| scholarly article | Q13442814 |
| P2093 | author name string | Bernard Dutrillaux | |
| Michelle Debatisse | |||
| Lorène Rozier | |||
| Arnaud Coquelle | |||
| P2860 | cites work | The Fusion of Broken Ends of Chromosomes Following Nuclear Fusion | Q28913702 |
| Hypoxia-mediated selection of cells with diminished apoptotic potential in solid tumours | Q29618396 | ||
| Amplification of the human dihydrofolate reductase gene via double minutes is initiated by chromosome breaks | Q30305771 | ||
| Sister chromatid fusion initiates amplification of the dihydrofolate reductase gene in Chinese hamster cells | Q30467650 | ||
| Translocation breakpoints in FHIT and FRA3B in both homologs of chromosome 3 in an esophageal adenocarcinoma | Q31913764 | ||
| Oncogene amplification in solid tumors | Q33711338 | ||
| Chromosome breaks and genomic instability | Q33853722 | ||
| Chasing the cancer demon | Q34090719 | ||
| Fragile and unstable chromosomes in cancer: causes and consequences | Q34264402 | ||
| Double minute chromosomes can be produced from precursors derived from a chromosomal deletion | Q36786407 | ||
| Formation of an inverted duplication can be an initial step in gene amplification | Q36847076 | ||
| Chromosomal breakage-fusion-bridge events cause genetic intratumor heterogeneity | Q36973681 | ||
| Fusions near telomeres occur very early in the amplification of CAD genes in Syrian hamster cells | Q37059409 | ||
| Chromosomal instability and cytoskeletal defects in oral cancer cells | Q37150932 | ||
| Transient hypoxia enhances the frequency of dihydrofolate reductase gene amplification in Chinese hamster ovary cells | Q37394864 | ||
| Group I introns as mobile genetic elements: facts and mechanistic speculations--a review | Q38304950 | ||
| Cancer genetics, cytogenetics--defining the enemy within | Q38501800 | ||
| Frequent chromosomal translocations induced by DNA double-strand breaks | Q40872179 | ||
| Gene amplification and tumor progression | Q40912793 | ||
| A new role for hypoxia in tumor progression: induction of fragile site triggering genomic rearrangements and formation of complex DMs and HSRs | Q41009556 | ||
| Expression of fragile sites triggers intrachromosomal mammalian gene amplification and sets boundaries to early amplicons | Q41115353 | ||
| A central role for chromosome breakage in gene amplification, deletion formation, and amplicon integration | Q41696612 | ||
| Distinctive chromosomal structures are formed very early in the amplification of CAD genes in Syrian hamster cells | Q41710188 | ||
| Effect of transient hypoxia on sensitivity to doxorubicin in human and murine cell lines | Q41736961 | ||
| Early dihydrofolate reductase gene amplification events in CHO cells usually occur on the same chromosome arm as the original locus | Q41759145 | ||
| Interstitial deletions and intrachromosomal amplification initiated from a double-strand break targeted to a mammalian chromosome | Q42636230 | ||
| Introduction of double-strand breaks into the genome of mouse cells by expression of a rare-cutting endonuclease | Q42832099 | ||
| The chromosomal basis of human neoplasia. | Q53762162 | ||
| The origin of chromosome rearrangements at early stages of AMPD2 gene amplification in Chinese hamster cells. | Q54047889 | ||
| Life (and death) in a malignant tumour | Q59098941 | ||
| The Characterization of the Common Fragile Site FRA16D and Its Involvement in Multiple Myeloma Translocations | Q59150865 | ||
| A role for common fragile site induction in amplification of human oncogenes | Q63869173 | ||
| Mapping FRA11A, a folate-sensitive fragile site in human chromosome band 11q13.3. | Q74416922 | ||
| Fragile sites still breaking | Q77724528 | ||
| P433 | issue | 50 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 7671-7679 | |
| P577 | publication date | 2002-10-01 | |
| P1433 | published in | Oncogene | Q1568657 |
| P1476 | title | Induction of multiple double-strand breaks within an hsr by meganucleaseI-SceI expression or fragile site activation leads to formation of double minutes and other chromosomal rearrangements | |
| P478 | volume | 21 |
| Q27930879 | A mechanism of gene amplification driven by small DNA fragments |
| Q45001863 | Characterization of a conserved aphidicolin-sensitive common fragile site at human 4q22 and mouse 6C1: possible association with an inherited disease and cancer |
| Q30300009 | DNA secondary structure at chromosomal fragile sites in human disease |
| Q90380663 | Double-strand breakage in the extrachromosomal double minutes triggers their aggregation in the nucleus, micronucleation, and morphological transformation |
| Q47243157 | ERBB2, TBX2, RPS6KB1, and MYC alterations in breast tissues of BRCA1 and BRCA2 mutation carriers |
| Q42112893 | Gene amplification in human cells knocked down for RAD54 |
| Q64068884 | Generation and maintenance of acentric stable double minutes from chromosome arms in inter-species hybrid cells |
| Q40195901 | High-resolution mapping identifies a commonly amplified 11q13.3 region containing multiple genes flanked by segmental duplications |
| Q35213126 | How a replication origin and matrix attachment region accelerate gene amplification under replication stress in mammalian cells |
| Q34364066 | Inactivating Mutations in GT198 in Familial and Early-Onset Breast and Ovarian Cancers |
| Q22122003 | Mechanisms of change in gene copy number |
| Q37388726 | Molecular structure of double-minute chromosomes bearing amplified copies of the epidermal growth factor receptor gene in gliomas |
| Q38468410 | Multifaceted control of DNA repair pathways by the hypoxic tumor microenvironment |
| Q35790060 | Mutually exclusive promoter hypermethylation patterns of hMLH1 and O6-methylguanine DNA methyltransferase in colorectal cancer |
| Q80151845 | Non-incidental coamplification of Myc and ERBB2, and Myc and EGFR, in gastric adenocarcinomas |
| Q37843627 | Nurturing the genome: A-type lamins preserve genomic stability |
| Q58797497 | Patterns and mechanisms of structural variations in human cancer |
| Q36532727 | Reduced cohesin destabilizes high-level gene amplification by disrupting pre-replication complex bindings in human cancers with chromosomal instability |
| Q33350550 | Stably transfected common fragile site sequences exhibit instability at ectopic sites |
| Q35170919 | The role of DNA breaks in genomic instability and tumorigenesis |
| Q38155002 | Tumor hypoxia as a driving force in genetic instability. |
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