scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1022058381 |
P356 | DOI | 10.1038/SJ.CDD.4401062 |
P698 | PubMed publication ID | 12181741 |
P5875 | ResearchGate publication ID | 11205131 |
P2093 | author name string | Betticher DC | |
Schneider E | |||
Borner MM | |||
Pirnia F | |||
P2860 | cites work | Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner | Q22008727 |
An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9 | Q22009407 | ||
FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis | Q24307362 | ||
The 40-kDa subunit of DNA fragmentation factor induces DNA fragmentation and chromatin condensation during apoptosis | Q24316760 | ||
Mammalian caspases: structure, activation, substrates, and functions during apoptosis | Q28139429 | ||
A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD | Q28258328 | ||
Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization | Q28275527 | ||
IAP family proteins--suppressors of apoptosis | Q28297514 | ||
Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase | Q28302692 | ||
Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes | Q28506930 | ||
Bcl-2 and Fas/APO-1 regulate distinct pathways to lymphocyte apoptosis | Q28588677 | ||
Target protease specificity of the viral serpin CrmA. Analysis of five caspases | Q28640190 | ||
Caspases: enemies within | Q29547315 | ||
Mitochondria and apoptosis | Q29547905 | ||
Caspase-3 is required for DNA fragmentation and morphological changes associated with apoptosis | Q29614983 | ||
Caspases: killer proteases | Q29616315 | ||
Two CD95 (APO-1/Fas) signaling pathways | Q29619631 | ||
Activation of caspase-8 in drug-induced apoptosis of B-lymphoid cells is independent of CD95/Fas receptor-ligand interaction and occurs downstream of caspase-3. | Q32061783 | ||
Overexpression of caspase-3 restores sensitivity for drug-induced apoptosis in breast cancer cell lines with acquired drug resistance | Q33182147 | ||
Molecular effects of paclitaxel: myths and reality (a critical review). | Q33725219 | ||
p53 and chemosensitivity. | Q33779334 | ||
Biochemical pathways of caspase activation during apoptosis | Q33804316 | ||
Overexpression of the human BCL-2 gene product results in growth enhancement of Epstein-Barr virus-immortalized B cells | Q33844249 | ||
A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes | Q33888184 | ||
Ionizing radiation and chemotherapeutic drugs induce apoptosis in lymphocytes in the absence of Fas or FADD/MORT1 signaling. Implications for cancer therapy | Q36375843 | ||
p53 activates the CD95 (APO-1/Fas) gene in response to DNA damage by anticancer drugs | Q36401199 | ||
Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L) | Q36774959 | ||
Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53. | Q39777499 | ||
Characterization of distinct consecutive phases in non-genotoxic p53-induced apoptosis of Ewing tumor cells and the rate-limiting role of caspase 8. | Q40858469 | ||
The CD95/CD95 ligand system is not the major effector in anticancer drug-mediated apoptosis | Q40961120 | ||
Mechanisms of resistance of human small cell lung cancer lines selected in VP-16 and cisplatin | Q41202524 | ||
Cleavage of human inhibitor of apoptosis protein XIAP results in fragments with distinct specificities for caspases | Q42657216 | ||
Distinct caspase cascades are initiated in receptor-mediated and chemical-induced apoptosis. | Q51091938 | ||
Uncertain identity of doxorubicin-resistant MCF-7 cell lines expressing mutated p53. | Q54036312 | ||
Adriamycin-induced DNA adducts inhibit the DNA interactions of transcription factors and RNA polymerase. | Q54592064 | ||
Activation of Mitochondria and Release of Mitochondrial Apoptogenic Factors by Betulinic Acid | Q57243601 | ||
Apaf-1 is a transcriptional target for E2F and p53 | Q57274134 | ||
DNA damage can induce apoptosis in proliferating lymphoid cells via p53-independent mechanisms inhibitable by Bcl-2 | Q57338694 | ||
Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells | Q57384660 | ||
Caspase-8/FLICE functions as an executioner caspase in anticancer drug-induced apoptosis | Q73073993 | ||
Inhibition of fas death signals by FLIPs | Q77485563 | ||
P433 | issue | 9 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 905-914 | |
P577 | publication date | 2002-09-01 | |
P1433 | published in | Cell Death & Differentiation | Q2943974 |
P1476 | title | Mitomycin C induces apoptosis and caspase-8 and -9 processing through a caspase-3 and Fas-independent pathway | |
P478 | volume | 9 |
Q54453231 | 17β-estradiol impedes Bax-involved mitochondrial apoptosis of retinal nerve cells induced by oxidative damage via the phosphatidylinositol 3-kinase/Akt signal pathway. |
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Q33916804 | Mitomycin-DNA adducts induce p53-dependent and p53-independent cell death pathways |
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