Habitual behavior and dopamine cell vulnerability in Parkinson disease

scientific article published on 6 August 2015

Habitual behavior and dopamine cell vulnerability in Parkinson disease is …
instance of (P31):
scholarly articleQ13442814
editorialQ871232

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P356DOI10.3389/FNANA.2015.00099
P932PMC publication ID4526812
P698PubMed publication ID26300740
P5875ResearchGate publication ID281485913

P2093author name stringPeter Redgrave
José A Obeso
Ledia F Hernández
P2860cites workThe progression of pathology in longitudinally followed patients with Parkinson's disease.Q51895882
Building neural representations of habits.Q52030807
Pathogenesis of Parkinson's diseaseQ84882756
Neuromelanin in human dopamine neurons: Comparison with peripheral melanins and relevance to Parkinson's diseaseQ22251216
Neuromelanin Accumulation with Age in Catecholaminergic Neurons from Macaca fascicularis BrainstemQ22337017
Two types of dopamine neuron distinctly convey positive and negative motivational signalsQ24658285
Oxidative stress and Parkinson's diseaseQ26798897
A neural substrate of prediction and rewardQ27860851
Large-scale meta-analysis of genome-wide association data identifies six new risk loci for Parkinson's diseaseQ28244731
Variability in neuronal expression of dopamine receptors and transporters in the substantia nigraQ28290596
'Rejuvenation' protects neurons in mouse models of Parkinson's diseaseQ28305585
Parkinson's disease: mechanisms and modelsQ29547424
Ageing and Parkinson's disease: substantia nigra regional selectivityQ29615835
The role of the basal ganglia in habit formationQ29619829
Robust pacemaking in substantia nigra dopaminergic neuronsQ30492001
Start/stop signals emerge in nigrostriatal circuits during sequence learningQ30525757
Learning and memory functions of the Basal GangliaQ34132332
Midbrain dopaminergic neurons in the mouse that contain calbindin-D28k exhibit reduced vulnerability to MPTP-induced neurodegenerationQ34422971
Striatonigrostriatal pathways in primates form an ascending spiral from the shell to the dorsolateral striatumQ34507515
Goal-directed and habitual control in the basal ganglia: implications for Parkinson's diseaseQ35072636
When brawn benefits brain: physical activity and Parkinson's disease riskQ35383208
The role of calcium and mitochondrial oxidant stress in the loss of substantia nigra pars compacta dopaminergic neurons in Parkinson's diseaseQ35628604
Calcium entry and α-synuclein inclusions elevate dendritic mitochondrial oxidant stress in dopaminergic neuronsQ36928454
Mitochondria in the aetiology and pathogenesis of Parkinson's diseaseQ37037953
The progression of pathology in Parkinson's diseaseQ37690360
Patterns of α-synuclein pathology in incidental cases and clinical subtypes of Parkinson's diseaseQ37968067
Neuronal vulnerability, pathogenesis, and Parkinson's diseaseQ38099288
Glucocerebrosidase mutations and the pathogenesis of Parkinson diseaseQ38161987
The disease intersection of susceptibility and exposure: chemical exposures and neurodegenerative disease riskQ38219878
Living on the edge with too many mouths to feed: why dopamine neurons die.Q41732813
Activity of striatal neurons reflects dynamic encoding and recoding of procedural memoriesQ42485542
Progression of dopaminergic depletion in a model of MPTP-induced Parkinsonism in non-human primates. An (18)F-DOPA and (11)C-DTBZ PET studyQ43123132
Distinct representations of cognitive and motivational signals in midbrain dopamine neuronsQ43505389
Lesions of dorsolateral striatum preserve outcome expectancy but disrupt habit formation in instrumental learningQ44747862
Expression of dopamine and vesicular monoamine transporters and differential vulnerability of mesostriatal dopaminergic neuronsQ45081906
Single nigrostriatal dopaminergic neurons form widely spread and highly dense axonal arborizations in the neostriatum.Q46162532
The substantia nigra of the human brain. II. Patterns of loss of dopamine-containing neurons in Parkinson's diseaseQ48147372
Living and dying with Parkinson's diseaseQ48250448
Is Parkinson's disease a vesicular dopamine storage disorder? Evidence from a study in isolated synaptic vesicles of human and nonhuman primate striatumQ48301354
GBA-associated Parkinson's disease: reduced survival and more rapid progression in a prospective longitudinal studyQ48436320
Melanin, tyrosine hydroxylase, calbindin and substance P in the human midbrain and substantia nigra in relation to nigrostriatal projections and differential neuronal susceptibility in Parkinson's diseaseQ48478915
P2507corrigendum / erratumCorrigendum: Habitual Behavior and Dopamine Cell Vulnerability in Parkinson DiseaseQ43172542
P921main subjectParkinson's diseaseQ11085
P304page(s)99
P577publication date2015-08-06
P1433published inFrontiers in NeuroanatomyQ1893158
P1476titleHabitual behavior and dopamine cell vulnerability in Parkinson disease
P478volume9

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cites work (P2860)
Q41846573Editorial: Parkinson's disease: cell vulnerability and disease progression
Q42378714Pedunculopontine glutamatergic neurons control spike patterning in substantia nigra dopaminergic neurons.
Q41108071Sensory Processing in the Dorsolateral Striatum: The Contribution of Thalamostriatal Pathways

Q43172542Corrigendum: Habitual Behavior and Dopamine Cell Vulnerability in Parkinson Diseasemain subjectP921

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