The protection mechanism of proline from D-galactosamine hepatitis involves the early activation of ROS-eliminating pathway in the liver

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The protection mechanism of proline from D-galactosamine hepatitis involves the early activation of ROS-eliminating pathway in the liver is …
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scholarly articleQ13442814

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P6179Dimensions Publication ID1006409562
P356DOI10.1186/S40064-015-0969-8
P8608Fatcat IDrelease_4ae6nci6wzbnzjxlopgbnyhvey
P932PMC publication ID4424230
P698PubMed publication ID25984437
P5875ResearchGate publication ID276132220

P2093author name stringMichio Takahashi
Masato Mori
Shintaro Yoshida
Harumi Arisaka
Yoko Obayashi
P2860cites workSuperoxide anion generation in the liver during the early stage of endotoxemia in rats.Q54710671
Proline protects liver from D-galactosamine hepatitis by activating the IL-6/STAT3 survival signaling pathway.Q65000713
Significance of endotoxaemia in experimental "galactosamine-hepatitis" in the ratQ67569126
Status of the mitochondrial pool of glutathione in the isolated hepatocyteQ70288719
The relationship of biliary glutathione disulfide efflux and intracellular glutathione disulfide content in perfused rat liverQ70312114
Catalytic and regulatory properties of the heavy subunit of rat kidney gamma-glutamylcysteine synthetaseQ72231174
[Metabolic aspects of endotoxin as a model of septic shock--approached from oxidative stress]Q76378924
Dietary requirements of synthesizable amino acids by animals: a paradigm shift in protein nutritionQ26995057
Amino acid sequence and function of the light subunit of rat kidney gamma-glutamylcysteine synthetaseQ28576255
Mechanism and significance of increased glutathione level in human hepatocellular carcinoma and liver regenerationQ31635948
Functional amino acids in growth, reproduction, and healthQ34591925
The role of the liver in the response to LPS: experimental and clinical findingsQ35049361
Oxidative stress and septic shock: metabolic aspects of oxygen-derived free radicals generated in the liver during endotoxemiaQ36532248
Mitochondrial glutathione: hepatocellular survival-death switchQ36588190
Proline and hydroxyproline metabolism: implications for animal and human nutritionQ37171748
Oxidant stress, mitochondria, and cell death mechanisms in drug-induced liver injury: lessons learned from acetaminophen hepatotoxicityQ37656707
Dietary requirements of "nutritionally non-essential amino acids" by animals and humansQ38068133
An ultrastructural and histochemical study of the prominent inflammatory response in D(+)-galactosamine hepatotoxicityQ41557520
GSH transport in mitochondria: defense against TNF-induced oxidative stress and alcohol-induced defect.Q41558522
Toxicity by pyruvate in HepG2 cells depleted of glutathione: role of mitochondriaQ43832680
Low-dose TNF-alpha protects against hepatic ischemia-reperfusion injury in mice: implications for preconditioningQ44261195
Loss of suppression of GSH synthesis at low cell density in primary cultures of rat hepatocytesQ46096882
Changes in glutathione homeostasis during liver regeneration in the rat.Q46260844
Ischemic preconditioning enhances scavenging activity of reactive oxygen species and diminishes transmural difference of infarct sizeQ46616108
P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P304page(s)199
P577publication date2015-04-28
P1433published inSpringerPlusQ24055190
P1476titleThe protection mechanism of proline from D-galactosamine hepatitis involves the early activation of ROS-eliminating pathway in the liver
P478volume4