nitric oxide synthases | Q47891975 | ||
Lipopolysaccharide induces oxidative cardiac mitochondrial damage and biogenesis | Q47911806 | ||
Clinical management of the cardiovascular failure in sepsis | Q48496620 | ||
Evaluation of the role of cellular hypoxia in sepsis by the hypoxic marker [18F]fluoromisonidazole | Q48642254 | ||
Competitive and noncompetitive inhibition of myocardial cytochrome C oxidase in sepsis | Q48917473 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 7 | |
P407 | language of work or name | German | Q188 |
P921 | main subject | sepsis | Q183134 |
P1104 | number of pages | 12 | |
P304 | page(s) | 479-490 | |
P577 | publication date | 2017-07-04 | |
P1433 | published in | Anaesthesist | Q1192168 |
P1476 | title | Heart in sepsis : Molecular mechanisms, diagnosis and therapy of septic cardiomyopathy | |
P478 | volume | 66 |
Q48124080 | Ablation of the Right Cardiac Vagus Nerve Reduces Acetylcholine Content without Changing the Inflammatory Response during Endotoxemia. |
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Q91741502 | Combination era, using combined vasopressors showed benefits in treating septic shock patients: a network meta-analysis of randomized controlled trials |
Q52608545 | Heparan Sulfate Induces Necroptosis in Murine Cardiomyocytes: A Medical-In silico Approach Combining In vitro Experiments and Machine Learning. |
Q64947650 | Levosimendan as a new force in the treatment of sepsis-induced cardiomyopathy: mechanism and clinical application. |
Q64235524 | Lipopolysaccharide (LPS) Aggravates High Glucose- and Hypoxia/Reoxygenation-Induced Injury through Activating ROS-Dependent NLRP3 Inflammasome-Mediated Pyroptosis in H9C2 Cardiomyocytes |
Q88412505 | [Sharpen the classical view of septic cardiomyopathy] |
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