scholarly article | Q13442814 |
P2093 | author name string | Yoko Tabe | |
Isao Nagaoka | |||
Hiroshi Tamura | |||
Toshiaki Iba | |||
Kyoko Kuwahara-Arai | |||
Taisuke Murakami | |||
Johannes Reich | |||
Zhongshuang Hu | |||
P2860 | cites work | Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicans | Q21090509 |
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Neutrophil serine proteases: specific regulators of inflammation | Q28248366 | ||
Neutrophil extracellular traps promote thrombin generation through platelet-dependent and platelet-independent mechanisms | Q87381493 | ||
Caspase-1 regulates Escherichia coli sepsis and splenic B cell apoptosis independently of interleukin-1beta and interleukin-18 | Q28257695 | ||
Macrophage clearance of neutrophil extracellular traps is a silent process | Q28295755 | ||
Pyroptosis: host cell death and inflammation | Q29615594 | ||
Neutrophils and immunity: challenges and opportunities | Q29617616 | ||
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The inflammasome recognizes cytosolic microbial and host DNA and triggers an innate immune response | Q29620414 | ||
The immunopathogenesis of sepsis | Q29622909 | ||
Neutrophil extracellular traps sequester circulating tumor cells and promote metastasis | Q30541882 | ||
IL-1beta processing in host defense: beyond the inflammasomes | Q33535878 | ||
Extracellular histones are major mediators of death in sepsis | Q34019901 | ||
Dying for a cause: NETosis, mechanisms behind an antimicrobial cell death modality | Q34162570 | ||
Neutrophil extracellular traps directly induce epithelial and endothelial cell death: a predominant role of histones | Q34184548 | ||
Extracellular histones are mediators of death through TLR2 and TLR4 in mouse fatal liver injury | Q35175597 | ||
Deoxyribonuclease is a potential counter regulator of aberrant neutrophil extracellular traps formation after major trauma | Q35725494 | ||
Caspase-8 as an Effector and Regulator of NLRP3 Inflammasome Signaling | Q35953090 | ||
Cutting edge: IL-1β processing during Pseudomonas aeruginosa infection is mediated by neutrophil serine proteases and is independent of NLRC4 and caspase-1. | Q36351389 | ||
Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock | Q36596738 | ||
Apoptosis and caspases regulate death and inflammation in sepsis | Q36622618 | ||
Depletion of neutrophil extracellular traps in vivo results in hypersusceptibility to polymicrobial sepsis in mice | Q36633941 | ||
AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASC. | Q37090120 | ||
Cell death during sepsis: integration of disintegration in the inflammatory response to overwhelming infection | Q37387080 | ||
Antimicrobial cathelicidin peptide LL-37 inhibits the LPS/ATP-induced pyroptosis of macrophages by dual mechanism | Q37488081 | ||
Molecular mechanisms involved in inflammasome activation | Q37588947 | ||
Cutting edge: Endoplasmic reticulum stress licenses macrophages to produce mature IL-1β in response to TLR4 stimulation through a caspase-8- and TRIF-dependent pathway | Q37604958 | ||
IL-1 pathways in inflammation and human diseases | Q37696955 | ||
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Modeling nuclear molecule release during in vitro cell death. | Q38067757 | ||
Targeting neutrophil apoptosis for enhancing the resolution of inflammation | Q38202818 | ||
Neutrophil extracellular traps in sepsis | Q38227298 | ||
Stimulation of Toll-like receptor 3 and 4 induces interleukin-1beta maturation by caspase-8. | Q39947388 | ||
Toll-like receptor 4 plays a role in macrophage phagocytosis during peritoneal sepsis | Q40121684 | ||
Delayed but not Early Treatment with DNase Reduces Organ Damage and Improves Outcome in a Murine Model of Sepsis | Q41203449 | ||
Antimicrobial cathelicidin peptide LL-37 inhibits the pyroptosis of macrophages and improves the survival of polybacterial septic mice | Q41949365 | ||
Intravascular neutrophil extracellular traps capture bacteria from the bloodstream during sepsis | Q42512724 | ||
Proinflammatory role of neutrophil extracellular traps in abdominal sepsis. | Q50451884 | ||
Mycobacterium tuberculosis- induced neutrophil extracellular traps activate human macrophages. | Q51026879 | ||
Aggregated neutrophil extracellular traps limit inflammation by degrading cytokines and chemokines. | Q51726697 | ||
Inhibitor of apoptosis proteins limit RIP3 kinase-dependent interleukin-1 activation. | Q53183279 | ||
Plasma cytokine and endotoxin levels correlate with survival in patients with the sepsis syndrome | Q72939014 | ||
Sepsis | Q80150077 | ||
P921 | main subject | lipopolysaccharide | Q421804 |
Neutrophil extracellular traps | Q7003141 | ||
P577 | publication date | 2017-01-27 | |
P1433 | published in | International Journal of Molecular Medicine | Q6051492 |
P1476 | title | Neutrophil extracellular traps induce IL-1β production by macrophages in combination with lipopolysaccharide |
Q59791521 | Betaine Inhibits Interleukin-1β Production and Release: Potential Mechanisms |
Q64104047 | Cold-inducible RNA-binding Protein Induces Neutrophil Extracellular Traps in the Lungs during Sepsis |
Q60918203 | Increased neutrophil extracellular traps activate NLRP3 and inflammatory macrophages in adult-onset Still's disease |
Q90100427 | Intra- and Extracellular Degradation of Neutrophil Extracellular Traps by Macrophages and Dendritic Cells |
Q100568733 | NET-associated citrullinated histones promote LDL aggregation and foam cell formation in vitro |
Q90234207 | Neutrophil extracellular traps activate IL-8 and IL-1 expression in human bronchial epithelia |
Q55018449 | Neutrophil extracellular traps promote macrophage pyroptosis in sepsis. |
Q48109327 | Recent advances in pathophysiology of disseminated intravascular coagulation: the role of circulating histones and neutrophil extracellular traps |
Q96167094 | Surgical trauma-induced immunosuppression in cancer: Recent advances and the potential therapies |
Q61807028 | The lncRNA Connection Between Cellular Metabolism and Epigenetics in Trained Immunity |
Q90388955 | l-Serine Lowers the Inflammatory Responses during Pasteurella multocida Infection |
Q47652559 | β-Nitrostyrene derivatives attenuate LPS-mediated acute lung injury via the inhibition of neutrophil-platelet interactions and NET release |
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