Epothilone B Benefits Nigrostriatal Pathway Recovery by Promoting Microtubule Stabilization After Intracerebral Hemorrhage

scientific article published on 18 January 2018

Epothilone B Benefits Nigrostriatal Pathway Recovery by Promoting Microtubule Stabilization After Intracerebral Hemorrhage is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1161/JAHA.117.007626
P932PMC publication ID5850167
P698PubMed publication ID29348323

P2093author name stringYang Yang
Rong Hu
Wei Liu
John H Zhang
Xin Liu
Xuan Zhang
Xiaotang Fan
Yuanyuan Ma
Hongfei Ge
Hua Feng
Yujie Chen
Hengli Zhao
Qianwei Chen
Jichao Yuan
Weixiang Chen
Rongwei Li
Eryi Sun
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Delayed treatment with monoclonal antibody IN-1 1 week after stroke results in recovery of function and corticorubral plasticity in adult ratsQ48889125
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Axonal regeneration. Systemic administration of epothilone B promotes axon regeneration after spinal cord injuryQ27331607
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P4510describes a project that usesImageJQ1659584
P433issue2
P577publication date2018-01-18
P1433published inJournal of the American Heart Association: Cardiovascular and Cerebrovascular DiseaseQ19880670
P1476titleEpothilone B Benefits Nigrostriatal Pathway Recovery by Promoting Microtubule Stabilization After Intracerebral Hemorrhage
P478volume7

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cites work (P2860)
Q92068835Ambroxol Improves Neuronal Survival and Reduces White Matter Damage through Suppressing Endoplasmic Reticulum Stress in Microglia after Intracerebral Hemorrhage
Q57490051Epothilone B Benefits Nigral Dopaminergic Neurons by Attenuating Microglia Activation in the 6-Hydroxydopamine Lesion Mouse Model of Parkinson's Disease
Q96303157Epothilone B Facilitates Peripheral Nerve Regeneration by Promoting Autophagy and Migration in Schwann Cells
Q89566145Naturally Occurring Antibodies to Tau Exists in Human Blood and Are Not Changed in Alzheimer's Disease
Q90680000SVCT2 Promotes Neural Stem/Progenitor Cells Migration Through Activating CDC42 After Ischemic Stroke
Q58714411Stably maintained microtubules protect dopamine neurons and alleviate depression-like behavior after intracerebral hemorrhage
Q64105224TRPA1 Activation-Induced Myelin Degradation Plays a Key Role in Motor Dysfunction After Intracerebral Hemorrhage

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