Loperamide Restricts Intracellular Growth of Mycobacterium tuberculosis in Lung Macrophages.

scientific article published on 28 July 2016

Loperamide Restricts Intracellular Growth of Mycobacterium tuberculosis in Lung Macrophages. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1165/RCMB.2015-0383OC
P698PubMed publication ID27468130

P50authorJaime ChávezQ64034504
Martha TorresQ77437183
Esmeralda JuárezQ59167074
P2093author name stringGuadalupe Sánchez
Carmen Sarabia
Eduardo Sada
Claudia Carranza
Mitzi González
P2860cites workLC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processingQ24597817
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Effect of PI3K- and mTOR-specific inhibitors on spontaneous B-cell follicular lymphomas in PTEN/LKB1-deficient miceQ41826974
Analgesic action of loperamide, an opioid agonist, and its blocking action on voltage-dependent Ca2+ channelsQ44342256
New insights into the P-glycoprotein-mediated effluxes of rhodamines.Q53943797
P433issue6
P921main subjectMycobacterium tuberculosisQ130971
loperamideQ423751
P304page(s)837-847
P577publication date2016-12-01
P1433published inAmerican Journal of Respiratory Cell and Molecular BiologyQ4744268
P1476titleLoperamide Restricts Intracellular Growth of Mycobacterium tuberculosis in Lung Macrophages
P478volume55

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cites work (P2860)
Q91890852Autophagy-activating strategies to promote innate defense against mycobacteria
Q57466482Autophagy: A new strategy for host-directed therapy of tuberculosis
Q64093968Exploring the Drug Repurposing Versatility of Valproic Acid as a Multifunctional Regulator of Innate and Adaptive Immune Cells
Q99237959Host-Directed Therapy in Tuberculosis: Targeting Host Metabolism
Q52324318Innate Immune Responses in Leprosy.
Q39299815Role of apoptosis and autophagy in tuberculosis
Q91474358Therapeutic host-directed strategies to improve outcome in tuberculosis
Q90914855Unpacking the Pathogen Box-An Open Source Tool for Fighting Neglected Tropical Disease
Q91316115Valproic acid promotes a decrease in mycobacterial survival by enhancing nitric oxide production in macrophages stimulated with IFN-γ

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