Neutrophil Gelatinase-Associated Lipocalin, a Novel Mineralocorticoid Biotarget, Mediates Vascular Profibrotic Effects of Mineralocorticoids.

scientific article published on 18 May 2015

Neutrophil Gelatinase-Associated Lipocalin, a Novel Mineralocorticoid Biotarget, Mediates Vascular Profibrotic Effects of Mineralocorticoids. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1161/HYPERTENSIONAHA.115.05431
P698PubMed publication ID25987661
P5875ResearchGate publication ID277004504

P50authorAntoine TarjusQ42364390
Ernesto Martinez-MartinezQ42364398
Frederic JaisserQ42364405
P2093author name stringFaiez Zannad
Tak W Mak
Patrick Rossignol
Nicolette Farman
Soumaya El Moghrabi
Thorsten Berger
Natalia López-Andrés
Renaud Fay
Céline Latouche
Cristian Amador
P433issue1
P407language of work or nameEnglishQ1860
P304page(s)158-166
P577publication date2015-05-18
P1433published inHypertensionQ5958695
P1476titleNeutrophil Gelatinase-Associated Lipocalin, a Novel Mineralocorticoid Biotarget, Mediates Vascular Profibrotic Effects of Mineralocorticoids
P478volume66

Reverse relations

cites work (P2860)
Q40159826Experimental and Human Evidence for Lipocalin-2 (Neutrophil Gelatinase-Associated Lipocalin [NGAL]) in the Development of Cardiac Hypertrophy and heart failure
Q99616727Hepatic lipocalin 2 promotes liver fibrosis and portal hypertension
Q58760700Lipocalin-2 derived from adipose tissue mediates aldosterone-induced renal injury
Q47386642Measuring non-polyaminated lipocalin-2 for cardiometabolic risk assessment
Q92031976MiRNA-181a is a novel regulator of aldosterone-mineralocorticoid receptor-mediated cardiac remodelling
Q58722306Mineralocorticoid Receptor and Aldosterone-Related Biomarkers of End-Organ Damage in Cardiometabolic Disease
Q91594685Myocardial Injury After Ischemia/Reperfusion Is Attenuated By Pharmacological Galectin-3 Inhibition
Q47230073Neutrophil Gelatinase-Associated Lipocalin from immune cells is mandatory for aldosterone-induced cardiac remodeling and inflammation.
Q52375176Nitric oxide-sensitive guanylyl cyclase stimulation improves experimental heart failure with preserved ejection fraction.
Q38988806Progression of chronic kidney disease: too much cellular talk causes damage
Q50349533Stat3 Controls Tubulointerstitial Communication during CKD.
Q39199991Steroidal and Novel Non-steroidal Mineralocorticoid Receptor Antagonists in Heart Failure and Cardiorenal Diseases: Comparison at Bench and Bedside
Q35738034The Central Role of Neutrophil Gelatinase-Associated Lipocalin in Cardiovascular Fibrosis

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