scholarly article | Q13442814 |
P356 | DOI | 10.1007/BF01232969 |
P698 | PubMed publication ID | 2011791 |
P2093 | author name string | Turker MS | |
Cooper GE | |||
DiMartino DL | |||
P433 | issue | 2 | |
P304 | page(s) | 105-116 | |
P577 | publication date | 1991-03-01 | |
P1433 | published in | Somatic Cell and Molecular Genetics | Q7559232 |
P1476 | title | Molecular analysis of APRT deficiency in mouse P19 teratocarcinoma stem cell line | |
P478 | volume | 17 |
Q40727004 | A mouse kidney cell line with a G:C --> C:G transversion mutator phenotype |
Q28512293 | A role for certain mouseAprt sequences in resistance to toxic adenine analogs |
Q39638182 | Aberrantly silenced promoters retain a persistent memory of the silenced state after long-term reactivation |
Q41557956 | Hemidemethylation is sufficient for chromatin relaxation and transcriptional activation of methylatedaprt gene in mouse P19 embryonal carcinoma cell line |
Q41505845 | Isolation and Characterization of Mutations in the Mouse APRT Gene that Encode Functional Enzymes with Resistance to Toxic Adenine Analogs |
Q41592974 | Molecular and biochemical elucidation of a cellular phenotype characterized by adenine analogue resistance in the presence of high levels of adenine phosphoribosyltransferase activity |
Q73217526 | Molecular evidence for the induction of large interstitial deletions on mouse chromosome 8 by ionizing radiation |
Q48138932 | Region-specific rates of molecular evolution: a fourfold reduction in the rate of accumulation of "silent" mutations in transcribed versus nontranscribed regions of homologous DNA fragments derived from two closely related mouse species |
Q41330895 | Spontaneous and ionizing radiation induced mutations involve large events when selecting for loss of an autosomal locus |
Q40790684 | Spontaneously immortalized cell lines obtained from adult Atm null mice retain sensitivity to ionizing radiation and exhibit a mutational pattern suggestive of oxidative stress |
Q41505861 | The Mouse APRT Gene as a Model for Studying Epigenetic Gene Inactivation |
Q38331684 | The primary function of a redundant Sp1 binding site in the mouse aprt gene promoter is to block epigenetic gene inactivation |
Search more.