Alteration of the phosphocreatine energy shuttle components in diabetic rat heart

scientific article published on November 1, 1991

Alteration of the phosphocreatine energy shuttle components in diabetic rat heart is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/0022-2828(91)90089-5
P953full work available at URLhttps://api.elsevier.com/content/article/PII:0022-2828(91)90089-5?httpAccept=text/plain
https://api.elsevier.com/content/article/PII:0022-2828(91)90089-5?httpAccept=text/xml
P698PubMed publication ID1803023

P2093author name stringA. Kirsch
F. Savabi
P433issue11
P407language of work or nameEnglishQ1860
P921main subjectCardiology and cardiovascular medicineQ96320350
heart contractionQ5165683
P304page(s)1323-1333
P577publication date1991-11-01
P1433published inJournal of Molecular and Cellular CardiologyQ2061932
P1476titleAlteration of the phosphocreatine energy shuttle components in diabetic rat heart
P478volume23

Reverse relations

cites work (P2860)
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Q40561430In situ study of myofibrils, mitochondria and bound creatine kinases in experimental cardiomyopathies
Q35294180In vivo creatine kinase reaction kinetics at rest and stress in type II diabetic rat heart
Q35554282Increased expression of mitochondrial-encoded genes in skeletal muscle of humans with diabetes mellitus
Q60307777Insights on the impact of mitochondrial organisation on bioenergetics in high-resolution computational models of cardiac cell architecture
Q40961818MB fraction of cumulative creatine kinase correlates with insulin secretion in patients with acute myocardial infarction: insulin as a possible determinant of myocardial MB creatine kinase
Q38459035Metabolic abnormalities of the heart in type II diabetes
Q41992226Mitochondrial creatine kinase isoform expression does not correlate with its mode of action.
Q34464960Oxygen-bridged dinuclear ruthenium amine complex specifically inhibits Ca2+ uptake into mitochondria in vitro and in situ in single cardiac myocytes
Q39427090Phosphocreatine protects endothelial cells from Methylglyoxal induced oxidative stress and apoptosis via the regulation of PI3K/Akt/eNOS and NF-κB pathway

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