scholarly article | Q13442814 |
P50 | author | Arantxa Tabernero | Q42838796 |
P2093 | author name string | José M Medina | |
Rosa Sánchez-Alvarez | |||
P2860 | cites work | Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals | Q24314293 |
Connexin43: a protein from rat heart homologous to a gap junction protein from liver | Q24680192 | ||
Connexin 43 mediated gap junctional communication enhances breast tumor cell diapedesis in culture | Q24810763 | ||
A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding | Q25938984 | ||
The retinoblastoma protein and cell cycle control | Q27860722 | ||
p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21 | Q28505037 | ||
Colony-stimulating factor 1 regulates novel cyclins during the G1 phase of the cell cycle | Q33319961 | ||
The Ki-67 protein: fascinating forms and an unknown function | Q33334637 | ||
The Rb/E2F pathway: expanding roles and emerging paradigms | Q34052659 | ||
Gap junctions and tumour progression | Q34588622 | ||
Cyclin D-dependent kinases, INK4 inhibitors and cancer | Q34605533 | ||
Transport and phosphorylation of hexoses in normal and rous sarcoma virus-transformed chick embryo fibroblasts | Q40198378 | ||
D-type cyclins | Q40443885 | ||
Negative regulation of p21 by beta-catenin/TCF signaling: a novel mechanism by which cell adhesion molecules regulate cell proliferation | Q40613480 | ||
Growth inhibition in G(1) and altered expression of cyclin D1 and p27(kip-1 )after forced connexin expression in lung and liver carcinoma cells | Q40857018 | ||
Identification of tumor-suppressor genes using human melanoma cell lines UACC903, UACC903(+6), and SRS3 by comparison of expression profiles | Q40867761 | ||
Analysis of site-specific phosphorylation of the retinoblastoma protein during cell cycle progression. | Q40964495 | ||
The gap junction communication channel | Q40974984 | ||
Endothelin-1 regulates glucose utilization in cultured astrocytes by controlling intercellular communication through gap junctions | Q41220649 | ||
Aberrant glycolytic metabolism of cancer cells: a remarkable coordination of genetic, transcriptional, post-translational, and mutational events that lead to a critical role for type II hexokinase | Q41654938 | ||
Incorporation of the gene for a cell-cell channel protein into transformed cells leads to normalization of growth | Q41655636 | ||
Lipogenesis from lactate in rat neurons and astrocytes in primary culture | Q42811876 | ||
The enhancement of glucose uptake caused by the collapse of gap junction communication is due to an increase in astrocyte proliferation | Q43718629 | ||
Proliferation of C6 glioma cells is blunted by the increase in gap junction communication caused by tolbutamide | Q43821363 | ||
Reversible inhibition of intercellular junctional communication by glycyrrhetinic acid | Q44325495 | ||
Effect of endothelin-1 on astrocytic protein content | Q44429854 | ||
Endothelin-1 decreases gap junctional intercellular communication by inducing phosphorylation of connexin 43 in human ovarian carcinoma cells | Q44540521 | ||
Endothelin-1 stimulates the translocation and upregulation of both glucose transporter and hexokinase in astrocytes: relationship with gap junctional communication | Q44846038 | ||
Astrocyte progression from G1 to S phase of the cell cycle depends upon multiple protein interaction | Q46163966 | ||
Expression of two type II-like tumor hexokinase RNA transcripts in cancer cell lines | Q47785008 | ||
The K-ATP channel regulates the effect of Ca2+ on gap junction permeability in cultured astrocytes | Q48012316 | ||
Reduced connexin43 expression in high-grade human brain glioma cells | Q48282845 | ||
Gap junctions in cultured astrocytes: Single-channel currents and characterization of channel-forming protein | Q48823097 | ||
Reduced expression of connexin-43 and functional gap junction coupling in human gliomas | Q49024828 | ||
Gene expression of GLUT3 and GLUT1 glucose transporters in human brain tumors. | Q55481614 | ||
Free calcium rise and mitogenesis in glial cells caused by endothelin. | Q55484913 | ||
P433 | issue | 4 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | astrocyte | Q502961 |
P304 | page(s) | 973-982 | |
P577 | publication date | 2006-01-12 | |
P1433 | published in | Journal of Neurochemistry | Q6295643 |
P1476 | title | Increased levels of cyclins D1 and D3 after inhibition of gap junctional communication in astrocytes | |
P478 | volume | 96 |
Q46341626 | A potential role of connexin 43 in epidermal growth factor-induced proliferation of mouse embryonic stem cells: involvement of Ca2+/PKC, p44/42 and p38 MAPKs pathways |
Q30436052 | Biological and biophysical properties of vascular connexin channels |
Q27322751 | Connexin 30 expression inhibits growth of human malignant gliomas but protects them against radiation therapy |
Q39672915 | Connexin43 inhibits the oncogenic activity of c-Src in C6 glioma cells |
Q46404427 | Connexin43 is involved in the effect of endothelin-1 on astrocyte proliferation and glucose uptake |
Q34288480 | Cooperativity and complementarity: synergies in non-classical and classical glucocorticoid signaling |
Q36583476 | Dynamic changes in protein interaction between AKAP95 and Cx43 during cell cycle progression of A549 cells |
Q36561137 | Glucose metabolism and proliferation in glia: role of astrocytic gap junctions |
Q34182724 | HIF-1 and c-Src mediate increased glucose uptake induced by endothelin-1 and connexin43 in astrocytes |
Q28387518 | Liposomal clodronate selectively eliminates microglia from primary astrocyte cultures |
Q48372793 | Reduced connexin43 expression correlates with c-Src activation, proliferation, and glucose uptake in reactive astrocytes after an excitotoxic insult |
Q36218243 | Rett syndrome astrocytes are abnormal and spread MeCP2 deficiency through gap junctions |
Q41684033 | Temporal Changes in Cortical and Hippocampal Expression of Genes Important for Brain Glucose Metabolism Following Controlled Cortical Impact Injury in Mice |
Q34262821 | The role of gap junction channels during physiologic and pathologic conditions of the human central nervous system |
Search more.