scholarly article | Q13442814 |
P50 | author | Brian D. Lowes | Q39486752 |
P2093 | author name string | JoAnn Lindenfeld | |
Michael R Bristow | |||
Mark Geraci | |||
Eugene E Wolfel | |||
Joseph Cleveland | |||
Michael Reed | |||
Andreas Brieke | |||
Ronald Zolty | |||
Norman Gray | |||
Simon F Shakar | |||
Wayne Minobe | |||
Mihail Calalb | |||
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Inflammatory mediators and the failing heart: past, present, and the foreseeable future | Q35011415 | ||
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Long-term destination therapy with the HeartMate XVE left ventricular assist device: improved outcomes since the REMATCH study | Q43558666 | ||
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Effects of sustained-release moxonidine, an imidazoline agonist, on plasma norepinephrine in patients with chronic heart failure | Q43961153 | ||
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The structural examination of myocardial samples from patients with end-stage heart failure supported by ventricular assist devices using electron microscopy and amino acid analysis reveals low degree of reverse remodeling | Q44829171 | ||
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Carvedilol prevents cardiac hypertrophy and overexpression of hypoxia-inducible factor-1alpha and vascular endothelial growth factor in pressure-overloaded rat heart | Q46534000 | ||
Gene profiling changes in cytoskeletal proteins during clinical recovery after left ventricular-assist device support | Q46701008 | ||
Differential gene expression and genomic patient stratification following left ventricular assist device support | Q47990565 | ||
Sarcomeric genes involved in reverse remodeling of the heart during left ventricular assist device support | Q49142828 | ||
Long-term results in patients with idiopathic dilated cardiomyopathy after weaning from left ventricular assist devices. | Q51355859 | ||
Influence of prolonged ventricular assistance on myocardial histopathology in intact heart | Q70928470 | ||
Left ventricular echocardiographic and histologic changes: impact of chronic unloading by an implantable ventricular assist device | Q71038269 | ||
The effect of load on atrophy, myosin isoform shifts and contractile function: studies in a novel rat heart transplant preparation | Q72262217 | ||
Chronic unloading by left ventricular assist device reverses contractile dysfunction and alters gene expression in end-stage heart failure | Q73230419 | ||
Decreased SLIM1 expression and increased gelsolin expression in failing human hearts measured by high-density oligonucleotide arrays | Q73306462 | ||
Signaling pathways responsible for fetal gene induction in the failing human heart: evidence for altered thyroid hormone receptor gene expression | Q73549977 | ||
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Myocardial gene transfer and overexpression of beta2-adrenergic receptors potentiates the functional recovery of unloaded failing hearts | Q74397788 | ||
Comparison of exercise performance in patients with chronic severe heart failure versus left ventricular assist devices | Q77319721 | ||
Effects of carvedilol on left ventricular mass, chamber geometry, and mitral regurgitation in chronic heart failure | Q77361698 | ||
Left ventricular assist devices and the failing heart: a bridge to recovery, a permanent assist device, or a bridge too far? | Q77614521 | ||
Atrophy of myocardium and its myocytes by left ventricular assist device | Q78197916 | ||
Phospholamban ablation by RNA interference increases Ca2+ uptake into rat cardiac myocyte sarcoplasmic reticulum | Q80532852 | ||
Pharmacological stimulation of beta2-adrenergic receptors (beta2AR) enhances therapeutic effectiveness of beta1AR blockade in rodent dilated ischemic cardiomyopathy | Q83048014 | ||
HIF-1: hypoxia-inducible factor or dysoxia-inducible factor? | Q83289607 | ||
Specific and sustained down-regulation of genes involved in fatty acid metabolism is not a hallmark of progression to cardiac failure in mice | Q83333541 | ||
P433 | issue | 11 | |
P304 | page(s) | 1170-1176 | |
P577 | publication date | 2007-11-01 | |
P1433 | published in | The Journal of Heart and Lung Transplantation | Q2364382 |
P1476 | title | Assist devices fail to reverse patterns of fetal gene expression despite beta-blockers | |
P478 | volume | 26 |
Q51944941 | CELF-mediated alternative splicing is required for cardiac function during early, but not later, postnatal life. |
Q47096841 | Hypoxia favors myosin heavy chain beta gene expression in an Hif-1alpha-dependent manner. |
Q37995890 | Is myocardial recovery possible and how do you measure it? |
Q38792510 | Left ventricular assist device-induced reverse remodeling: it's not just about myocardial recovery |
Q36065639 | Reverse remodeling with left ventricular assist devices: a review of clinical, cellular, and molecular effects |
Q49686229 | Right ventricular involution: What can we learn from nature's model of compensated hypertrophy? |
Q39156232 | Targeted myocardial gene expression in failing hearts by RNA sequencing |
Q38223705 | The Notch pathway: a novel target for myocardial remodelling therapy? |
Q28081504 | The role of notch in the cardiovascular system: potential adverse effects of investigational notch inhibitors |
Q38294845 | β-myosin heavy chain is induced by pressure overload in a minor subpopulation of smaller mouse cardiac myocytes |
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