scholarly article | Q13442814 |
P50 | author | Bela Joos | Q57571699 |
P2093 | author name string | Béla Joós | |
Catherine E Morris | |||
Pierre-Alexandre Boucher | |||
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Lipid rafts as a membrane-organizing principle | Q29615727 | ||
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Disruption of the axon initial segment cytoskeleton is a new mechanism for neuronal injury | Q33570384 | ||
Automated and quantitative image analysis of ischemic dendritic blebbing using in vivo 2-photon microscopy data | Q33780205 | ||
Riluzole protects against cardiac ischaemia and reperfusion damage via block of the persistent sodium current | Q34112946 | ||
Cell surface area regulation and membrane tension | Q34163175 | ||
Membrane stretch affects gating modes of a skeletal muscle sodium channel | Q34170990 | ||
Voltage-dependent sodium channel function is regulated through membrane mechanics | Q34171460 | ||
A membrane-access mechanism of ion channel inhibition by voltage sensor toxins from spider venom | Q34331989 | ||
Phosphatidylserine, a death knell | Q34355981 | ||
Imbalance of ionic conductances contributes to diverse symptoms of demyelination | Q34375881 | ||
Differential action of riluzole on tetrodotoxin-sensitive and tetrodotoxin-resistant sodium channels | Q34435972 | ||
Sodium channelopathy induced by mild axonal trauma worsens outcome after a repeat injury | Q34458292 | ||
Action potential generation requires a high sodium channel density in the axon initial segment | Q34587263 | ||
Extracellular annexin A5: functions of phosphatidylserine-binding and two-dimensional crystallization | Q34760775 | ||
Potent inhibition of anoxic depolarization by the sodium channel blocker dibucaine | Q34785671 | ||
Ischemia/reperfusion injury of primary porcine cardiomyocytes in a low-shear microfluidic culture and analysis device | Q34996241 | ||
Role of membrane integrity on G protein-coupled receptors: Rhodopsin stability and function | Q35042815 | ||
Electrophysiologic basis for the antiarrhythmic actions of ranolazine | Q35091243 | ||
Raft domains of variable properties and compositions in plasma membrane vesicles | Q35105065 | ||
Inhibition of Neuronal Voltage-Gated Sodium Channels by Brilliant Blue G | Q35122828 | ||
Thiazolidinedione insulin sensitizers alter lipid bilayer properties and voltage-dependent sodium channel function: implications for drug discovery | Q35146426 | ||
Riluzole specifically blocks inactivated Na channels in myelinated nerve fibre | Q43795777 | ||
Conductance of the sodium channel in myelinated nerve fibres with modified sodium inactivation | Q45399251 | ||
Subthreshold sodium current from rapidly inactivating sodium channels drives spontaneous firing of tuberomammillary neurons | Q45713980 | ||
Design and assessment of a potent sodium channel blocking derivative of mexiletine for minimizing experimental neuropathic pain in several rat models. | Q45979049 | ||
Contribution to ischemic injury of rat optic nerves by intracellular sodium overload | Q46775304 | ||
Lipid composition alters drug action at the nicotinic acetylcholine receptor. | Q46868994 | ||
KCNQ channels mediate IKs, a slow K+ current regulating excitability in the rat node of Ranvier | Q46983632 | ||
Kinetic changes and modulation by carbamazepine on voltage-gated sodium channels in rat CA1 neurons after epilepsy | Q48366814 | ||
Bioenergetic abnormalities in discrete cerebral motor pathways presage spinal cord pathology in the G93A SOD1 mouse model of ALS. | Q48582883 | ||
Coupled left-shift of Nav channels: modeling the Na⁺-loading and dysfunctional excitability of damaged axons. | Q48651678 | ||
Impaired stretch modulation in potentially lethal cardiac sodium channel mutants | Q48706868 | ||
Neuropathic pain is associated with increased nodal persistent Na(+) currents in human diabetic neuropathy | Q48828917 | ||
Upregulation of persistent sodium conductances in familial ALS. | Q48838979 | ||
Histopathological changes at central nodes of Ranvier after stretch-injury | Q48942464 | ||
Action potentials and membrane currents in the human node of Ranvier | Q51602048 | ||
Comparative neuroprotective effect of sodium channel blockers after experimental spinal cord injury | Q53550176 | ||
Latent addition in human motor and sensory axons: Different site-dependent changes across the carpal tunnel related to persistent Na+ currents | Q56772194 | ||
Lipidomics: coming to grips with lipid diversity | Q57959842 | ||
Lack of colonic inflammation-induced acute visceral hypersensitivity to colorectal distension in Nav1.9 knockout mice | Q60413857 | ||
Cholesterol alters the binding of Ca2+ channel blockers to the membrane lipid bilayer | Q68053212 | ||
Na current in membrane blebs: implications for channel mobility and patch clamp recording | Q68453570 | ||
Interaction of 1,4 dihydropyridine calcium channel antagonists with biological membranes: lipid bilayer partitioning could occur before drug binding to receptors | Q69352328 | ||
Loss of asymmetric distribution of sarcolemmal phosphatidylethanolamine during simulated ischemia in the isolated neonatal rat cardiomyocyte | Q72873667 | ||
