scholarly article | Q13442814 |
P356 | DOI | 10.1007/978-3-319-57379-3_24 |
P698 | PubMed publication ID | 28971465 |
P2093 | author name string | Nelina P Angelova | |
P2860 | cites work | Modeling the mitochondrial dysfunction in neurogenerative diseases due to high H+ concentration | Q42122769 |
Altered proteasomal function in sporadic Parkinson's disease | Q44264446 | ||
Late-onset Huntington disease with intermediate CAG repeats: true or false? | Q45296632 | ||
Mitochondrial respiration and ATP production are significantly impaired in striatal cells expressing mutant huntingtin | Q45297269 | ||
Huntington's disease is a four-repeat tauopathy with tau nuclear rods. | Q45300275 | ||
The association of CAG repeat length with clinical progression in Huntington disease | Q45300690 | ||
Transcriptional repression of PGC-1alpha by mutant huntingtin leads to mitochondrial dysfunction and neurodegeneration. | Q45302702 | ||
Autopsy-proven Huntington's disease with 29 trinucleotide repeats | Q45303229 | ||
Mitochondrial dynamics and mitophagy in Parkinson's disease: A fly point of view | Q46642004 | ||
Different species of alpha-synuclein oligomers induce calcium influx and seeding. | Q52581429 | ||
Huntingtin interacting proteins are genetic modifiers of neurodegeneration | Q21145230 | ||
The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription | Q22254119 | ||
Interaction of Huntington disease protein with transcriptional activator Sp1 | Q24537687 | ||
Autophagy and the ubiquitin-proteasome system: collaborators in neuroprotection | Q24650675 | ||
It Is All about (U)biquitin: Role of Altered Ubiquitin-Proteasome System and UCHL1 in Alzheimer Disease | Q26765816 | ||
Alterations in Mitochondrial Quality Control in Alzheimer's Disease | Q26768215 | ||
Structural Basis of E2-25K/UBB+1 Interaction Leading to Proteasome Inhibition and Neurotoxicity | Q27664383 | ||
A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. | Q27860836 | ||
Huntingtin functions as a scaffold for selective macroautophagy | Q28118022 | ||
Loss of huntingtin-mediated BDNF gene transcription in Huntington's disease | Q28202050 | ||
REST and stress resistance in ageing and Alzheimer's disease | Q28236857 | ||
The role of mTOR signaling in Alzheimer disease | Q28256247 | ||
The role of autophagy in age-related neurodegeneration | Q28261965 | ||
Inhibition of mTOR induces autophagy and reduces toxicity of polyglutamine expansions in fly and mouse models of Huntington disease | Q28261987 | ||
The roles of intracellular protein-degradation pathways in neurodegeneration | Q28269322 | ||
Protein aggregation and neurodegenerative disease | Q28273600 | ||
Targeted disruption of the Huntington's disease gene results in embryonic lethality and behavioral and morphological changes in heterozygotes | Q28302701 | ||
Inhibition of mitochondrial protein import by mutant huntingtin | Q28394803 | ||
Huntingtin associated protein 1 regulates trafficking of the amyloid precursor protein and modulates amyloid beta levels in neurons | Q28575380 | ||
The regulation of autophagosome dynamics by huntingtin and HAP1 is disrupted by expression of mutant huntingtin, leading to defective cargo degradation | Q28591685 | ||
Mutant huntingtin fragments form oligomers in a polyglutamine length-dependent manner in vitro and in vivo | Q30494325 | ||
Cargo recognition failure is responsible for inefficient autophagy in Huntington's disease | Q33814719 | ||
Huntington's disease: from huntingtin function and dysfunction to therapeutic strategies. | Q36623762 | ||
An N-terminal nuclear export signal regulates trafficking and aggregation of Huntingtin (Htt) protein exon 1. | Q36647206 | ||
Cytoplasmic aggregates trap polyglutamine-containing proteins and block axonal transport in a Drosophila model of Huntington's disease. | Q36854847 | ||
Huntingtin modulates transcription, occupies gene promoters in vivo, and binds directly to DNA in a polyglutamine-dependent manner | Q36981055 | ||
Huntingtin as an essential integrator of intracellular vesicular trafficking | Q37408730 | ||
Small changes, big impact: posttranslational modifications and function of huntingtin in Huntington disease. | Q37839534 | ||
Mitochondrial dynamics and quality control in Huntington's disease. | Q38590033 | ||
Brain REST/NRSF Is Not Only a Silent Repressor but Also an Active Protector | Q38689612 | ||
Widespread disruption of repressor element-1 silencing transcription factor/neuron-restrictive silencer factor occupancy at its target genes in Huntington's disease. | Q39333674 | ||
Mutant huntingtin directly increases susceptibility of mitochondria to the calcium-induced permeability transition and cytochrome c release | Q40551652 | ||
Early mitochondrial calcium defects in Huntington's disease are a direct effect of polyglutamines. | Q40721588 | ||
Rapamycin protects against neuron death in in vitro and in vivo models of Parkinson's disease | Q40887263 | ||
The Aggregation of Huntingtin and α-Synuclein | Q41834257 | ||
P407 | language of work or name | English | Q1860 |
P921 | main subject | neurodegeneration | Q1755122 |
P1104 | number of pages | 13 | |
P304 | page(s) | 269-281 | |
P577 | publication date | 2017-01-01 | |
P1433 | published in | Advances in Experimental Medicine and Biology | Q4686385 |
P1476 | title | Searching for Correlations Between the Development of Neurodegenerative Hallmarks: Targeting Huntingtin as a Contributing Factor | |
P478 | volume | 987 |
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