| Q36956644 | 8. The relationship between electrographic seizure activity and neuronal injury |
| Q36956651 | 9. Excitotoxicity and epileptic brain damage |
| Q30658170 | A nuclear microscopic study of elemental changes in the rat hippocampus after kainate-induced neuronal injury |
| Q69831998 | Accumulation of calcium and loss of potassium in the hippocampus following transient cerebral ischemia: a proton microprobe study |
| Q46632903 | An in vitro model of anoxic-induced damage in mouse brain |
| Q38686417 | Arachidonic acid metabolism in seizures |
| Q40725978 | Bim, Bad, and Bax: a deadly combination in epileptic seizures |
| Q34245488 | Blockade of N -Methyl-D-Aspartate Receptors May Protect Against Ischemic Damage in the Brain |
| Q73483725 | CNS oxidative stress associated with the kainic acid rodent model of experimental epilepsy |
| Q31881842 | Ca(2+) entry through L-type Ca(2+) channels helps terminate epileptiform activity by activation of a Ca(2+) dependent afterhyperpolarisation in hippocampal CA3. |
| Q48628024 | Calcium overload in selectively vulnerable neurons of the hippocampus during and after ischemia: an electron microscopy study in the rat. |
| Q39440832 | Cellular and molecular events underlying epileptic brain damage. |
| Q68032482 | Cerebral perfusion of metabolic inactivators: a new method for rapid fixation of labile lipid pools in brain |
| Q42523142 | Chandelier cells in the auditory cortex of monkey and man: a Golgi study |
| Q48654640 | Changes in regional neurotransmitter amino acid levels in rat brain during seizures induced by L-allylglycine, bicuculline, and kainic acid |
| Q34763839 | Concept of activity-induced cell death in epilepsy: historical and contemporary perspectives |
| Q37269758 | Contribution of a mitochondrial pathway to excitotoxic neuronal necrosis |
| Q69963021 | Decrease in GABA immunoreactivity and alteration of GABA metabolism after kindling in the rat hippocampus |
| Q41345574 | Differential alteration of dopamine, acetylcholine, and glutamate release during anoxia and/or 3,4-diaminopyridine treatment |
| Q47104140 | Diverse Effects of an Acetylcholinesterase Inhibitor, Donepezil, on Hippocampal Neuronal Death after Pilocarpine-Induced Seizure |
| Q68506860 | Early Postischemic 45Ca Accumulation in Rat Dentate Hilus |
| Q48578715 | Effect of chlorpromazine on the localization of cAMP phosphodiesterase |
| Q48609930 | Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral Microdialysis |
| Q38608620 | Epilepsy: relationships between electrophysiology and intracellular mechanisms involving second messengers and gene expression |
| Q40444545 | Excitatory Amino Acid Receptors and Their Role in Epilepsy and Cerebral Ischemia |
| Q42437849 | Excitotoxic lesioning of the rat basal forebrain with S-AMPA: consequent mineralization and associated glial response. |
| Q45017796 | Excitotoxic mechanisms in hypoglycaemic hippocampal injury |
| Q40807326 | Excitotoxicity and selective neuronal loss in epilepsy |
| Q35225845 | Experimental complex partial seizures induced by a microinjection of kainic acid into limbic structures |
| Q43830916 | Formation of the Apaf-1/cytochrome c complex precedes activation of caspase-9 during seizure-induced neuronal death |
| Q94544485 | Going the Extra (Synaptic) Mile: Excitotoxicity as the Road Toward Neurodegenerative Diseases |
| Q67662663 | Golgi-like demonstration of "dark" neurons with an argyrophil III method for experimental neuropathology |
| Q34114403 | Hippocampal lesions in epilepsy: a historical review |
| Q44046532 | Hippocampal neuronal density in temporal lobe epilepsy with and without gliomas |
| Q40708029 | Hyperthermia aggravates and hypothermia ameliorates epileptic brain damage |
| Q70727941 | Immunohistochemical localization of extravasated serum albumin in the hippocampus of human subjects with partial and generalized epilepsies and epileptiform convulsions |
| Q72140318 | In vitro ischemia-induced intracellular Ca2+ elevation in cerebellar slices: a comparative study with the values found in hippocampal slices |
| Q30833898 | In vitro status epilepticus causes sustained elevation of intracellular calcium levels in hippocampal neurons |
| Q68274158 | Inhibition of sodium-potassium-ATPase: a potentially ubiquitous mechanism contributing to central nervous system neuropathology |
| Q44189968 | Intracellular calcium levels in canine basilar artery smooth muscle following experimental subarachnoid hemorrhage: an electron microscopic cytochemical study |
| Q48392256 | Intraendothelial accumulation of calcium in the hippocampus and thalamus of rats after systemic kainic acid administration |
| Q42458117 | Kainic acid seizures and the reversibility of calcium loading in vulnerable neurons in the hippocampus |
| Q48120871 | Long-term development of selective neuronal loss and the mechanism of protection by 2-amino-7-phosphonoheptanoate in a rat model of incomplete forebrain ischaemia |
| Q41660762 | Metabolic and Homeostatic Changes in Seizures and Acquired Epilepsy-Mitochondria, Calcium Dynamics and Reactive Oxygen Species |
| Q41182685 | Mitochondrial Dysfunction Mediated by Poly(ADP-Ribose) Polymerase-1 Activation Contributes to Hippocampal Neuronal Damage Following Status Epilepticus. |
| Q60949053 | Mitochondrial Metabolism in Major Neurological Diseases |
| Q43838192 | Neuronal alterations in hippocampus following severe hypoglycaemia: a light microscopic and ultrastructural study in the rat. |
| Q39588887 | Neuropathological consequences of chemically and electrically induced seizures |
| Q40772151 | Pathogenesis of mesial temporal sclerosis. |
| Q48882015 | Pathologic features of dysplasia and accompanying alterations observed in surgical specimens from patients with intractable epilepsy |
| Q40852693 | Pathophysiological mechanisms of brain damage from status epilepticus |
| Q48846946 | Pentylenetetrazol-induced kindling: early involvement of excitatory and inhibitory systems |
| Q51791866 | Probable Ca2+-mediated inactivation of Ca2+ currents in mammalian brain neurons. |
| Q46574453 | Programmed neuronal necrosis and status epilepticus |
| Q69870236 | Regional changes in transmitter amino acids during focal and generalized seizures in rats |
| Q43785285 | Seizure activity results in increased tyrosine phosphorylation of the N-methyl-D-aspartate receptor in the hippocampus |
| Q69385841 | Seizure-induced damage to substantia nigra and globus pallidus is accompanied by pronounced intra- and extracellular acidosis |
| Q48095986 | Selective damage in striatum and hippocampus with in vitro anoxia |
| Q37105984 | Status epilepticus: the past and the future |
| Q49090222 | Subcellular distribution of calcium and ultrastructural changes after cerebral hypoxia-ischemia in immature rats |
| Q93533168 | Substantia nigra lesions in mercaptopropionic acid induced status epilepticus: a light and electron microscopic study |
| Q33643179 | The Ca2+/calmodulin signaling system in the neural response to excitability. Involvement of neuronal and glial cells |
| Q48131356 | The nature and timing of excitotoxic neuronal necrosis in the cerebral cortex, hippocampus and thalamus due to flurothyl-induced status epilepticus |
| Q38429588 | The role of excitotoxic programmed necrosis in acute brain injury |
| Q36477555 | Time-course of changes in water, sodium, potassium and calcium contents of various brain regions in rats after systemic kainic acid administration |