Phosphatidylinositol 3-kinase-dependent activation of akt, an essential signal for hyperthermia-induced heat-shock protein 72, is attenuated in streptozotocin-induced diabetic heart.

scientific article

Phosphatidylinositol 3-kinase-dependent activation of akt, an essential signal for hyperthermia-induced heat-shock protein 72, is attenuated in streptozotocin-induced diabetic heart. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.2337/DB05-0266
P698PubMed publication ID16644687
P5875ResearchGate publication ID7135961

P50authorNaohiko TakahashiQ123913766
P2093author name stringHironobu Yoshimatsu
Mikiko Nakagawa
Tetsuji Shinohara
Hidetoshi Yonemochi
Masahide Hara
Sakuji Shigematsu
Tatsuhiko Ooie
Tetsunori Saikawa
P433issue5
P407language of work or nameEnglishQ1860
P304page(s)1307-1315
P577publication date2006-05-01
P1433published inDiabetesQ895262
P1476titlePhosphatidylinositol 3-kinase-dependent activation of akt, an essential signal for hyperthermia-induced heat-shock protein 72, is attenuated in streptozotocin-induced diabetic heart.
P478volume55

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cites work (P2860)
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Q36896750Heat shock proteins are important mediators of skeletal muscle insulin sensitivity
Q46990081Hyperthermia treatment prevents angiotensin II-mediated atrial fibrosis and fibrillation via induction of heat-shock protein 72.
Q35857763Hyperthermia with mild electrical stimulation protects pancreatic β-cells from cell stresses and apoptosis.
Q46113908Hyperthermia-induced cardioprotection is potentiated by ischemic postconditioning in rats
Q34505699Increased serum HSP70 levels are associated with the duration of diabetes
Q40382378Induction of heat shock proteins prevents the arrhythmogenic substrate for atrial fibrillation
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Q54630124Protective effect of ginsenoside Rb1 against myocardial ischemia/reperfusion injury in streptozotocin-induced diabetic rats
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Q33700820The importance of the cellular stress response in the pathogenesis and treatment of type 2 diabetes

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