Ischemic loss of sarcolemmal dystrophin and spectrin: correlation with myocardial injury | Q74162649 | ||
Endotoxin reduces availability of voltage-gated human skeletal muscle sodium channels at depolarized membrane potentials | Q80988753 | ||
A reversible form of axon damage in experimental autoimmune encephalomyelitis and multiple sclerosis | Q83729196 | ||
Functional group dependence of solute partitioning to various locations within a DOPC bilayer: a comparison of molecular dynamics simulations with experiment | Q83859436 | ||
Emerging roles for lipids in shaping membrane-protein function. | Q35203586 | ||
Inherited neuronal ion channelopathies: new windows on complex neurological diseases | Q35226085 | ||
Mechanisms of atrial-selective block of Na⁺ channels by ranolazine: II. Insights from a mathematical model | Q35396014 | ||
Tuning Membrane Phase Separation Using Nonlipid Amphiphiles | Q35743337 | ||
Tetracaine-membrane interactions: effects of lipid composition and phase on drug partitioning, location, and ionization | Q35794550 | ||
Nav channel mechanosensitivity: activation and inactivation accelerate reversibly with stretch | Q35878685 | ||
Noninactivating, tetrodotoxin-sensitive Na+ conductance in rat optic nerve axons. | Q36443659 | ||
Continuous membrane-cytoskeleton adhesion requires continuous accommodation to lipid and cytoskeleton dynamics. | Q36475413 | ||
Studying the mechanosensitivity of voltage-gated channels using oocyte patches | Q36494493 | ||
Nav1.4 deregulation in dystrophic skeletal muscle leads to Na+ overload and enhanced cell death. | Q36790081 | ||
S4-based voltage sensors have three major conformations | Q36976792 | ||
Voltage-dependent K+ channel gating and voltage sensor toxin sensitivity depend on the mechanical state of the lipid membrane | Q37039516 | ||
Mechanisms of disease: sodium channels and neuroprotection in multiple sclerosis-current status | Q37070221 | ||
Inactivation of sodium channels underlies reversible neuropathy during critical illness in rats | Q37170792 | ||
The influence of neuroactive steroid lipophilicity on GABAA receptor modulation: evidence for a low-affinity interaction | Q37298786 | ||
Order of lipid phases in model and plasma membranes | Q37377268 | ||
Biochemical, structural, and biomarker evidence for calpain-mediated cytoskeletal change after diffuse brain injury uncomplicated by contusion | Q37425364 | ||
Pharmacologic intervention in axonal excitability: in vivo assessment of nodal persistent sodium currents in human neuropathies | Q37659058 | ||
Subcellular targeting strategies for drug design and delivery | Q37665298 | ||
Voltage-gated sodium channels as therapeutic targets in epilepsy and other neurological disorders. | Q37714086 | ||
Sodium channel blockers for neuropathic pain. | Q37729164 | ||
Isoform-specific and pan-channel partners regulate trafficking and plasma membrane stability; and alter sodium channel gating properties | Q37785310 | ||
Duchenne muscular dystrophy--what causes the increased membrane permeability in skeletal muscle? | Q37810351 | ||
Pumping lipids with P4-ATPases. | Q37824863 | ||
Modulation of KvAP unitary conductance and gating by 1-alkanols and other surface active agents | Q39343471 | ||
Biophysical characterisation of the persistent sodium current of the Nav1.6 neuronal sodium channel: a single-channel analysis | Q39732040 | ||
Membrane trauma and Na+ leak from Nav1.6 channels | Q39816346 | ||
Membrane interaction of calcium channel antagonists modulated by cholesterol. Implications for drug activity | Q40855418 | ||
Dual stretch responses of mHCN2 pacemaker channels: accelerated activation, accelerated deactivation. | Q41947639 | ||
Voltage-gated channel mechanosensitivity: fact or friction? | Q42215065 | ||
Interaction of the P-glycoprotein multidrug efflux pump with cholesterol: effects on ATPase activity, drug binding and transport | Q42445797 | ||
The voltage-gated sodium channel Nav1.9 is required for inflammation-based urinary bladder dysfunction | Q42447844 | ||
Increased persistent sodium current determines cortical hyperexcitability in a genetic model of amyotrophic lateral sclerosis | Q42605747 | ||
Mechanosensitivity of Nav1.5, a voltage-sensitive sodium channel | Q42680525 | ||
Hypoxic injury of isolated axons is independent of ionotropic glutamate receptors | Q42745798 | ||
Solubilisation of drugs within liposomal bilayers: alternatives to cholesterol as a membrane stabilising agent. | Q42840446 | ||
Lipid membrane interactions of indacaterol and salmeterol: do they influence their pharmacological properties? | Q43264026 | ||
Myocardial protection by F 15845, a persistent sodium current blocker, in an ischemia-reperfusion model in the pig. | Q43271899 | ||
Gating behaviour of sodium currents in adult mouse muscle recorded with an improved two-electrode voltage clamp. | Q43418379 | ||
P304 | page(s) | 19 | |
P577 | publication date | 2012-02-23 | |
P1433 | published in | Frontiers in Pharmacology | Q2681208 |
P1476 | title | Left-shifted nav channels in injured bilayer: primary targets for neuroprotective nav antagonists? | |
P478 | volume | 3 |