Molecular analysis of APRT deficiency in mouse P19 teratocarcinoma stem cell line

scientific article published on March 1991

Molecular analysis of APRT deficiency in mouse P19 teratocarcinoma stem cell line is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1007/BF01232969
P698PubMed publication ID2011791

P2093author name stringTurker MS
Cooper GE
DiMartino DL
P433issue2
P304page(s)105-116
P577publication date1991-03-01
P1433published inSomatic Cell and Molecular GeneticsQ7559232
P1476titleMolecular analysis of APRT deficiency in mouse P19 teratocarcinoma stem cell line
P478volume17

Reverse relations

cites work (P2860)
Q40727004A mouse kidney cell line with a G:C --> C:G transversion mutator phenotype
Q28512293A role for certain mouseAprt sequences in resistance to toxic adenine analogs
Q39638182Aberrantly silenced promoters retain a persistent memory of the silenced state after long-term reactivation
Q41557956Hemidemethylation is sufficient for chromatin relaxation and transcriptional activation of methylatedaprt gene in mouse P19 embryonal carcinoma cell line
Q41505845Isolation and Characterization of Mutations in the Mouse APRT Gene that Encode Functional Enzymes with Resistance to Toxic Adenine Analogs
Q41592974Molecular and biochemical elucidation of a cellular phenotype characterized by adenine analogue resistance in the presence of high levels of adenine phosphoribosyltransferase activity
Q73217526Molecular evidence for the induction of large interstitial deletions on mouse chromosome 8 by ionizing radiation
Q48138932Region-specific rates of molecular evolution: a fourfold reduction in the rate of accumulation of "silent" mutations in transcribed versus nontranscribed regions of homologous DNA fragments derived from two closely related mouse species
Q41330895Spontaneous and ionizing radiation induced mutations involve large events when selecting for loss of an autosomal locus
Q40790684Spontaneously immortalized cell lines obtained from adult Atm null mice retain sensitivity to ionizing radiation and exhibit a mutational pattern suggestive of oxidative stress
Q41505861The Mouse APRT Gene as a Model for Studying Epigenetic Gene Inactivation
Q38331684The primary function of a redundant Sp1 binding site in the mouse aprt gene promoter is to block epigenetic gene inactivation

Search more.