| scholarly article | Q13442814 |
| P356 | DOI | 10.1002/GCC.20516 |
| P698 | PubMed publication ID | 18008368 |
| P2093 | author name string | Christian Sutter | |
| Peter Schirmacher | |||
| Gunhild Mechtersheimer | |||
| Roland Penzel | |||
| Matthias Kloor | |||
| Ralf J Rieker | |||
| Hendrik Bläker | |||
| Michael A Kern | |||
| Christian Hertkorn | |||
| P2860 | cites work | Genomic alterations in distal bile duct carcinoma by comparative genomic hybridization and karyotype analysis | Q73031231 |
| Revised Bethesda Guidelines for hereditary nonpolyposis colorectal cancer (Lynch syndrome) and microsatellite instability | Q24618618 | ||
| A genetic model for colorectal tumorigenesis | Q27860582 | ||
| The ABC of APC | Q28205388 | ||
| Progenetix.net: an online repository for molecular cytogenetic aberration data | Q30667230 | ||
| Allelotype of colorectal carcinomas | Q34461209 | ||
| AFAP: variety is the spice of life | Q35352693 | ||
| Identification of mismatch repair gene mutations in young patients with colorectal cancer and in patients with multiple tumours associated with hereditary non-polyposis colorectal cancer. | Q35760971 | ||
| Germline, somatic and epigenetic events underlying mismatch repair deficiency in colorectal and HNPCC-related cancers. | Q38361824 | ||
| Large-scale allelotype of pancreaticobiliary carcinoma provides quantitative estimates of genome-wide allelic loss | Q40590830 | ||
| Deletion of chromosomal region 6q14-16 in prostate cancer | Q40696693 | ||
| How many mutations in a cancer? | Q41834410 | ||
| The type of somatic mutation at APC in familial adenomatous polyposis is determined by the site of the germline mutation: a new facet to Knudson's 'two-hit' hypothesis | Q44463232 | ||
| T25 repeat in the 3' untranslated region of the CASP2 gene: a sensitive and specific marker for microsatellite instability in colorectal cancer | Q46706099 | ||
| Genetics of adenocarcinomas of the small intestine: frequent deletions at chromosome 18q and mutations of the SMAD4 gene. | Q52545014 | ||
| Gene conversion is a frequent mechanism of inactivation of the wild-type allele in cancers from MLH1/MSH2 deletion carriers. | Q53350238 | ||
| Genome-wide differences between microsatellite stable and unstable colorectal tumors. | Q53352742 | ||
| Molecular genetic alterations and clinical features in early-onset colorectal carcinomas and their role for the recognition of hereditary cancer syndromes. | Q54478476 | ||
| Chromosomal instability in flat adenomas and carcinomas of the colon. | Q54682707 | ||
| Use of Molecular Tumor Characteristics to Prioritize Mismatch Repair Gene Testing in Early-Onset Colorectal Cancer | Q57265834 | ||
| A National Cancer Institute Workshop on Hereditary Nonpolyposis Colorectal Cancer Syndrome: Meeting Highlights and Bethesda Guidelines | Q57978042 | ||
| The risk of upper gastrointestinal cancer in familial adenomatous polyposis | Q67488531 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | susceptibility locus | Q62091149 |
| P304 | page(s) | 159-164 | |
| P577 | publication date | 2008-02-01 | |
| P1433 | published in | Genes, Chromosomes and Cancer | Q5532697 |
| P1476 | title | Recurrent deletions at 6q in early age of onset non-HNPCC- and non-FAP-associated intestinal carcinomas. Evidence for a novel cancer susceptibility locus at 6q14-q22. | |
| P478 | volume | 47 |
| Q84567714 | A study on MSH2 and MLH1 mutations in hereditary nonpolyposis colorectal cancer families from the Basque Country, describing four new germline mutations |
| Q58048546 | CLC and IFNAR1 are differentially expressed and a global immunity score is distinct between early- and late-onset colorectal cancer |
| Q41478453 | Contribution of large genomic rearrangements in Italian Lynch syndrome patients: characterization of a novel alu-mediated deletion |
| Q33575005 | Distinct high resolution genome profiles of early onset and late onset colorectal cancer integrated with gene expression data identify candidate susceptibility loci |
| Q83379045 | Genetics and epigenetics of small bowel adenocarcinoma: the interactions of CIN, MSI, and CIMP |
| Q55283890 | MAP3K7 is recurrently deleted in pediatric T-lymphoblastic leukemia and affects cell proliferation independently of NF-κB. |
| Q64102177 | PTPRK suppresses progression and chemo-resistance of colon cancer cells via direct inhibition of pro-oncogenic CD133 |
| Q53045957 | Receptor-type protein tyrosine phosphatase κ directly dephosphorylates CD133 and regulates downstream AKT activation. |
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