Molecular and pharmacological approaches to inhibiting nitric oxide after burn trauma

scientific article published on 8 May 2003

Molecular and pharmacological approaches to inhibiting nitric oxide after burn trauma is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1152/AJPHEART.00061.2002
P698PubMed publication ID12738625
P5875ResearchGate publication ID7316932

P2093author name stringJean White
Jureta W Horton
Deborah L Carlson
David L Maass
Marita Thompson
Brett Giroir
Billy Sanders
P2860cites workApparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxideQ29547245
Tumor necrosis factor-alpha gene and protein expression in adult feline myocardium after endotoxin administrationQ33826955
Abnormal contractile function due to induction of nitric oxide synthesis in rat cardiac myocytes follows exposure to activated macrophage-conditioned mediumQ33900508
Control of cardiac muscle cell function by an endogenous nitric oxide signaling systemQ36047757
Nitric oxide and cardiac function.Q41090625
Dedifferentiated human ventricular cardiac myocytes express inducible nitric oxide synthase mRNA but not protein in response to IL-1, TNF, IFNgamma, and LPS.Q41117907
Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serumQ41903981
Opposite effects of cyclic GMP and cyclic AMP on Ca2+ current in single heart cellsQ42487358
Induction and potential biological relevance of a Ca(2+)-independent nitric oxide synthase in the myocardiumQ42712096
Burn-induced impairment of cardiac contractile function is due to gut-derived factors transported in mesenteric lymphQ44158047
Burn-induced alterations in cardiac beta-adrenergic receptorsQ46492764
Long-term implications of reocclusion on left ventricular size and function after successful thrombolysis for first anterior myocardial infarctionQ47315798
Nitric oxide modulation of TNF-alpha-induced cardiac contractile dysfunction is concentration dependent.Q50511493
Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inflammatory cytokine TNF-alpha.Q51360555
Mechanical transduction of nitric oxide synthesis in the beating heart.Q52041726
Expression of the inducible nitric oxide synthase gene in diaphragm and skeletal muscle.Q54575813
P433issue4
P304page(s)H1616-25
P577publication date2003-05-08
P1433published inAmerican Journal of Physiology Heart and Circulatory PhysiologyQ3193662
P1476titleMolecular and pharmacological approaches to inhibiting nitric oxide after burn trauma
P478volume285

Reverse relations

cites work (P2860)
Q36998303Antithrombin attenuates myocardial dysfunction and reverses systemic fluid accumulation following burn and smoke inhalation injury: a randomized, controlled, experimental study
Q41334331Burn-induced organ dysfunction: vagus nerve stimulation improves cardiac function
Q26771250Cardiovascular Dysfunction Following Burn Injury: What We Have Learned from Rat and Mouse Models
Q46134163Inhibition of inducible nitric oxide synthase reduces an acute peripheral motor neuropathy produced by dermal burn injury in mice
Q45253905Mechanical traumatic injury without circulatory shock causes cardiomyocyte apoptosis: role of reactive nitrogen and reactive oxygen species
Q99549050Pathological Responses of Cardiac Mitochondria to Burn Trauma

Search more.