| scholarly article | Q13442814 |
| P6179 | Dimensions Publication ID | 1046317111 |
| P356 | DOI | 10.1038/CR.2016.87 |
| P2888 | exact match | https://scigraph.springernature.com/pub.10.1038/cr.2016.87 |
| P8608 | Fatcat ID | release_j6rfzbkmhrhhzo7nb7ebrrsvcq |
| P932 | PMC publication ID | 4973336 |
| P698 | PubMed publication ID | 27444869 |
| P2093 | author name string | Tao Li | |
| Swati Choksi | |||
| Xue-Min Zhang | |||
| Zheng-Gang Liu | |||
| Zhenyu Cai | |||
| Weihua Li | |||
| Anling Zhang | |||
| P2860 | cites work | TACE activation by MAPK-mediated regulation of cell surface dimerization and TIMP3 association. | Q34626693 |
| The ADAMs family of metalloproteases: multidomain proteins with multiple functions | Q35039313 | ||
| A Staphylococcus aureus pore-forming toxin subverts the activity of ADAM10 to cause lethal infection in mice | Q35332249 | ||
| ADAM9 inhibition increases membrane activity of ADAM10 and controls α-secretase processing of amyloid precursor protein | Q35562470 | ||
| Necrotic death as a cell fate | Q36359618 | ||
| Tissue organization by cadherin adhesion molecules: dynamic molecular and cellular mechanisms of morphogenetic regulation | Q36567245 | ||
| The "a disintegrin and metalloprotease" (ADAM) family of sheddases: physiological and cellular functions | Q37339450 | ||
| Necrotic cells trigger a sterile inflammatory response through the Nlrp3 inflammasome | Q37453447 | ||
| Dishonorable discharge: the oncogenic roles of cleaved E-cadherin fragments | Q38015669 | ||
| Pore-forming bacterial toxins and antimicrobial peptides as modulators of ADAM function | Q38042836 | ||
| Metalloproteinases and their natural inhibitors in inflammation and immunity. | Q38130854 | ||
| MLKL compromises plasma membrane integrity by binding to phosphatidylinositol phosphates. | Q38996643 | ||
| Identification of a key pathway required for the sterile inflammatory response triggered by dying cells | Q40119160 | ||
| Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation | Q44710589 | ||
| NEDD4 ubiquitinates TRAF3 to promote CD40-mediated AKT activation | Q46195385 | ||
| FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation. | Q52614520 | ||
| Expression of full-length desmosomol glycoproteins (desmocollins) is not sufficient to confer strong adhesion on transfected L929 cells | Q71049525 | ||
| N-cadherin expressed on malignant T cell lymphoma cells is functional, and promotes heterotypic adhesion between the lymphoma cells and mesenchymal cells expressing N-cadherin | Q77787167 | ||
| Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule | Q24290576 | ||
| Orai1 is an essential pore subunit of the CRAC channel | Q24300383 | ||
| Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase | Q24302381 | ||
| Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis | Q24311815 | ||
| Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha | Q24338113 | ||
| Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation | Q24338129 | ||
| Programmed necrosis in the cross talk of cell death and inflammation | Q27026709 | ||
| A HIF-1 target, ATIA, protects cells from apoptosis by modulating the mitochondrial thioredoxin, TRX2 | Q28588113 | ||
| RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis | Q28594624 | ||
| Translocation of mixed lineage kinase domain-like protein to plasma membrane leads to necrotic cell death | Q28660873 | ||
| Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury | Q29616008 | ||
| Role of a disintegrin and metalloprotease 10 in Staphylococcus aureus alpha-hemolysin-mediated cellular injury | Q30156106 | ||
| ADAM10 mediates E-cadherin shedding and regulates epithelial cell-cell adhesion, migration, and beta-catenin translocation. | Q33878896 | ||
| Necroptosis: the release of damage-associated molecular patterns and its physiological relevance | Q34035754 | ||
| Regulated necrosis: the expanding network of non-apoptotic cell death pathways. | Q34039639 | ||
| Adherens junctions: from molecules to morphogenesis | Q34122302 | ||
| E-cadherin is crucial for embryonic stem cell pluripotency and can replace OCT4 during somatic cell reprogramming | Q34187775 | ||
| Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis | Q34197341 | ||
| Mixed lineage kinase domain-like protein MLKL causes necrotic membrane disruption upon phosphorylation by RIP3. | Q34413744 | ||
| Membrane-enabled dimerization of the intrinsically disordered cytoplasmic domain of ADAM10. | Q34526248 | ||
| P433 | issue | 8 | |
| P921 | main subject | necroptosis | Q3346925 |
| P304 | page(s) | 886-900 | |
| P577 | publication date | 2016-07-22 | |
| P1433 | published in | Cell Research | Q1524018 |
| P1476 | title | Activation of cell-surface proteases promotes necroptosis, inflammation and cell migration | |
| P478 | volume | 26 |
| Q49875840 | A Disintegrin and Metalloprotease-22 Attenuates Hypertrophic Remodeling in Mice Through Inhibition of the Protein Kinase B Signaling Pathway. |
| Q50190887 | Biological events and molecular signaling following MLKL activation during necroptosis. |
| Q91260101 | Current translational potential and underlying molecular mechanisms of necroptosis |
| Q89640262 | Depletion of MLKL inhibits invasion of radioresistant nasopharyngeal carcinoma cells by suppressing epithelial-mesenchymal transition |
| Q47551118 | Epstein-Barr virus encoded latent membrane protein 1 suppresses necroptosis through targeting RIPK1/3 ubiquitination |
| Q89790755 | Inhibition of keratinocyte necroptosis mediated by RIPK1/RIPK3/MLKL provides a protective effect against psoriatic inflammation |
| Q47843948 | Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. |
| Q41048038 | Monitoring proteolytic processing events by quantitative mass spectrometry. |
| Q48171705 | Necroptosis Resumes Apoptosis in Hippocampus but Not in Frontal Cortex. |
| Q93061867 | Necroptosis in pancreatic cancer promotes cancer cell migration and invasion by release of CXCL5 |
| Q92544553 | Necroptosis, tumor necrosis and tumorigenesis |
| Q47659073 | Necroptotic cell death in anti-cancer therapy |
| Q64067286 | NrCAM is a marker for substrate-selective activation of ADAM10 in Alzheimer's disease |
| Q50094523 | Plasma membrane changes during programmed cell deaths. |
| Q55333214 | Targeting CPT1A-mediated fatty acid oxidation sensitizes nasopharyngeal carcinoma to radiation therapy. |
| Q38723480 | The effects and regulatory mechanism of RIP3 on RGC-5 necroptosis following elevated hydrostatic pressure |
| Q52645849 | The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis. |
| Q90152760 | To the edge of cell death and back |
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