scholarly article | Q13442814 |
P356 | DOI | 10.1038/LEU.2017.203 |
P698 | PubMed publication ID | 28659618 |
P50 | author | Thilo Bracht | Q57165790 |
P2093 | author name string | M Schneider | |
L Klein-Hitpass | |||
R Küppers | |||
S Hartmann | |||
M-L Hansmann | |||
M A Weniger | |||
B Sitek | |||
J Arnolds | |||
A L Weiß | |||
A Lollies | |||
P2860 | cites work | Cooperative epigenetic modulation by cancer amplicon genes | Q24608701 |
Molecular diagnosis of primary mediastinal B cell lymphoma identifies a clinically favorable subgroup of diffuse large B cell lymphoma related to Hodgkin lymphoma | Q24672476 | ||
The biology and management of systemic anaplastic large cell lymphoma | Q27009497 | ||
Batf3 Deficiency Reveals a Critical Role for CD8 + Dendritic Cells in Cytotoxic T Cell Immunity | Q27489852 | ||
Anaplastic lymphoma kinase (ALK) activates Stat3 and protects hematopoietic cells from cell death | Q28202067 | ||
A detailed inventory of DNA copy number alterations in four commonly used Hodgkin's lymphoma cell lines | Q28235071 | ||
Analyzing primary Hodgkin and Reed-Sternberg cells to capture the molecular and cellular pathogenesis of classical Hodgkin lymphoma | Q39284003 | ||
AP-1 mediated relief of repressive activity of the CD30 promoter microsatellite in Hodgkin and Reed-Sternberg cells | Q39392332 | ||
Aberrantly expressed c-Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF-kappa B. | Q39647760 | ||
Gene expression profiling of isolated tumour cells from anaplastic large cell lymphomas: insights into its cellular origin, pathogenesis and relation to Hodgkin lymphoma. | Q39816246 | ||
c-Jun expression and activation are restricted to CD30+ lymphoproliferative disorders | Q40166030 | ||
JunB induced by constitutive CD30-extracellular signal-regulated kinase 1/2 mitogen-activated protein kinase signaling activates the CD30 promoter in anaplastic large cell lymphoma and reed-sternberg cells of Hodgkin lymphoma | Q40377891 | ||
Origin and pathogenesis of nodular lymphocyte-predominant Hodgkin lymphoma as revealed by global gene expression analysis | Q42097322 | ||
JunB expression is a common feature of CD30+ lymphomas and lymphomatoid papulosis | Q42278940 | ||
The platelet-derived growth factor controls c-myc expression through a JNK- and AP-1-dependent signaling pathway. | Q42835267 | ||
The proto-oncogene MYC is required for selection in the germinal center and cyclic reentry | Q43091743 | ||
The oncogenic JUNB/CD30 axis contributes to cell cycle deregulation in ALK+ anaplastic large cell lymphoma. | Q54327994 | ||
Detection of genomic imbalances in microdissected Hodgkin and Reed-Sternberg cells of classical Hodgkin's lymphoma by array-based comparative genomic hybridization. | Q54523702 | ||
Genomic profiling reveals different genetic aberrations in systemic ALK-positive and ALK-negative anaplastic large cell lymphomas. | Q54546572 | ||
Expression pattern of MUM1/IRF4 in the spectrum of pathology of Hodgkin's disease | Q59647766 | ||
Genomic imbalances including amplification of the tyrosine kinase gene JAK2 in CD30+ Hodgkin cells | Q73453075 | ||
Signal transducer and activator of transcription 6 is frequently activated in Hodgkin and Reed-Sternberg cells of Hodgkin lymphoma | Q77461381 | ||
AP1-dependent galectin-1 expression delineates classical hodgkin and anaplastic large cell lymphomas from other lymphoid malignancies with shared molecular features | Q81385857 | ||
The case of the missing c-Myc | Q85227334 | ||
Expression profiles of MYC protein and MYC gene rearrangement in lymphomas | Q86515333 | ||
Aberrant expression of the Th2 cytokine IL-21 in Hodgkin lymphoma cells regulates STAT3 signaling and attracts Treg cells via regulation of MIP-3alpha | Q28289924 | ||
Oncogenically active MYD88 mutations in human lymphoma | Q28301648 | ||
Compensatory dendritic cell development mediated by BATF-IRF interactions | Q28586184 | ||
BATF-JUN is critical for IRF4-mediated transcription in T cells | Q28590152 | ||
Exploiting synthetic lethality for the therapy of ABC diffuse large B cell lymphoma | Q28649793 | ||
Deep sequencing of MYC DNA-binding sites in Burkitt lymphoma | Q28742928 | ||
AP-1: a double-edged sword in tumorigenesis | Q30014831 | ||
Mutations of the tumor suppressor gene SOCS-1 in classical Hodgkin lymphoma are frequent and associated with nuclear phospho-STAT5 accumulation | Q33236181 | ||
Gene expression profiling uncovers molecular classifiers for the recognition of anaplastic large-cell lymphoma within peripheral T-cell neoplasms | Q34099059 | ||
Gene expression profiling of microdissected Hodgkin Reed-Sternberg cells correlates with treatment outcome in classical Hodgkin lymphoma | Q34407534 | ||
Mapping of transcription factor motifs in active chromatin identifies IRF5 as key regulator in classical Hodgkin lymphoma | Q34408939 | ||
