| scholarly article | Q13442814 |
| P356 | DOI | 10.1111/JRE.12391 |
| P698 | PubMed publication ID | 27397896 |
| P2093 | author name string | T-S Yang | |
| C-Y Chiang | |||
| H-C Chiu | |||
| E Fu | |||
| H-P Tu | |||
| K-C Shih | |||
| Y-T Chin | |||
| F-G Lin | |||
| M M-J Fu | |||
| P2860 | cites work | RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides | Q22010236 |
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| Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics? | Q24655246 | ||
| Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy | Q24682750 | ||
| The S100 protein family: history, function, and expression | Q28293937 | ||
| TIRAP, an adaptor protein for TLR2/4, transduces a signal from RAGE phosphorylated upon ligand binding | Q28741936 | ||
| Actinobacillus actinomycetemcomitans and Porphyromonas gingivalis in human periodontal disease: occurrence and treatment | Q33752036 | ||
| Toll-like receptors regulate B cell cytokine production in patients with diabetes | Q33954398 | ||
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| Bidirectional interrelationships between diabetes and periodontal diseases: an epidemiologic perspective | Q34117734 | ||
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| Treatment of periodontal disease and control of diabetes: an assessment of the evidence and need for future research | Q34557660 | ||
| Advanced glycation end products induce the expression of interleukin-6 and interleukin-8 by receptor for advanced glycation end product-mediated activation of mitogen-activated protein kinases and nuclear factor-κB in human osteoarthritis chondrocyt | Q34802374 | ||
| Epigallocatechin-3-gallate attenuates Porphyromonas gingivalis lipopolysaccharide-enhanced matrix metalloproteinase-1 production through inhibition of interleukin-6 in gingival fibroblasts | Q35038327 | ||
| Glycation products and the pathogenesis of diabetic complications | Q35594899 | ||
| Role of bacteria in health and disease of periodontal tissues | Q36362681 | ||
| Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1. | Q37417494 | ||
| Dietary advanced glycation end-product restriction for the attenuation of insulin resistance, oxidative stress and endothelial dysfunction: a systematic review. | Q38077658 | ||
| RAGE and TLRs: relatives, friends or neighbours? | Q38129659 | ||
| Effect of advanced glycation end product intake on inflammation and aging: a systematic review. | Q38250741 | ||
| Methylglyoxal and advanced glycation end-products promote cytokines expression in peritoneal mesothelial cells via MAPK signaling | Q38920739 | ||
| The anti-inflammation effect of Moutan Cortex on advanced glycation end products-induced rat mesangial cells dysfunction and High-glucose-fat diet and streptozotocin-induced diabetic nephropathy rats | Q39056911 | ||
| Inhibitory effect of gallic acid on advanced glycation end products induced up-regulation of inflammatory cytokines and matrix proteins in H9C2 (2-1) cells | Q39092599 | ||
| Stimulatory effects of glucose and Porphyromonas gingivalis lipopolysaccharide on the secretion of inflammatory mediators from human macrophages | Q39174296 | ||
| High glucose increases nitric oxide generation in lipopolysaccharide-activated macrophages by enhancing activity of protein kinase C-α/δ and NF-κB. | Q39329869 | ||
| Septic shock is associated with receptor for advanced glycation end products ligation of LPS. | Q39602172 | ||
| The anti-inflammatory role of heme oxygenase-1 in lipopolysaccharide and cytokine-stimulated inducible nitric oxide synthase and nitric oxide production in human periodontal ligament cells | Q39766929 | ||
| Interleukin-6 released from fibroblasts is essential for up-regulation of matrix metalloproteinase-1 expression by U937 macrophages in coculture: cross-talking between fibroblasts and U937 macrophages exposed to high glucose | Q42058025 | ||
| Spontaneous periodontitis development in diabetic rats involves an unrestricted expression of inflammatory cytokines and tissue destructive factors in the absence of major changes in commensal oral microbiota | Q42073506 | ||
| Effect of periodontal treatment on metabolic control, systemic inflammation and cytokines in patients with type 2 diabetes. | Q43227921 | ||
| HMGB1 enhances the proinflammatory activity of lipopolysaccharide by promoting the phosphorylation of MAPK p38 through receptor for advanced glycation end products | Q43248408 | ||
| Calculating measures of biological interaction | Q43922226 | ||
| Increased serum levels of the specific AGE-compound methylglyoxal-derived hydroimidazolone in patients with type 2 diabetes. | Q44325186 | ||
| Advanced glycation endproducts (AGEs) induce oxidant stress in the gingiva: a potential mechanism underlying accelerated periodontal disease associated with diabetes | Q48900914 | ||
| Short-term effects of non-surgical periodontal treatment on the gingival crevicular fluid cytokine profiles in sites with induced periodontal defects: a study on dogs with and without streptozotocin-induced diabetes. | Q51322469 | ||
| Progression of periodontal destruction and the roles of advanced glycation end products in experimental diabetes. | Q52624942 | ||
| Enhanced interaction of advanced glycation end products with their cellular receptor RAGE: implications for the pathogenesis of accelerated periodontal disease in diabetes. | Q54126440 | ||
| Toll-like receptor 4-mediated hyper-responsiveness of gingival epithelial cells to lipopolysaccharide in high-glucose environments. | Q54206207 | ||
| Enhancement of lipopolysaccharide-induced toll-like receptor 2 expression and inflammatory cytokine secretion in HUVECs under high glucose conditions. | Q54273713 | ||
| Involvement of HMGB1 and RAGE in IL-1β-induced gingival inflammation. | Q54352589 | ||
| Matrix metalloproteinase-1 of gingival fibroblasts influenced by advanced glycation end products (AGEs) and their association with receptor for AGEs and nuclear factor-κB in gingival connective tissue. | Q54373503 | ||
| Advanced glycation end products and lipopolysaccharide synergistically stimulate proinflammatory cytokine/chemokine production in endothelial cells via activation of both mitogen-activated protein kinases and nuclear factor-kappaB. | Q54729321 | ||
| Treatment of periodontal disease in diabetics reduces glycated hemoglobin | Q73674125 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | lipopolysaccharide | Q421804 |
| Porphyromonas gingivalis | Q3214147 | ||
| P304 | page(s) | 268-276 | |
| P577 | publication date | 2016-07-11 | |
| P1433 | published in | Journal of Periodontal Research | Q15759338 |
| P1476 | title | Effect of high glucose, Porphyromonas gingivalis lipopolysaccharide and advanced glycation end-products on production of interleukin-6/-8 by gingival fibroblasts | |
| P478 | volume | 52 |
| Q38695799 | Advanced glycation end-products and Porphyromonas gingivalis lipopolysaccharide increase calprotectin expression in human gingival epithelial cells. |
| Q46250505 | Advanced glycation end-products increase IL-6 and ICAM-1 expression via RAGE, MAPK and NF-κB pathways in human gingival fibroblasts. |
| Q38889082 | Comparison of periodontal and peri-implant inflammatory parameters among patients with prediabetes, type 2 diabetes mellitus and non-diabetic controls |
| Q99238223 | Current Knowledge Regarding the Interaction Between Oral Bone Metabolic Disorders and Diabetes Mellitus |
| Q38734421 | MicroRNA-126 Regulates Inflammatory Cytokine Secretion in Human Gingival Fibroblasts Under High Glucose Via Targeting Tumor Necrosis Factor Receptor Associated Factor 6. |
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