scholarly article | Q13442814 |
P356 | DOI | 10.1097/00024382-200309000-00013 |
P698 | PubMed publication ID | 12923501 |
P2093 | author name string | Rei-Cheng Yang | |
Hsiang-Wen Chen | |||
Shu-Jung Wang | |||
Tzong-Shi Lu | |||
Chin Hsu | |||
P433 | issue | 3 | |
P921 | main subject | sepsis | Q183134 |
P304 | page(s) | 274-279 | |
P577 | publication date | 2003-09-01 | |
P1433 | published in | Shock | Q7499831 |
P1476 | title | Heat shock pretreatment prevents cardiac mitochondrial dysfunction during sepsis | |
P478 | volume | 20 |
Q89444899 | Age-dependent cardiac function during experimental sepsis: effect of pharmacological activation of AMP-activated protein kinase by AICAR |
Q33838958 | Circulating heat shock protein 70 (HSPA1A) in normal and pathological pregnancies. |
Q46927405 | Early septic shock induces loss of oxidative phosphorylation yield plasticity in liver mitochondria. |
Q46314577 | Exercise pretraining attenuates endotoxin-induced hemodynamic alteration in type I diabetic rats |
Q37673307 | Exogenous cytochrome C restores myocardial cytochrome oxidase activity into the late phase of sepsis |
Q39367163 | Extracellular heat shock protein 72 protects schwann cells from hydrogen peroxide-induced apoptosis |
Q37569981 | Eye-specific gene expression following embryonic ethanol exposure in zebrafish: roles for heat shock factor 1. |
Q46173386 | Heat shock response protects human peritoneal mesothelial cells from dialysate-induced oxidative stress and mitochondrial injury |
Q37308055 | Heat-shock response protects peripheral blood mononuclear cells (PBMCs) from hydrogen peroxide-induced mitochondrial disturbance |
Q33788489 | Increased heat shock protein 70 expression in the pancreas of rats with endotoxic shock |
Q81210879 | Induction of the heat shock response in vivo inhibits NF-kappaB activity and protects murine liver from endotoxemia-induced injury |
Q35378402 | Inhibition of c-Jun-N-terminal kinase increases cardiac peroxisome proliferator-activated receptor alpha expression and fatty acid oxidation and prevents lipopolysaccharide-induced heart dysfunction. |
Q44089868 | Mitochondrial function in sepsis |
Q54976590 | Overexpressed UCP2 regulates mitochondrial flashes and reverses lipopolysaccharide-induced cardiomyocytes injury. |
Q28079058 | Pathophysiology of sepsis-induced myocardial dysfunction |
Q35761448 | Pathophysiology of sepsis-related cardiac dysfunction: driven by inflammation, energy mismanagement, or both? |
Q42111451 | Polyethylene glycol-superoxide dismutase prevents endotoxin-induced cardiac dysfunction |
Q34002691 | Proteomic profiling of endothelin-1-stimulated hypertrophic cardiomyocytes reveals the increase of four different desmin species and alpha-B-crystallin |
Q36923921 | Regulation of apoptotic and inflammatory cell signaling in cerebral ischemia: the complex roles of heat shock protein 70 |
Q42362538 | Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments |
Q35687065 | Sepsis-induced cardiomyopathy |
Q37767173 | Sepsis-induced cardiomyopathy: a review of pathophysiologic mechanisms |
Q50052866 | The 70-kDa heat shock protein (Hsp70) as a therapeutic target for stroke |
Search more.