scholarly article | Q13442814 |
P2093 | author name string | Christian Zuppinger | |
Thomas M Suter | |||
Francesco Timolati | |||
Maria-Iris Romay-Ogando | |||
Polychronis Dimitrakis | |||
P2860 | cites work | Aggregates of oxidized proteins (lipofuscin) induce apoptosis through proteasome inhibition and dysregulation of proapoptotic proteins | Q46394496 |
Atrogin-1 ubiquitin ligase is upregulated by doxorubicin via p38-MAP kinase in cardiac myocytes | Q46696596 | ||
Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes | Q23757358 | ||
LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing | Q24597817 | ||
Juvenile exposure to anthracyclines impairs cardiac progenitor cell function and vascularization resulting in greater susceptibility to stress-induced myocardial injury in adult mice | Q24632487 | ||
Dissection of autophagosome formation using Apg5-deficient mouse embryonic stem cells | Q24671346 | ||
Molecular determinants for the recruitment of the ubiquitin-ligase MuRF-1 onto M-line titin | Q27643507 | ||
The ubiquitin-proteasome system in cardiac physiology and pathology | Q28299196 | ||
Transcription factor GATA4 inhibits doxorubicin-induced autophagy and cardiomyocyte death | Q28579471 | ||
A critical evaluation of the mechanisms of action proposed for the antitumor effects of the anthracycline antibiotics adriamycin and daunorubicin | Q33540751 | ||
Activation of the ubiquitin-proteasome system in doxorubicin cardiomyopathy. | Q33699632 | ||
Mechanisms of anthracycline cardiac injury: can we identify strategies for cardioprotection? | Q34102834 | ||
Autophagy: cellular and molecular mechanisms | Q34103624 | ||
Autophagy: assays and artifacts | Q34341096 | ||
Differences in doxorubicin-induced apoptotic signaling in adult and immature cardiomyocytes | Q34576324 | ||
Autophagy as a therapeutic target in cardiovascular disease | Q35223861 | ||
Cardiac autophagy is a maladaptive response to hemodynamic stress | Q35839989 | ||
Decreased proteolysis caused by protein aggregates, inclusion bodies, plaques, lipofuscin, ceroid, and 'aggresomes' during oxidative stress, aging, and disease. | Q35869405 | ||
New insights into doxorubicin-induced cardiotoxicity: the critical role of cellular energetics. | Q36552392 | ||
Tuning flux: autophagy as a target of heart disease therapy | Q36714637 | ||
Role of mtDNA lesions in anthracycline cardiotoxicity | Q36891738 | ||
Protein quality control and degradation in cardiomyocytes | Q36952213 | ||
Intracellular protein aggregation is a proximal trigger of cardiomyocyte autophagy | Q37012635 | ||
The role of autophagy in the heart. | Q37325466 | ||
The role of cell death and myofibrillar damage in contractile dysfunction of long-term cultured adult cardiomyocytes exposed to doxorubicin | Q37480989 | ||
Role of autophagy in heart failure associated with aging | Q37728936 | ||
Anthracycline cardiotoxicity in the elderly cancer patient: a SIOG expert position paper. | Q37801220 | ||
Muscle actin is polyubiquitinylated in vitro and in vivo and targeted for breakdown by the E3 ligase MuRF1. | Q39505410 | ||
Protein metabolism of the heart | Q40170901 | ||
Cardiac toxicity 4 to 20 years after completing anthracycline therapy | Q41142694 | ||
Doxorubicin-induced apoptosis in spontaneously hypertensive rats: differential effects in heart, kidney and intestine, and inhibition by ICRF-187. | Q42552722 | ||
Adriamycin cardiotoxicity | Q42583095 | ||
Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes | Q43153846 | ||
Calcium signaling mechanisms in dedifferentiated cardiac myocytes: comparison with neonatal and adult cardiomyocytes. | Q43811952 | ||
Age-dependent declines in proteasome activity in the heart | Q43857550 | ||
Myocytes die by multiple mechanisms in failing human hearts | Q44370216 | ||
P433 | issue | 2 | |
P921 | main subject | doxorubicin | Q18936 |
autophagy | Q288322 | ||
ubiquitin-proteasome system | Q47175589 | ||
P304 | page(s) | 361-372 | |
P577 | publication date | 2012-08-04 | |
P1433 | published in | Cell and Tissue Research | Q1524113 |
P1476 | title | Effects of doxorubicin cancer therapy on autophagy and the ubiquitin-proteasome system in long-term cultured adult rat cardiomyocytes | |
P478 | volume | 350 |
Q89967798 | 3D Co-culture of hiPSC-Derived Cardiomyocytes With Cardiac Fibroblasts Improves Tissue-Like Features of Cardiac Spheroids |
Q36466224 | Advanced Glycation End Products (AGE) Potently Induce Autophagy through Activation of RAF Protein Kinase and Nuclear Factor κB (NF-κB). |
Q39120233 | Autophagic homeostasis is required for the pluripotency of cancer stem cells |
Q39264180 | Autophagy and mitophagy in the context of doxorubicin-induced cardiotoxicity. |
Q33573823 | Cardiomyocyte autophagy and cancer chemotherapy |
Q26780547 | Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress |
Q37116964 | Development and testing of gold nanoparticles for drug delivery and treatment of heart failure: a theranostic potential for PPP cardiology |
Q36869450 | Doxorubicin Blocks Cardiomyocyte Autophagic Flux by Inhibiting Lysosome Acidification. |
Q50965031 | Doxorubicin-induced markers of myocardial autophagic signaling in sedentary and exercise trained animals. |
Q32872193 | Exercise and Doxorubicin Treatment Modulate Cardiac Mitochondrial Quality Control Signaling. |
Q89964455 | Luteolin Attenuates Doxorubicin-Induced Cardiotoxicity Through Promoting Mitochondrial Autophagy |
Q33660939 | Nrf2 deficiency exaggerates doxorubicin-induced cardiotoxicity and cardiac dysfunction. |
Q30090238 | PICT-1 triggers a pro-death autophagy through inhibiting rRNA transcription and AKT/mTOR/p70S6K signaling pathway |
Q89520711 | Protective effect of flavonoids from Rosa roxburghii Tratt on myocardial cells via autophagy |
Q41861138 | Protective role of Osthole on myocardial cell apoptosis induced by doxorubicin in rats |
Q28304890 | Proteotoxicity: an underappreciated pathology in cardiac disease |
Q26801409 | Resveratrol, a polyphenol phytoalexin, protects against doxorubicin-induced cardiotoxicity |
Q41905674 | The nucleolar protein nucleophosmin is essential for autophagy induced by inhibiting Pol I transcription |
Q36670771 | Transcriptional induction of the heat shock protein B8 mediates the clearance of misfolded proteins responsible for motor neuron diseases |
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