MIR137 is the key gene mediator of the syndromic obesity phenotype of patients with 1p21.3 microdeletions

scientific article published on 3 November 2016

MIR137 is the key gene mediator of the syndromic obesity phenotype of patients with 1p21.3 microdeletions is …
instance of (P31):
case reportQ2782326
scholarly articleQ13442814

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P6179Dimensions Publication ID1044856784
P356DOI10.1186/S13039-016-0289-X
P8608Fatcat IDrelease_zzjwom5yhjek7ivf5fa3pltjai
P932PMC publication ID5093957
P698PubMed publication ID27822311

P50authorSusanna EspositoQ24239756
Donatella MilaniQ57235159
Claudia CiaccioQ88309414
P2093author name stringGiulietta Scuvera
Arianna Tucci
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Two New Cases of 1p21.3 Deletions and an Unbalanced Translocation t(8;12) among Individuals with Syndromic Obesity.Q35905457
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miRNA genes and the brain: implications for psychiatric disorders.Q37781312
Chromosome 1p21.3 microdeletions comprising DPYD and MIR137 are associated with intellectual disabilityQ38331099
Transcriptional targets of the schizophrenia risk gene MIR137.Q38979078
A direct molecular link between the autism candidate gene RORa and the schizophrenia candidate MIR137.Q39026765
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Hypothalamic miR-103 protects from hyperphagic obesity in mice.Q48284060
Hemizygous deletions on chromosome 1p21.3 involving the DPYD gene in individuals with autism spectrum disorderQ50301287
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Differentiation of neural stem cells into oligodendrocytes: involvement of the polycomb group protein Ezh2Q81757434
P921main subjectobesityQ12174
P304page(s)80
P577publication date2016-11-03
P1433published inMolecular CytogeneticsQ15761790
P1476titleMIR137 is the key gene mediator of the syndromic obesity phenotype of patients with 1p21.3 microdeletions
P478volume9