scholarly article | Q13442814 |
P50 | author | Martin Dichgans | Q30422338 |
Stefan F Lichtenthaler | Q63092222 | ||
P2093 | author name string | Thomas Misgeld | |
P2860 | cites work | A mutation in APP protects against Alzheimer’s disease and age-related cognitive decline | Q22251083 |
NLRP3 is activated in Alzheimer's disease and contributes to pathology in APP/PS1 mice | Q24604235 | ||
Pathogenic protein seeding in Alzheimer disease and other neurodegenerative disorders | Q26850298 | ||
The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics | Q27860914 | ||
Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide | Q28131750 | ||
TRAK/Milton motor-adaptor proteins steer mitochondrial trafficking to axons and dendrites | Q28594138 | ||
Genome-wide association study identifies a variant in HDAC9 associated with large vessel ischemic stroke | Q29417129 | ||
Axonal transport deficits and degeneration can evolve independently in mouse models of amyotrophic lateral sclerosis. | Q30511133 | ||
Microtubule stabilization reduces scarring and causes axon regeneration after spinal cord injury | Q30513561 | ||
2-photon imaging of phagocyte-mediated T cell activation in the CNS. | Q30536629 | ||
Loss of ALS-associated TDP-43 in zebrafish causes muscle degeneration, vascular dysfunction, and reduced motor neuron axon outgrowth | Q30538184 | ||
Cerebral amyloid-β proteostasis is regulated by the membrane transport protein ABCC1 in mice | Q34212267 | ||
Three-dimensional imaging of the unsectioned adult spinal cord to assess axon regeneration and glial responses after injury. | Q34242950 | ||
ABC transporters B1, C1 and G2 differentially regulate neuroregeneration in mice | Q34251174 | ||
Purified and synthetic Alzheimer's amyloid beta (Aβ) prions | Q36079533 | ||
Secretome protein enrichment identifies physiological BACE1 protease substrates in neurons | Q36103683 | ||
β-Site amyloid precursor protein (APP)-cleaving enzyme 1 (BACE1)-deficient mice exhibit a close homolog of L1 (CHL1) loss-of-function phenotype involving axon guidance defects | Q36385744 | ||
Divergent roles of ALS-linked proteins FUS/TLS and TDP-43 intersect in processing long pre-mRNAs | Q36650975 | ||
Advances in understanding the molecular basis of frontotemporal dementia | Q36775677 | ||
Mitochondrial DNA polymorphisms specifically modify cerebral β-amyloid proteostasis | Q36960466 | ||
Axonal transport deficits and neurodegenerative diseases | Q38077679 | ||
The changing scene of amyotrophic lateral sclerosis | Q38086771 | ||
Loss of fused in sarcoma (FUS) promotes pathological Tau splicing | Q42138048 | ||
Generation of Alzheimer disease-associated amyloid β42/43 peptide by γ-secretase can be inhibited directly by modulation of membrane thickness | Q42202065 | ||
Commensal microbiota and myelin autoantigen cooperate to trigger autoimmune demyelination. | Q48819558 | ||
Alzheimer's research. Stopping Alzheimer's before it starts | Q84824173 | ||
P433 | issue | 6 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 489-492 | |
P577 | publication date | 2013-05-14 | |
P1433 | published in | EMBO Reports | Q5323356 |
P1476 | title | Between new genetic discoveries and large randomized trials--neurological research in the era of systems medicine | |
P478 | volume | 14 |
Q38299414 | Advances in the therapy of Alzheimer's disease: targeting amyloid beta and tau and perspectives for the future | cites work | P2860 |
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