Profiling of Hodgkin's lymphoma cell line L1236 and germinal center B cells: identification of Hodgkin's lymphoma-specific genes | Q34520636 | ||
The expression of the Hodgkin's disease associated antigen Ki-1 in reactive and neoplastic lymphoid tissue: evidence that Reed-Sternberg cells and histiocytic malignancies are derived from activated lymphoid cells | Q34693140 | ||
Recurrent somatic mutations of PTPN1 in primary mediastinal B cell lymphoma and Hodgkin lymphoma | Q35095028 | ||
Constitutive AP-1 activity and EBV infection induce PD-L1 in Hodgkin lymphomas and posttransplant lymphoproliferative disorders: implications for targeted therapy | Q35875897 | ||
Hodgkin disease: Hodgkin and Reed-Sternberg cells picked from histological sections show clonal immunoglobulin gene rearrangements and appear to be derived from B cells at various stages of development | Q35888216 | ||
Overexpression of Batf induces an apoptotic defect and an associated lymphoproliferative disorder in mice | Q36004222 | ||
Hodgkin and Reed-Sternberg cells in Hodgkin's disease represent the outgrowth of a dominant tumor clone derived from (crippled) germinal center B cells. | Q36367466 | ||
Constitutively activated STAT3 promotes cell proliferation and survival in the activated B-cell subtype of diffuse large B-cell lymphomas | Q36407235 | ||
Recurrent somatic loss of TNFRSF14 in classical Hodgkin lymphoma | Q36463861 | ||
Cooperative signaling through the signal transducer and activator of transcription 3 and nuclear factor-{kappa}B pathways in subtypes of diffuse large B-cell lymphoma | Q36508931 | ||
IL-21 is expressed in Hodgkin lymphoma and activates STAT5: evidence that activated STAT5 is required for Hodgkin lymphomagenesis | Q36591359 | ||
IRF4 addiction in multiple myeloma | Q36891060 | ||
MicroRNA expression profiling identifies molecular signatures associated with anaplastic large cell lymphoma | Q37186470 | ||
Pathogenesis of classical and lymphocyte-predominant Hodgkin lymphoma | Q37463052 | ||
Epigenetic gene regulation by Janus kinase 1 in diffuse large B-cell lymphoma. | Q37469613 | ||
Specificity through cooperation: BATF-IRF interactions control immune-regulatory networks | Q38115966 | ||
Stat3 is required for ALK-mediated lymphomagenesis and provides a possible therapeutic target | Q38326252 | ||
Gene expression profiling reveals a highly specialized genetic program of plasma cells | Q38358380 | ||
A multicolor panel of novel lentiviral "gene ontology" (LeGO) vectors for functional gene analysis. | Q38514169 | ||
Convergent mutations and kinase fusions lead to oncogenic STAT3 activation in anaplastic large cell lymphoma. | Q38887338 | ||
Essential role of IRF4 and MYC signaling for survival of anaplastic large cell lymphoma | Q38943591 | ||
PDGFR blockade is a rational and effective therapy for NPM-ALK-driven lymphomas | Q39259618 | ||
P433 | issue | 1 | |
P921 | main subject | Hodgkin lymphoma | Q209369 |
P304 | page(s) | 92-101 | |
P577 | publication date | 2017-06-29 | |
P1433 | published in | Leukemia | Q6534498 |
P1476 | title | An oncogenic axis of STAT-mediated BATF3 upregulation causing MYC activity in classical Hodgkin lymphoma and anaplastic large cell lymphoma | |
P478 | volume | 32 |
Q59357963 | Contribution of Epstein⁻Barr Virus Latent Proteins to the Pathogenesis of Classical Hodgkin Lymphoma |
Q52718917 | Ectopic expression of transcription factor BATF3 induces B-cell lymphomas in a murine B-cell transplantation model. |
Q96576631 | Genomic deregulation of PRMT5 supports growth and stress tolerance in chronic lymphocytic leukemia |
Q64917080 | Human CD30+ B cells represent a unique subset related to Hodgkin lymphoma cells. |
Q49186472 | IRF4 Mediates the Oncogenic Effects of STAT3 in Anaplastic Large Cell Lymphomas. |
Q95936602 | Intestinal epithelium-derived BATF3 promotes colitis-associated colon cancer through facilitating CXCL5-mediated neutrophils recruitment |
Q64076640 | Investigating the Role of BATF3 in Grass Carp () Immune Modulation: A Fundamental Functional Analysis |
Q52585070 | MiR-760 suppresses human colorectal cancer growth by targeting BATF3/AP-1/cyclinD1 signaling. |
Q46210826 | New developments in the pathology of malignant lymphoma: a review of the literature published from May to August 2017. |
Q45066703 | S1PR1 drives a feedforward signalling loop to regulate BATF3 and the transcriptional programme of Hodgkin lymphoma cells |
Q52806518 | The AP-1-BATF and -BATF3 module is essential for growth, survival and TH17/ILC3 skewing of anaplastic large cell lymphoma. |
Q54117760 | The Pathological Spectrum of Systemic Anaplastic Large Cell Lymphoma (ALCL). |
Q52337767 | The Role of Activator Protein-1 (AP-1) Family Members in CD30-Positive Lymphomas. |
Q58099160 | The c-Jun and JunB transcription factors facilitate the transit of classical Hodgkin lymphoma tumour cells through G |
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