scholarly article | Q13442814 |
P50 | author | Fabio Di Domenico | Q38804465 |
Shelby Meier | Q59693208 | ||
Frederick A. Schmitt | Q64778804 | ||
Chiara Lanzillotta | Q87777117 | ||
P2093 | author name string | Jose F Abisambra | |
Marzia Perluigi | |||
Eugenio Barone | |||
Antonella Tramutola | |||
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Increased Mammalian Target of Rapamycin Signaling Contributes to the Accumulation of Protein Oxidative Damage in a Mouse Model of Down's Syndrome | Q38816807 | ||
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Loss of correlations among proteins in brains of the Ts65Dn mouse model of down syndrome. | Q48731504 | ||
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A mouse model for Down syndrome exhibits learning and behaviour deficits. | Q52013436 | ||
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The prevalence of dementia in Down syndrome | Q71603737 | ||
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ATF6 is a transcription factor specializing in the regulation of quality control proteins in the endoplasmic reticulum | Q80935578 | ||
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Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase | Q28296183 | ||
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum | Q28509611 | ||
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Redox proteomics analysis to decipher the neurobiology of Alzheimer-like neurodegeneration: overlaps in Down's syndrome and Alzheimer's disease brain. | Q30367032 | ||
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The neurology of Alzheimer disease in Down syndrome | Q35417016 | ||
Bach1 overexpression in Down syndrome correlates with the alteration of the HO-1/BVR-a system: insights for transition to Alzheimer's disease | Q36367840 | ||
Glucose-regulated protein 94 triage of mutant myocilin through endoplasmic reticulum-associated degradation subverts a more efficient autophagic clearance mechanism | Q36418920 | ||
Molecular characterization of the translocation breakpoints in the Down syndrome mouse model Ts65Dn. | Q36422268 | ||
Beta-amyloid, oxidative stress and down syndrome | Q36683240 | ||
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Tau accumulation activates the unfolded protein response by impairing endoplasmic reticulum-associated degradation | Q37069517 | ||
Aging in Down Syndrome and the Development of Alzheimer's Disease Neuropathology | Q37102001 | ||
Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype | Q37141585 | ||
The unfolded protein response is activated in pretangle neurons in Alzheimer's disease hippocampus | Q37164517 | ||
Modelling Down syndrome | Q37332055 | ||
Endoplasmic reticulum protein quality control in neurodegenerative disease: the good, the bad and the therapy | Q37387412 | ||
The unfolded protein response is activated in disease-affected brain regions in progressive supranuclear palsy and Alzheimer's disease | Q37485744 | ||
Impairment of proteostasis network in Down syndrome prior to the development of Alzheimer's disease neuropathology: redox proteomics analysis of human brain. | Q37613081 | ||
Activation of the unfolded protein response is an early event in Alzheimer's and Parkinson's disease. | Q37981067 | ||
P433 | issue | 1 | |
P921 | main subject | Down syndrome | Q47715 |
P304 | page(s) | 347-359 | |
P577 | publication date | 2018-01-01 | |
P1433 | published in | Journal of Alzheimer's Disease | Q6294755 |
P1476 | title | Early and Selective Activation and Subsequent Alterations to the Unfolded Protein Response in Down Syndrome Mouse Models | |
P478 | volume | 62 |
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Q98177367 | Down Syndrome Is a Metabolic Disease: Altered Insulin Signaling Mediates Peripheral and Brain Dysfunctions |
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