| scholarly article | Q13442814 |
| P356 | DOI | 10.1073/PNAS.1615774114 |
| P8608 | Fatcat ID | release_c4tmmeuiljgqvgh5gtiplorjfa |
| P932 | PMC publication ID | 5321040 |
| P698 | PubMed publication ID | 28137877 |
| P2093 | author name string | Alfred L George | |
| Christopher H Thompson | |||
| Jennifer A Kearney | |||
| Nicole A Hawkins | |||
| P2860 | cites work | Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sites | Q24315218 |
| Noninactivating voltage-gated sodium channels in severe myoclonic epilepsy of infancy | Q24563329 | ||
| Molecular basis of an inherited epilepsy | Q28207595 | ||
| Deletion of the Kv2.1 delayed rectifier potassium channel leads to neuronal and behavioral hyperexcitability. | Q28510330 | ||
| A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice | Q28587278 | ||
| Voltage-gated potassium channel KCNV2 (Kv8.2) contributes to epilepsy susceptibility | Q30499291 | ||
| Confirmation of an epilepsy modifier locus on mouse chromosome 11 and candidate gene analysis by RNA-Seq | Q30515384 | ||
| Na+ channel regulation by Ca2+/calmodulin and Ca2+/calmodulin-dependent protein kinase II in guinea-pig ventricular myocytes | Q33573637 | ||
| Inherited disorders of voltage-gated sodium channels | Q33905815 | ||
| Novel SCN5A mutation in amiodarone-responsive multifocal ventricular ectopy-associated cardiomyopathy | Q33940611 | ||
| The action potential in mammalian central neurons | Q34004305 | ||
| Reciprocal changes in phosphorylation and methylation of mammalian brain sodium channels in response to seizures | Q34075908 | ||
| An EF-hand in the sodium channel couples intracellular calcium to cardiac excitability | Q34300731 | ||
| Identification of novel interaction sites that determine specificity between fibroblast growth factor homologous factors and voltage-gated sodium channels | Q35085135 | ||
| FGF14 modulates resurgent sodium current in mouse cerebellar Purkinje neurons | Q35166993 | ||
| Regulation of sodium channel activity by phosphorylation | Q36176745 | ||
| Sodium channel mutations in epilepsy and other neurological disorders | Q36216288 | ||
| Divergent sodium channel defects in familial hemiplegic migraine | Q36775272 | ||
| Polarized localization of voltage-gated Na+ channels is regulated by concerted FGF13 and FGF14 action | Q36905057 | ||
| Divergent biophysical defects caused by mutant sodium channels in dilated cardiomyopathy with arrhythmia | Q37019675 | ||
| Cacna1g is a genetic modifier of epilepsy caused by mutation of voltage-gated sodium channel Scn2a | Q37175540 | ||
| Scn2a sodium channel mutation results in hyperexcitability in the hippocampus in vitro | Q37286952 | ||
| CaMKII Phosphorylation of Na(V)1.5: Novel in Vitro Sites Identified by Mass Spectrometry and Reduced S516 Phosphorylation in Human Heart Failure | Q37601736 | ||
| Strain- and age-dependent hippocampal neuron sodium currents correlate with epilepsy severity in Dravet syndrome mice | Q37670832 | ||
| NaV1.1 channels and epilepsy | Q37701079 | ||
| Sodium channel gene family: epilepsy mutations, gene interactions and modifier effects | Q37721012 | ||
| Neuronal excitability and calcium/calmodulin-dependent protein kinase type II: location, location, location | Q38011833 | ||
| Seeing the forest through the trees: towards a unified view on physiological calcium regulation of voltage-gated sodium channels | Q38071198 | ||
| Hlf is a genetic modifier of epilepsy caused by voltage-gated sodium channel mutations | Q41059506 | ||
| SCN1A splice variants exhibit divergent sensitivity to commonly used antiepileptic drugs | Q42088430 | ||
| Intracellular calcium attenuates late current conducted by mutant human cardiac sodium channels | Q42234927 | ||
| Structural analyses of Ca²⁺/CaM interaction with NaV channel C-termini reveal mechanisms of calcium-dependent regulation | Q42908541 | ||
| Status epilepticus results in an N-methyl-D-aspartate receptor-dependent inhibition of Ca2+/calmodulin-dependent kinase II activity in the rat. | Q44868458 | ||
| A mechanism for the inactivation of Ca2+/calmodulin-dependent protein kinase II during prolonged seizure activity and its consequence after the recovery from seizure activity in rats in vivo | Q48571867 | ||
| A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities | Q49049294 | ||
| Genetic modifiers affecting severity of epilepsy caused by mutation of sodium channel Scn2a. | Q54634416 | ||
| P433 | issue | 7 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 1696-1701 | |
| P577 | publication date | 2017-01-30 | |
| P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
| P1476 | title | CaMKII modulates sodium current in neurons from epileptic Scn2a mutant mice | |
| P478 | volume | 114 |
| Q42630091 | Altered Norbin Expression in Patients with Epilepsy and a Rat Model |
| Q64040232 | Further corroboration of distinct functional features in SCN2A variants causing intellectual disability or epileptic phenotypes |
| Q57810027 | Gene expression profiling in a mouse model of Dravet syndrome |
| Q39457643 | Models for discovery of targeted therapy in genetic epileptic encephalopathies |
| Q57306574 | SCN1A variants associated with sudden infant death syndrome |
| Q91704914 | SCN2A channelopathies in the autism spectrum of neuropsychiatric disorders: a role for pluripotent stem cells? |
| Q58710088 | The Effect of Ca, Lobe-Specificity, and CaMKII on CaM Binding to Na1.1 |
| Q56365819 | The novel sodium channel modulator GS-458967 (GS967) is an effective treatment in a mouse model of SCN8A encephalopathy |
Search more.