Chronic prenatal exposure to carbon monoxide results in a reduction in tyrosine hydroxylase-immunoreactivity and an increase in choline acetyltransferase-immunoreactivity in the fetal medulla: implications for Sudden Infant Death Syndrome

scientific article published in March 2000

Chronic prenatal exposure to carbon monoxide results in a reduction in tyrosine hydroxylase-immunoreactivity and an increase in choline acetyltransferase-immunoreactivity in the fetal medulla: implications for Sudden Infant Death Syndrome is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1093/JNEN/59.3.218
P698PubMed publication ID10744060

P2093author name stringWalker D
Rees S
Tolcos M
McGregor H
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Correlation of Astrogliosis and Substance P Immunoreactivity in the Brainstem of Victims of Sudden Infant Death SyndromeQ48245399
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Alteration in the postnatal ontogeny of cerebellar norepinephrine content following chronic prenatal carbon monoxideQ48456466
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Chronic placental insufficiency in the fetal guinea pig affects neurochemical and neuroglial development but not neuronal numbers in the brainstem: a new method for combined stereology and immunohistochemistryQ48768573
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P433issue3
P407language of work or nameEnglishQ1860
P921main subjectcarbon monoxideQ2025
P304page(s)218-228
P577publication date2000-03-01
P1433published inJournal of Neuropathology & Experimental NeurologyQ15716771
P1476titleChronic prenatal exposure to carbon monoxide results in a reduction in tyrosine hydroxylase-immunoreactivity and an increase in choline acetyltransferase-immunoreactivity in the fetal medulla: implications for Sudden Infant Death Syndrome
P478volume59

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cites work (P2860)
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Q44451513The fetal brainstem is relatively spared from injury following intrauterine hypoxemia
Q48258499Vertebral artery insufficiency as a possible mechanism for sudden infant death--in vivo evidence does not support findings from postmortem studies

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