A function retained by the common mutant CLN3 protein is responsible for the late onset of juvenile neuronal ceroid lipofuscinosis.

scientific article published on 18 October 2007

A function retained by the common mutant CLN3 protein is responsible for the late onset of juvenile neuronal ceroid lipofuscinosis. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1093/HMG/DDM306
P698PubMed publication ID17947292
P5875ResearchGate publication ID5898962

P50authorSara E. MoleQ42563205
Daniel F CutlerQ57037508
Claudia KitzmüllerQ73893954
P2093author name stringSandra Codlin
Rebecca L Haines
P433issue2
P407language of work or nameEnglishQ1860
P921main subjectjuvenile neuronal ceroid lipofuscinosisQ1753778
P304page(s)303-312
P577publication date2007-10-18
P1433published inHuman Molecular GeneticsQ2720965
P1476titleA function retained by the common mutant CLN3 protein is responsible for the late onset of juvenile neuronal ceroid lipofuscinosis
P478volume17

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cites work (P2860)
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