scholarly article | Q13442814 |
P356 | DOI | 10.1007/S12185-017-2346-6 |
P698 | PubMed publication ID | 29022209 |
P50 | author | Irina Kozhukharova | Q57013148 |
P2093 | author name string | Victoria Zemelko | |
Nikolay Nikolsky | |||
Olga Lyublinskaya | |||
Larisa Alekseenko | |||
Zoya Kovaleva | |||
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Paracrine factors of mesenchymal stem cells recruit macrophages and endothelial lineage cells and enhance wound healing | Q33326623 | ||
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Persistent DNA damage-induced premature senescence alters the functional features of human bone marrow mesenchymal stem cells. | Q35390924 | ||
A therapeutic dose of doxorubicin activates ubiquitin-proteasome system-mediated proteolysis by acting on both the ubiquitination apparatus and proteasome. | Q37124125 | ||
Sublethal oxidative stress induces the premature senescence of human mesenchymal stem cells derived from endometrium | Q37156197 | ||
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Anthracycline cardiomyopathy is mediated by depletion of the cardiac stem cell pool and is rescued by restoration of progenitor cell function | Q41883542 | ||
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P921 | main subject | menstruation | Q12171 |
doxorubicin | Q18936 | ||
P577 | publication date | 2017-10-11 | |
P1433 | published in | International Journal of Hematology | Q6051416 |
P1476 | title | Therapeutic doses of doxorubicin induce premature senescence of human mesenchymal stem cells derived from menstrual blood, bone marrow and adipose tissue |
Q89657824 | Cell-Based Nanoparticles Delivery Systems for Targeted Cancer Therapy: Lessons from Anti-Angiogenesis Treatments |
Q98178384 | Development of Natural-Based Bone Cement for a Controlled Doxorubicin-Drug Release |
Q61801033 | High doses of synthetic antioxidants induce premature senescence in cultivated mesenchymal stem cells |
Q93147053 | Mesenchymal Stem Cell Therapy for Doxorubicin-Induced Cardiomyopathy: Potential Mechanisms, Governing Factors, and Implications of the Heart Stem Cell Debate |
Q61455264 | Quiescent Human Mesenchymal Stem Cells Are More Resistant to Heat Stress than Cycling Cells |
Q96954753 | Three-Dimensional Compaction Switches Stress Response Programs and Enhances Therapeutic Efficacy of Endometrial Mesenchymal Stem/Stromal Cells |
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