scholarly article | Q13442814 |
P50 | author | Zofia M Chrzanowska-Lightowlers | Q39034391 |
P2093 | author name string | Robert N Lightowlers | |
Richard J Temperley | |||
Paul M Smith | |||
Sara H Seneca | |||
P2860 | cites work | Identification and cloning of human mitochondrial translational release factor 1 and the ribosome recycling factor | Q22008466 |
Identification and characterization of mammalian mitochondrial tRNA nucleotidyltransferases | Q24291562 | ||
Termination of translation: interplay of mRNA, rRNAs and release factors? | Q24540309 | ||
Reduced-median-network analysis of complete mitochondrial DNA coding-region sequences for the major African, Asian, and European haplogroups | Q24563892 | ||
Sequence and organization of the human mitochondrial genome | Q27860659 | ||
Reanalysis and revision of the Cambridge reference sequence for human mitochondrial DNA | Q27860870 | ||
Exosome-mediated recognition and degradation of mRNAs lacking a termination codon | Q27939297 | ||
Investigation of a pathogenic mtDNA microdeletion reveals a translation-dependent deadenylation decay pathway in human mitochondria | Q28201407 | ||
Mitochondrial respiratory-chain diseases | Q29614474 | ||
tRNA punctuation model of RNA processing in human mitochondria | Q29616348 | ||
Role of a peptide tagging system in degradation of proteins synthesized from damaged messenger RNA | Q29617951 | ||
Blue native electrophoresis for isolation of membrane protein complexes in enzymatically active form | Q29619372 | ||
Making sense of mimic in translation termination | Q33338049 | ||
The RNase P associated with HeLa cell mitochondria contains an essential RNA component identical in sequence to that of the nuclear RNase P. | Q33966946 | ||
An mRNA surveillance mechanism that eliminates transcripts lacking termination codons | Q34120142 | ||
Quality control of mRNA function | Q34156109 | ||
Epidemiology and treatment of mitochondrial disorders | Q34386766 | ||
Blue Native electrophoresis to study mitochondrial and other protein complexes | Q34675865 | ||
Mitochondrial encephalomyopathies | Q35085834 | ||
Class-1 translation termination factors: invariant GGQ minidomain is essential for release activity and ribosome binding but not for stop codon recognition | Q39229931 | ||
Mitochondrial dysfunction in a cell culture model of familial amyotrophic lateral sclerosis | Q40723426 | ||
In vivo labeling and analysis of human mitochondrial translation products | Q41244321 | ||
A bacterial RNA that functions as both a tRNA and an mRNA. | Q41710118 | ||
Mutations in the highly conserved GGQ motif of class 1 polypeptide release factors abolish ability of human eRF1 to trigger peptidyl-tRNA hydrolysis. | Q43206485 | ||
Post-transcriptional addition of polyadenylic acid to mitochondrial RNA by a cordycepin-insensitive process | Q43643234 | ||
Human polynucleotide phosphorylase, hPNPase, is localized in mitochondria. | Q54523690 | ||
A mitochondrial DNA microdeletion in a newborn girl with transient lactic acidosis | Q71345252 | ||
Studies on mitochondrial oxidative phosphorylation in permeabilized human skin fibroblasts: application to mitochondrial encephalomyopathies | Q72799018 | ||
Interaction of mitochondrial elongation factor Tu with aminoacyl-tRNA and elongation factor Ts | Q73763064 | ||
The epidemiology of pathogenic mitochondrial DNA mutations | Q74159368 | ||
Decreased ATP synthesis is phenotypically expressed during increased energy demand in fibroblasts containing mitochondrial tRNA mutations | Q77892921 | ||
P433 | issue | Pt 3 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 725-731 | |
P577 | publication date | 2004-02-01 | |
P1433 | published in | Biochemical Journal | Q864221 |
P1476 | title | Functional polypeptides can be synthesized from human mitochondrial transcripts lacking termination codons | |
P478 | volume | 377 |
Q33506537 | A unique genetic code change in the mitochondrial genome of the parasitic nematode Radopholus similis |
Q38920032 | Alteration of structure and function of ATP synthase and cytochrome c oxidase by lack of Fo-a and Cox3 subunits caused by mitochondrial DNA 9205delTA mutation |
Q33300478 | Broad genomic and transcriptional analysis reveals a highly derived genome in dinoflagellate mitochondria |
Q33316556 | Development of a human mitochondrial oligonucleotide microarray (h-MitoArray) and gene expression analysis of fibroblast cell lines from 13 patients with isolated F1Fo ATP synthase deficiency. |
Q42806022 | Diminished synthesis of subunit a (ATP6) and altered function of ATP synthase and cytochrome c oxidase due to the mtDNA 2 bp microdeletion of TA at positions 9205 and 9206. |
Q44673190 | Exaggerated status of "novel" and "pathogenic" mtDNA sequence variants due to inadequate database searches |
Q34772842 | HMRF1L is a human mitochondrial translation release factor involved in the decoding of the termination codons UAA and UAG. |
Q57910058 | Human Mitochondrial mRNAs Are Stabilized with Polyadenylation Regulated by Mitochondria-specific Poly(A) Polymerase and Polynucleotide Phosphorylase |
Q37693914 | Identification of a novel human nuclear-encoded mitochondrial poly(A) polymerase |
Q35840151 | Mitochondrial poly(A) polymerase and polyadenylation |
Q40226349 | Plant mitochondrial genes can be expressed from mRNAs lacking stop codons. |
Q24303855 | Stable PNPase RNAi silencing: its effect on the processing and adenylation of human mitochondrial RNA |
Q35781669 | Translation of nonSTOP mRNA is repressed post-initiation in mammalian cells |
Q35170120 | Turnover of ATP synthase subunits in F1-depleted HeLa and yeast cells |
Search more.