| scholarly article | Q13442814 |
| P356 | DOI | 10.1152/AJPLUNG.00119.2003 |
| P698 | PubMed publication ID | 14672918 |
| P2093 | author name string | Marcienne M Wright | |
| Robert M Jackson | |||
| Charles S Powell | |||
| P2860 | cites work | MKK3- and MKK6-regulated gene expression is mediated by the p38 mitogen-activated protein kinase signal transduction pathway | Q24650627 |
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| Large unphosphorylated aggregates as the active form of hsp27 which controls intracellular reactive oxygen species and glutathione levels and generates a protection against TNFalpha in NIH-3T3-ras cells | Q41066626 | ||
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| Significant neuroprotection against ischemic brain injury by inhibition of the MEK1 protein kinase in mice: exploration of potential mechanism associated with apoptosis | Q44254966 | ||
| Sequential activation of activator protein-1-related transcription factors and JNK protein kinases may contribute to apoptotic death induced by transient hypoxia in developing brain neurons | Q48337860 | ||
| Oxidative stress activates anion exchange protein 2 and AP-1 in airway epithelial cells. | Q51704293 | ||
| P433 | issue | 4 | |
| P921 | main subject | hypoxia | Q105688 |
| P304 | page(s) | L826-33 | |
| P577 | publication date | 2003-12-12 | |
| P1433 | published in | American Journal of Physiology - Lung Cellular and Molecular Physiology | Q4352992 |
| P1476 | title | p38mapk and MEK1/2 inhibition contribute to cellular oxidant injury after hypoxia | |
| P478 | volume | 286 |
| Q41916170 | 2-cyclohexylamino-5,8-dimethoxy-1,4-naphthoquinone inhibits LPS-induced BV2 microglial activation through MAPK/NF-kB signaling pathways |
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| Q43175524 | Hypoxia and kinase activity regulate lung epithelial cell glutathione |
| Q39893836 | Hypoxia-associated p38 mitogen-activated protein kinase-mediated androgen receptor activation and increased HIF-1alpha levels contribute to emergence of an aggressive phenotype in prostate cancer |
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| Q34816615 | Subpollen particles: carriers of allergenic proteins and oxidases. |
| Q41686419 | Toll like receptors gene expression of human keratinocytes cultured of severe burn injury. |
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