Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy

scientific article published on 26 October 2016

Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1152/AJPREGU.00117.2016
P932PMC publication ID5256975
P698PubMed publication ID27784685

P50authorFlorian HerseQ47804678
Kedra WallaceQ56256861
Gerd WallukatQ63288594
Ralf DechendQ63812628
P2093author name stringJeremy Scott
Babbette LaMarca
Denise C Cornelius
Lorena M Amaral
Alexia J Thomas
Nathan Campbell
P2860cites workAT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factorQ73815485
Reduced uterine perfusion pressure (RUPP) model for studying cardiovascular-renal dysfunction in response to placental ischemiaQ82705098
Increased prevalence of IL-17-producing peripheral blood lymphocytes in pre-eclampsiaQ83391804
The predominance of Th17 lymphocytes and decreased number and function of Treg cells in preeclampsiaQ83523001
Hypertension in response to AT1-AA: role of reactive oxygen species in pregnancy-induced hypertensionQ83799951
A leading role for the immune system in the pathophysiology of preeclampsiaQ27003069
Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptorQ33848380
Recent insights into the pathophysiology of preeclampsiaQ34404445
Recent Insights into the pathogenesis of pre-eclampsiaQ34682287
CD4+ T-helper cells stimulated in response to placental ischemia mediate hypertension during pregnancyQ35130793
Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunctionQ35576417
IL-6-induced pathophysiology during pre-eclampsia: potential therapeutic role for magnesium sulfate?Q35581520
Activating autoantibodies to the angiotensin II type I receptor play an important role in mediating hypertension in response to adoptive transfer of CD4+ T lymphocytes from placental ischemic ratsQ35994304
Activating auto-antibodies against the AT1 receptor in preeclampsiaQ36010629
The Th1:th2 dichotomy of pregnancy and preterm labour.Q36037674
IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancyQ36178540
Agonistic antibodies directed at the angiotensin II, AT1 receptor in preeclampsiaQ36380482
The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancyQ36407259
The role of immune activation in contributing to vascular dysfunction and the pathophysiology of hypertension during preeclampsiaQ37088970
Hypertension in response to autoantibodies to the angiotensin II type I receptor (AT1-AA) in pregnant rats: role of endothelin-1.Q37447371
Role of reactive oxygen species in hypertension produced by reduced uterine perfusion in pregnant ratsQ37449704
Administration of interleukin-17 soluble receptor C suppresses TH17 cells, oxidative stress, and hypertension in response to placental ischemia during pregnancyQ37507162
Th1/Th2/Th17 and regulatory T-cell paradigm in pregnancyQ37747554
Deciphering the role of Th17 cells in human diseaseQ37940695
Th17 and regulatory T cells in women with recurrent pregnancy lossQ37989545
Interleukin-6 in pregnancy and gestational disordersQ38028200
Systemic increase in the ratio between Foxp3+ and IL-17-producing CD4+ T cells in healthy pregnancy but not in preeclampsiaQ46332630
Effects of reduced uterine perfusion pressure on blood pressure and metabolic factors in pregnant rats.Q51470693
Emerging roles of antiangiogenic and angiogenic proteins in pathogenesis and prediction of preeclampsiaQ61845360
P433issue6
P921main subjectpre-eclampsiaQ61335
pathophysiologyQ1135939
P304page(s)R1192-R1199
P577publication date2016-10-26
P1433published inAmerican Journal of Physiology - Regulatory, Integrative and Comparative PhysiologyQ2201819
P1476titleReduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy
P478volume311

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cites work (P2860)
Q64068024Chronic infusion of interleukin-17 promotes hypertension, activation of cytolytic natural killer cells, and vascular dysfunction in pregnant rats
Q89522650Dietary Protein: Mechanisms Influencing Hypertension and Renal Disease
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Q49600503Immune regulation of systemic hypertension, pulmonary arterial hypertension, and preeclampsia: shared disease mechanisms and translational opportunities
Q46300954Natural killer cells and T lymphocytes in pregnancy and pre-eclampsia
Q47637232Natural killer cells mediate pathophysiology in response to reduced uterine perfusion pressure
Q90628338Neurodevelopmental Outcomes of Prenatal Preeclampsia Exposure
Q88542480Placental ischemia-stimulated T-helper 17 cells induce preeclampsia-associated cytolytic natural killer cells during pregnancy
Q104137018Preeclampsia: Linking Placental Ischemia with Maternal Endothelial and Vascular Dysfunction
Q57057008Regulatory T cells in embryo implantation and the immune response to pregnancy
Q47819556The placental bed vascular pathology revisited: a risk indicator for cardiovascular disease
Q64065212Therapeutic Potential of Regulatory T Cells in Preeclampsia-Opportunities and Challenges
Q54680135Uncovering novel roles for matrix metalloproteinases in preeclampsia.
Q47968875Undifferentiated connective tissue diseases and adverse pregnancy outcomes. An undervalued association?

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